A physiologically based approach to the study of bisphenol A and other estrogenic chemicals on the size of reproductive organs, daily sperm production, and behavior.
about
The toxic origins of diseaseExposure to a low dose of bisphenol A during fetal life or in adulthood alters maternal behavior in miceBiotransformations of bisphenol A in a mammalian model: answers and new questions raised by low-dose metabolic fate studies in pregnant CD1 miceBisphenol A is released from used polycarbonate animal cages into water at room temperatureThe estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistanceUrinary concentrations of bisphenol A and 4-nonylphenol in a human reference populationAn extensive new literature concerning low-dose effects of bisphenol A shows the need for a new risk assessmentEnvironment and health: 6. Endocrine disruption and potential human health implicationsAre environmental levels of bisphenol a associated with reproductive function in fertile men?Polycarbonate bottle use and urinary bisphenol A concentrationsWhy public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol ADirect evidence revealing structural elements essential for the high binding ability of bisphenol A to human estrogen-related receptor-gammaExposure of the U.S. population to bisphenol A and 4-tertiary-octylphenol: 2003-2004Low phytoestrogen levels in feed increase fetal serum estradiol resulting in the "fetal estrogenization syndrome" and obesity in CD-1 miceEvaluating the effects of endocrine disruptors on endocrine function during developmentIn vivo effects of bisphenol A in laboratory rodent studiesDisposition of orally administered 2,2-Bis(4-hydroxyphenyl)propane (Bisphenol A) in pregnant rats and the placental transfer to fetusesHighly chlorinated PCBs inhibit the human xenobiotic response mediated by the steroid and xenobiotic receptor (SXR).Parent bisphenol A accumulation in the human maternal-fetal-placental unitEarly cerebral activities of the environmental estrogen bisphenol A appear to act via the somatostatin receptor subtype sst(2)Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levelsAggressive behavior and serum testosterone concentration during the maturation process of male mice: the effects of fetal exposure to bisphenol A.Relationship between expression of sex steroid receptors and structure of the seminal vesicles after neonatal treatment of rats with potent or weak estrogensLarge effects from small exposures. I. Mechanisms for endocrine-disrupting chemicals with estrogenic activityEstrogen regulation of testicular functionThe OECD program to validate the rat uterotrophic bioassay. Phase 2: dietary phytoestrogen analyses.In vivo imaging of activated estrogen receptors in utero by estrogens and bisphenol A.Non-monotonic dose-response relationships and endocrine disruptors: a qualitative method of assessmentLow-dose bioactivity of xenoestrogens in animals: fetal exposure to low doses of methoxychlor and other xenoestrogens increases adult prostate size in miceBisphenol A affects early bovine embryo development and metabolism that is negated by an oestrogen receptor inhibitorBisphenol A and Human Reproductive HealthEvidence that bisphenol A (BPA) can be accurately measured without contamination in human serum and urine, and that BPA causes numerous hazards from multiple routes of exposureBisphenol-A: epigenetic reprogramming and effects on reproduction and behaviorThe bisphenol A experience: a primer for the analysis of environmental effects on mammalian reproductionBisphenol A exposure in utero disrupts early oogenesis in the mouseExposure of neonatal rats to the endocrine disrupter Bisphenol A affects ontogenic expression pattern of testicular steroid receptors and their coregulatorsAn old culprit but a new story: bisphenol A and “NextGen” bisphenolsEffect of endocrine disruptors on male reproduction in humans: why the evidence is still lacking?Bisphenol-A (BPA), BPA glucuronide, and BPA sulfate in midgestation umbilical cord serum in a northern and central California population.Historical control data on reproductive abilities and incidences of spontaneous fetal malformations in Wistar Hannover GALAS rats.
P2860
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P2860
A physiologically based approach to the study of bisphenol A and other estrogenic chemicals on the size of reproductive organs, daily sperm production, and behavior.
description
1998 nî lūn-bûn
@nan
1998 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
A physiologically based approa ...... perm production, and behavior.
@ast
A physiologically based approa ...... perm production, and behavior.
@en
A physiologically based approa ...... perm production, and behavior.
@nl
type
label
A physiologically based approa ...... perm production, and behavior.
@ast
A physiologically based approa ...... perm production, and behavior.
@en
A physiologically based approa ...... perm production, and behavior.
@nl
prefLabel
A physiologically based approa ...... perm production, and behavior.
@ast
A physiologically based approa ...... perm production, and behavior.
@en
A physiologically based approa ...... perm production, and behavior.
@nl
P2093
P2860
P1476
A physiologically based approa ...... perm production, and behavior.
@en
P2093
D L Buchanan
F S vom Saal
K A Thayer
S Parmigiani
W V Welshons
P2860
P304
P356
10.1177/074823379801400115
P407
P577
1998-01-01T00:00:00Z