The septin-binding protein anillin is overexpressed in diverse human tumors.
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Cancer-Related Functions and Subcellular Localizations of SeptinsRegulation of distinct septin rings in a single cell by Elm1p and Gin4p kinases.Stabilization of the actomyosin ring enables spermatocyte cytokinesis in DrosophilaPCOPGene-Net: holistic characterisation of cellular states from microarray data based on continuous and non-continuous analysis of gene-expression relationships.Transcriptome sequencing identifies ANLN as a promising prognostic biomarker in bladder urothelial carcinoma.Identification of differentially expressed genes in uveal melanoma using suppressive subtractive hybridization.Septin 9 isoform expression, localization and epigenetic changes during human and mouse breast cancer progression.F-actin binding protein, anillin, regulates integrity of intercellular junctions in human epithelial cells.Conserved Senescence Associated Genes and Pathways in Primary Human Fibroblasts Detected by RNA-Seq.ANLN is a prognostic biomarker independent of Ki-67 and essential for cell cycle progression in primary breast cancer.The potent tumor suppressor miR-497 inhibits cancer phenotypes in nasopharyngeal carcinoma by targeting ANLN and HSPA4L.Majority of differentially expressed genes are down-regulated during malignant transformation in a four-stage modelThe G Protein-Coupled Estrogen Receptor Agonist G-1 Inhibits Nuclear Estrogen Receptor Activity and Stimulates Novel Phosphoproteomic SignaturesNCR-PCOPGene: An Exploratory Tool for Analysis of Sample-Classes Effect on Gene-Expression Relationships.SAP domain-dependent Mkl1 signaling stimulates proliferation and cell migration by induction of a distinct gene set indicative of poor prognosis in breast cancer patients.mRNA and microRNA expression patterns in adrenocortical cancer.Potential role of septins in oral carcinogenesis: An update and avenues for future research.Microtubules and actin crosstalk in cell migration and division.Comprehensive transcriptome profiling of squamous cell carcinoma of horn in Bos indicus.Regulation of cytokinesis and its clinical significance.Knockdown of ANLN by lentivirus inhibits cell growth and migration in human breast cancer.Anillin regulates cell-cell junction integrity by organizing junctional accumulation of Rho-GTP and actomyosin.Overexpression of Anillin (ANLN) is correlated with colorectal cancer progression and poor prognosis.Regulation of actin-binding protein ANLN by antitumor miR-217 inhibits cancer cell aggressiveness in pancreatic ductal adenocarcinoma.Subcellular localisation of anillin is associated with different survival outcomes in upper urinary tract urothelial carcinoma.A survey of immunohistochemical biomarkers for basal-like breast cancer against a gene expression profile gold standard.Identification of ANLN as ETV6 partner gene in recurrent t(7;12)(p15;p13): a possible role of deregulated ANLN expression in leukemogenesis.Knockdown of Anillin Actin Binding Protein Blocks Cytokinesis in Hepatocytes and Reduces Liver Tumor Development in Mice without Affecting Regeneration.Overexpression of ANLN contributed to poor prognosis of anthracycline-based chemotherapy in breast cancer patients.Anillin is required for tumor growth and regulated by miR-15a/miR-16-1 in HBV-related hepatocellular carcinoma
P2860
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P2860
The septin-binding protein anillin is overexpressed in diverse human tumors.
description
2005 nî lūn-bûn
@nan
2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
The septin-binding protein anillin is overexpressed in diverse human tumors.
@ast
The septin-binding protein anillin is overexpressed in diverse human tumors.
@en
The septin-binding protein anillin is overexpressed in diverse human tumors.
@en-gb
The septin-binding protein anillin is overexpressed in diverse human tumors.
@nl
type
label
The septin-binding protein anillin is overexpressed in diverse human tumors.
@ast
The septin-binding protein anillin is overexpressed in diverse human tumors.
@en
The septin-binding protein anillin is overexpressed in diverse human tumors.
@en-gb
The septin-binding protein anillin is overexpressed in diverse human tumors.
@nl
prefLabel
The septin-binding protein anillin is overexpressed in diverse human tumors.
@ast
The septin-binding protein anillin is overexpressed in diverse human tumors.
@en
The septin-binding protein anillin is overexpressed in diverse human tumors.
@en-gb
The septin-binding protein anillin is overexpressed in diverse human tumors.
@nl
P2093
P1476
The septin-binding protein anillin is overexpressed in diverse human tumors.
@en
P2093
Christopher B Todd
Kenneth J Hillan
Mit Dattani
Paula L Hyland
Peter A Hall
S E Hilary Russell
Simon S McDade
P304
P356
10.1158/1078-0432.CCR-05-0997
P407
P433
P577
2005-10-01T00:00:00Z