Transforming growth factor Beta 3 is required for excisional wound repair in vivo - Wikidata - awgldk - TriplyDB

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

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Palatogenesis and cutaneous repair: A two-headed coin Leptin promotes wound healing in the oral mucosa Associations Between TGFA/TGFB3/MSX1 Gene Polymorphisms and Congenital Non-Syndromic Hearing Impairment in a Chinese Population.Interferon regulatory factor 6 regulates keratinocyte migration.Transforming growth factor-β (TGF-β) expression is increased in the subsynovial connective tissue in a rabbit model of carpal tunnel syndrome.Digital imaging analysis to assess scar phenotype.Keratoconus in vitro and the key players of the TGF-β pathway.ALS skeletal muscle shows enhanced TGF-β signaling, fibrosis and induction of fibro/adipogenic progenitor markers.Toward an orofacial gene regulatory network.Epithelial-to-mesenchymal transition in cutaneous wound healing: Where we are and where we are heading.Transforming Growth Factor Beta Signaling in Cutaneous Wound Healing: Lessons Learned from Animal Studies.Current perspectives on biological approaches for osteoarthritis.Transforming growth factor beta (TGF-β) isoforms in wound healing and fibrosis.Platelet Rich Plasma: New Insights for Cutaneous Wound Healing Management.MiR-31 mediates inflammatory signaling to promote re-epithelialization during skin wound healing.Signalling by Transforming Growth Factor Beta Isoforms in Wound Healing and Tissue Regeneration.

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Transforming growth factor Beta 3 is required for excisional wound repair in vivo

http://www.wikidata.org/entity/Q34460888

description

2012 nî lūn-bûn

@nan

2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2012年の論文

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2012年論文

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2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@ast

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@en

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@nl

type

label

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@ast

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@en

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@nl

prefLabel

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@ast

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@en

Transforming growth factor Beta 3 is required for excisional wound repair in vivo

@nl

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JOURNAL.PONE.0048040

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Transforming growth factor Beta 3 is required for excisional wound repair in vivo

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Brian C Schutte

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Jasmine Morrison

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Leah C Biggs

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Lindsey Rhea

http://www.w3.org/2001/XMLSchema#string

Mark Le

http://www.w3.org/2001/XMLSchema#string

Martine Dunnwald

http://www.w3.org/2001/XMLSchema#string

Rachelle Naridze

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation and role of interleukin 6 in wounded human epithelial keratinocytes

Mammary serine protease inhibitor (Maspin) binds directly to interferon regulatory factor 6: identification of a novel serpin partnership

Abnormal skin, limb and craniofacial morphogenesis in mice deficient for interferon regulatory factor 6 (Irf6)

A "traffic control" role for TGFbeta3: orchestrating dermal and epidermal cell motility during wound healing

Generalized lacZ expression with the ROSA26 Cre reporter strain

Targeted disruption of the mouse transforming growth factor-beta 1 gene results in multifocal inflammatory disease

Prophylactic administration of avotermin for improvement of skin scarring: three double-blind, placebo-controlled, phase I/II studies

TGFbeta family signaling: novel insights in development and disease

Abnormal lung development and cleft palate in mice lacking TGF-beta 3 indicates defects of epithelial-mesenchymal interaction

Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response

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10.1371/JOURNAL.PONE.0048040

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2012-10-26T00:00:00Z

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3482237

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