Progression from high insulin resistance to type 2 diabetes does not entail additional visceral adipose tissue inflammation. - Wikidata - awgldk - TriplyDB

Progression from high insulin resistance to type 2 diabetes does not entail additional visceral adipose tissue inflammation.

Progression from high insulin resistance to type 2 diabetes does not entail additional visceral adipose tissue inflammation.

http://www.wikidata.org/entity/Q34461002

about

The Interactions of Obesity, Inflammation and Insulin Resistance in Breast Cancer Metabolic aspects of adult patients with nonalcoholic fatty liver disease Obesity and cancer: the role of adipose tissue and adipo-cytokines-induced chronic inflammation Pathophysiology and treatment of type 2 diabetes: perspectives on the past, present, and future.Alimentary regimen in non-alcoholic fatty liver disease: Mediterranean diet.Granulocyte colony-stimulating factor (G-CSF): A saturated fatty acid-induced myokine with insulin-desensitizing properties in humans.Can high altitude influence cytokines and sleep?Hydroxychloroquine hindering of diabetic isletopathy carries its signature on the inflammatory cytokines.Different response to hypoxia of adipose-derived multipotent cells from obese subjects with and without metabolic syndrome.Type 2 diabetes mellitus--an autoimmune disease?HMGB1 facilitates hypoxia-induced vWF upregulation through TLR2-MYD88-SP1 pathway.Abdominal subcutaneous and visceral adipocyte size, lipolysis and inflammation relate to insulin resistance in male obese humans.No Additive Effects of Polyphenol Supplementation and Exercise Training on White Adiposity Determinants of High-Fat Diet-Induced Obese Insulin-Resistant Rats.

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Progression from high insulin resistance to type 2 diabetes does not entail additional visceral adipose tissue inflammation.

http://www.wikidata.org/entity/Q34461002

description

2012 nî lūn-bûn

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2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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Progression from high insulin ...... al adipose tissue inflammation

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Francisco Jose Tinahones

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Lourdes Garrido-Sanchez

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Maria Dolores Mayas

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Maria Rosa Bernal-Lopez

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Nuria Barbarroja

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Rajaa El Bekay

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P2860

Insulin signalling and the regulation of glucose and lipid metabolism

Homeostasis model assessment: insulin resistance and ?-cell function from fasting plasma glucose and insulin concentrations in man

The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

Phosphorylation of Ser307 in insulin receptor substrate-1 blocks interactions with the insulin receptor and inhibits insulin action

STAT3 signalling is required for leptin regulation of energy balance but not reproduction

Clinical and pathophysiological consequences of abdominal adiposity and abdominal adipose tissue depots.

Glucose and fatty acids synergize to promote B-cell apoptosis through activation of glycogen synthase kinase 3β independent of JNK activation.

Selective inactivation of c-Jun NH2-terminal kinase in adipose tissue protects against diet-induced obesity and improves insulin sensitivity in both liver and skeletal muscle in mice.

Differential lipid partitioning between adipocytes and tissue macrophages modulates macrophage lipotoxicity and M2/M1 polarization in obese mice

Islet beta cell failure in type 2 diabetes

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scholarly article

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10.1371/JOURNAL.PONE.0048155

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Chary López-Pedrera

Wilfredo Oliva-Olivera

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2012-10-24T00:00:00Z

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232724940

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type-2 diabetes

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