The absence of mitochondrial thioredoxin 2 causes massive apoptosis, exencephaly, and early embryonic lethality in homozygous mice.
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Salmonella type III secretion effector SlrP is an E3 ubiquitin ligase for mammalian thioredoxinShort interfering RNA-mediated silencing of glutaredoxin 2 increases the sensitivity of HeLa cells toward doxorubicin and phenylarsine oxideEssential role for mitochondrial thioredoxin reductase in hematopoiesis, heart development, and heart functionStress induction and mitochondrial localization of Oxr1 proteins in yeast and humansIs the oxidative stress theory of aging dead?Thioredoxin and ventricular remodelingGeneration of reactive oxygen species in the anterior eye segment. Synergistic codrugs of N-acetylcarnosine lubricant eye drops and mitochondria-targeted antioxidant act as a powerful therapeutic platform for the treatment of cataracts and primary oMitochondrial Redox Signaling and Tumor ProgressionRedox modification of cell signaling in the cardiovascular systemRegulation of cell survival and death by pyridine nucleotidesOxr1 is essential for protection against oxidative stress-induced neurodegenerationAntioxidant activity of the yeast mitochondrial one-Cys peroxiredoxin is dependent on thioredoxin reductase and glutathione in vivo.NADPH oxidases: key modulators in aging and age-related cardiovascular diseases?Cross Talk between Two Antioxidant Systems, Thioredoxin and DJ-1: Consequences for CancerOxidative stress, unfolded protein response, and apoptosis in developmental toxicityMitochondria: Much ado about nothing? How dangerous is reactive oxygen species production?Redox regulation of mitochondrial functionThioredoxins, glutaredoxins, and peroxiredoxins--molecular mechanisms and health significance: from cofactors to antioxidants to redox signalingParadoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health ImplicationsThe effects of chromium(VI) on the thioredoxin system: implications for redox regulationDepletion of Plasmodium berghei plasmoredoxin reveals a non-essential role for life cycle progression of the malaria parasiteCytoplasmic thioredoxin reductase is essential for embryogenesis but dispensable for cardiac developmentMammalian thioredoxin reductase 1: roles in redox homoeostasis and characterization of cellular targetsA HIF-1 target, ATIA, protects cells from apoptosis by modulating the mitochondrial thioredoxin, TRX2Effects of thioredoxin reductase-1 deletion on embryogenesis and transcriptomeMitochondrial function and redox control in the aging eye: role of MsrA and other repair systems in cataract and macular degenerationsEffects of delayed NSAID administration after experimental eccentric contraction injury – A cellular and proteomics studyMitochondrial peptidase IMMP2L mutation causes early onset of age-associated disorders and impairs adult stem cell self-renewalThe characterization of the Caenorhabditis elegans mitochondrial thioredoxin system uncovers an unexpected protective role of thioredoxin reductase 2 in β-amyloid peptide toxicity.Control of mitochondrial outer membrane permeabilization and Bcl-xL levels by thioredoxin 2 in DT40 cells.The role of thioredoxin reductases in brain development.Overexpression of thioredoxins 1 and 2 increases retinal ganglion cell survival after pharmacologically induced oxidative stress, optic nerve transection, and in experimental glaucoma.Update on the oxidative stress theory of aging: does oxidative stress play a role in aging or healthy aging?Redox regulation of cell survival by the thioredoxin superfamily: an implication of redox gene therapy in the heart.A mammalian monothiol glutaredoxin, Grx3, is critical for cell cycle progression during embryogenesis.The Role of NOX4 and TRX2 in Angiogenesis and Their Potential Cross-Talk.Deficient Vitamin E Uptake During Development Impairs Neural Tube Closure in Mice Lacking Lipoprotein Receptor SR-BI.Glutaredoxin 2 reduces both thioredoxin 2 and thioredoxin 1 and protects cells from apoptosis induced by auranofin and 4-hydroxynonenal.Hepatocytes lacking thioredoxin reductase 1 have normal replicative potential during development and regeneration.Respiration-dependent H2O2 removal in brain mitochondria via the thioredoxin/peroxiredoxin system.
P2860
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P2860
The absence of mitochondrial thioredoxin 2 causes massive apoptosis, exencephaly, and early embryonic lethality in homozygous mice.
description
2003 nî lūn-bûn
@nan
2003 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
The absence of mitochondrial t ...... lethality in homozygous mice.
@ast
The absence of mitochondrial t ...... lethality in homozygous mice.
@en
The absence of mitochondrial t ...... lethality in homozygous mice.
@nl
type
label
The absence of mitochondrial t ...... lethality in homozygous mice.
@ast
The absence of mitochondrial t ...... lethality in homozygous mice.
@en
The absence of mitochondrial t ...... lethality in homozygous mice.
@nl
prefLabel
The absence of mitochondrial t ...... lethality in homozygous mice.
@ast
The absence of mitochondrial t ...... lethality in homozygous mice.
@en
The absence of mitochondrial t ...... lethality in homozygous mice.
@nl
P2093
P2860
P1476
The absence of mitochondrial t ...... lethality in homozygous mice.
@en
P2093
Larisa Nonn
Robert P Erickson
Ryan R Williams
P2860
P304
P356
10.1128/MCB.23.3.916-922.2003
P407
P50
P577
2003-02-01T00:00:00Z