Delayed conduction and its implications in murine Scn5a(+/-) hearts: independent and interacting effects of genotype, age, and sex - Wikidata - awgldk - TriplyDB

Delayed conduction and its implications in murine Scn5a(+/-) hearts: independent and interacting effects of genotype, age, and sex

Delayed conduction and its implications in murine Scn5a(+/-) hearts: independent and interacting effects of genotype, age, and sex

http://www.wikidata.org/entity/Q34467730

about

Novel trigenic CACNA1C/DES/MYPN mutations in a family of hypertrophic cardiomyopathy with early repolarization and short QT syndrome.Arrhythmic substrate, slowed propagation and increased dispersion in conduction direction in the right ventricular outflow tract of murine Scn5a+/- hearts.Nonlinearity between action potential alternans and restitution, which both predict ventricular arrhythmic properties in Scn5a+/- and wild-type murine hearts Frequency distribution analysis of activation times and regional fibrosis in murine Scn5a+/- hearts: the effects of ageing and sex Action potential wavelength restitution predicts alternans and arrhythmia in murine Scn5a(+/-) hearts.Pathophysiological Mechanisms of Sino-Atrial Dysfunction and Ventricular Conduction Disease Associated with SCN5A Deficiency: Insights from Mouse Models.Sodium channel haploinsufficiency and structural change in ventricular arrhythmogenesis.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Electrophysiological characterization of a large set of novel variants in the SCN5A-gene: identification of novel LQTS3 and BrS mutations.The effects of ageing and adrenergic challenge on electrocardiographic phenotypes in a murine model of long QT syndrome type 3 Age-dependent atrial arrhythmic phenotype secondary to mitochondrial dysfunction in Pgc-1β deficient murine hearts.Age-dependent electrocardiographic changes in Pgc-1β deficient murine hearts.Cardiomyocyte ionic currents in intact young and aged murine Pgc-1β-/- atrial preparations.Loss of Nav1.5 expression and function in murine atria containing the RyR2-P2328S gain-of-function mutation.Ventricular pro-arrhythmic phenotype, arrhythmic substrate, ageing and mitochondrial dysfunction in peroxisome proliferator activated receptor-γ coactivator-1β deficient (Pgc-1β-/-) murine hearts.

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P2860

Delayed conduction and its implications in murine Scn5a(+/-) hearts: independent and interacting effects of genotype, age, and sex

http://www.wikidata.org/entity/Q34467730

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Delayed conduction and its imp ...... ects of genotype, age, and sex

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Pfluegers Archiv

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Delayed conduction and its imp ...... ects of genotype, age, and sex

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Andrew A Grace

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Christopher L-H Huang

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Kamalan Jeevaratnam

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Laila Guzadhur

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Rebecca Rewbury

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Sui Poh Tee

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Yanmin Zhang

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P2860

Mouse model of SCN5A-linked hereditary Lenègre's disease: age-related conduction slowing and myocardial fibrosis

Slowed conduction and ventricular tachycardia after targeted disruption of the cardiac sodium channel gene Scn5a.

Impaired impulse propagation in Scn5a-knockout mice: combined contribution of excitability, connexin expression, and tissue architecture in relation to aging

"Brugada" syndrome: clinical data and suggested pathophysiological mechanism.

Novel SCN5A mutation leading either to isolated cardiac conduction defect or Brugada syndrome in a large French family.

Pathophysiological mechanisms of Brugada syndrome: depolarization disorder, repolarization disorder, or more?

Fragmented QRS as a marker of conduction abnormality and a predictor of prognosis of Brugada syndrome.

Variable Na(v)1.5 protein expression from the wild-type allele correlates with the penetrance of cardiac conduction disease in the Scn5a(+/-) mouse model

Cellular basis for the Brugada syndrome and other mechanisms of arrhythmogenesis associated with ST-segment elevation.

Paced ventricular electrogram fractionation and sudden death in hypertrophic cardiomyopathy and other non-coronary heart diseases.

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s00424-010-0906-1

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29-44

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1

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Rudolf Martin Duehmke

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2010-12-03T00:00:00Z

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