Bmi1 regulates self-renewal and epithelial to mesenchymal transition in breast cancer cells through Nanog.
about
TWIST1 and BMI1 in Cancer Metastasis and ChemoresistanceYin Yang 1 is associated with cancer stem cell transcription factors (SOX2, OCT4, BMI1) and clinical implicationPolycomb Group (PcG) Proteins and Human Cancers: Multifaceted Functions and Therapeutic ImplicationsThe Combination of Three Natural Compounds Effectively Prevented Lung Carcinogenesis by Optimal Wound HealingRole of the Drug Transporter ABCC3 in Breast Cancer ChemoresistanceExpression of embryonal stem cell transcription factors in breast cancer: Oct4 as an indicator for poor clinical outcome and tamoxifen resistance.Regulation of NANOG in cancer cells.The role of polycomb group protein Bmi-1 and Notch4 in breast cancer stem cell inhibition by benzyl isothiocyanate.Long noncoding RNA MALAT-1 enhances stem cell-like phenotypes in pancreatic cancer cellsParacrine factors from adipose-mesenchymal stem cells enhance metastatic capacity through Wnt signaling pathway in a colon cancer cell co-culture model.MicroRNA-218 inhibits the proliferation and metastasis of esophageal squamous cell carcinoma cells by targeting BMI1.Low adherent cancer cell subpopulations are enriched in tumorigenic and metastatic epithelial-to-mesenchymal transition-induced cancer stem-like cells.The role of the aryl hydrocarbon receptor in the development of cells with the molecular and functional characteristics of cancer stem-like cells.53BP1 suppresses epithelial-mesenchymal transition by downregulating ZEB1 through microRNA-200b/429 in breast cancer.Bmi-1: At the crossroads of physiological and pathological biology.miR-200c/Bmi1 axis and epithelial-mesenchymal transition contribute to acquired resistance to BRAF inhibitor treatment.Dysregulation of Bmi1 promotes malignant transformation of hepatic progenitor cellsTumor-Initiating Cells: a criTICal review of isolation approaches and new challenges in targeting strategies.Gene of the month: NANOG.Perspective: Cooperation of Nanog, NF-κΒ, and CXCR4 in a regulatory network for directed migration of cancer stem cells.miR-15a/miR-16 down-regulates BMI1, impacting Ub-H2A mediated DNA repair and breast cancer cell sensitivity to doxorubicinGaleterone and VNPT55 disrupt Mnk-eIF4E to inhibit prostate cancer cell migration and invasion.miR-376c inhibits cervical cancer cell proliferation and invasion by targeting BMI1.Enhanced SLC34A2 in breast cancer stem cell-like cells induces chemotherapeutic resistance to doxorubicin via SLC34A2-Bmi1-ABCC5 signaling.NANOG overexpression and its correlation with stem cell and differentiation markers in meningiomas of different WHO grades.Cell lineage determinants as regulators of breast cancer metastasis.Ectopic overexpression of Nanog induces tumorigenesis in non-tumorous fibroblasts.Upregulation of CYP17A1 by Sp1-mediated DNA demethylation confers temozolomide resistance through DHEA-mediated protection in glioma.VSV based virotherapy in ovarian cancer: the past, the present and …future?Transgenic overexpression of NanogP8 in the mouse prostate is insufficient to initiate tumorigenesis but weakly promotes tumor development in the Hi-Myc mouse model.Hsp90α Mediates BMI1 Expression in Breast Cancer Stem/Progenitor Cells through Facilitating Nuclear Translocation of c-Myc and EZH2.Inhibition of FOXC2 restores epithelial phenotype and drug sensitivity in prostate cancer cells with stem-cell propertiesEpigenetic Mechanisms of Tamoxifen Resistance in Luminal Breast Cancer.Cancer cell reprogramming to identify the genes competent for generating liver cancer stem cells.Carnosol controls the human glioblastoma stemness features through the epithelial-mesenchymal transition modulation and the induction of cancer stem cell apoptosis.BMI1 regulates androgen receptor in prostate cancer independently of the polycomb repressive complex 1.Regulating BMI1 expression via miRNAs promote Mesenchymal to Epithelial Transition (MET) and sensitizes breast cancer cell to chemotherapeutic drug.Role of miRNA dynamics and cytokine profile in governing CD44v6/Nanog/PTEN axis in oral cancer: modulating the master regulators.Embryonic stem cells markers Oct4 and Nanog correlate with perineural invasion in human salivary gland mucoepidermoid carcinoma.Reduced SLC27A2 induces cisplatin resistance in lung cancer stem cells by negatively regulating Bmi1-ABCG2 signaling.
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P2860
Bmi1 regulates self-renewal and epithelial to mesenchymal transition in breast cancer cells through Nanog.
description
2014 nî lūn-bûn
@nan
2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@ast
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@en
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@nl
type
label
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@ast
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@en
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@nl
prefLabel
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@ast
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@en
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@nl
P2093
P2860
P356
P1433
P1476
Bmi1 regulates self-renewal an ...... st cancer cells through Nanog.
@en
P2093
Annapoorni Rangarajan
Anurag N Paranjape
Esthelin Vittal Krushik
Geetashree Mukherjee
Pradeep Nagaraj
Sai A Balaji
Tamoghna Mandal
P2860
P2888
P356
10.1186/1471-2407-14-785
P407
P577
2014-10-28T00:00:00Z
P5875
P6179
1042341975