Secondary degeneration reduced by inosine after spinal cord injury in rats. - Wikidata - awgldk - TriplyDB

Secondary degeneration reduced by inosine after spinal cord injury in rats.

Secondary degeneration reduced by inosine after spinal cord injury in rats.

http://www.wikidata.org/entity/Q34471414

about

Homeostatic imbalance of purine catabolism in first-episode neuroleptic-naïve patients with schizophrenia Current and future medical therapeutic strategies for the functional repair of spinal cord injury Antioxidants, redox signaling, and pathophysiology in schizophrenia: an integrative view.Inosine alters gene expression and axonal projections in neurons contralateral to a cortical infarct and improves skilled use of the impaired limb.Associations between purine metabolites and monoamine neurotransmitters in first-episode psychosis.Inosine enhances axon sprouting and motor recovery after spinal cord injury.A systematic review of non-invasive pharmacologic neuroprotective treatments for acute spinal cord injury.Inosine Improves Neurogenic Detrusor Overactivity following Spinal Cord Injury An overview of pharmacological approaches for management and repair of spinal cord injuries.Inosine induces presynaptic inhibition of acetylcholine release by activation of A3 adenosine receptors at the mouse neuromuscular junction.The antidepressant-like effect of inosine in the FST is associated with both adenosine A1 and A 2A receptors.Expanding the molecular recognition repertoire of antifreeze polypeptides: effects on nucleoside crystal growth

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P2860

Secondary degeneration reduced by inosine after spinal cord injury in rats.

http://www.wikidata.org/entity/Q34471414

description

2005 nî lūn-bûn

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2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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type

label

Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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Secondary degeneration reduced by inosine after spinal cord injury in rats.

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P2860

Inosine induces axonal rewiring and improves behavioral outcome after stroke.

Uric acid, a natural scavenger of peroxynitrite, in experimental allergic encephalomyelitis and multiple sclerosis

Pathogenesis and pharmacological strategies for mitigating secondary damage in acute spinal cord injury.

Advances in secondary spinal cord injury: role of apoptosis.

Administration of methylprednisolone for 24 or 48 hours or tirilazad mesylate for 48 hours in the treatment of acute spinal cord injury. Results of the Third National Acute Spinal Cord Injury Randomized Controlled Trial. National Acute Spinal Cord I

Immunomodulatory and neuroprotective effects of inosine.

Inosine stimulates extensive axon collateral growth in the rat corticospinal tract after injury

Review of the secondary injury theory of acute spinal cord trauma with emphasis on vascular mechanisms.

Methylprednisolone in spinal cord injury: the possible mechanism of action.

Purines inhibit poly(ADP-ribose) polymerase activation and modulate oxidant-induced cell death.

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10.1038/SJ.SC.3101878

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