Predominant role of endothelial nitric oxide synthase in vascular endothelial growth factor-induced angiogenesis and vascular permeability. - Wikidata - awgldk - TriplyDB

Predominant role of endothelial nitric oxide synthase in vascular endothelial growth factor-induced angiogenesis and vascular permeability.

Predominant role of endothelial nitric oxide synthase in vascular endothelial growth factor-induced angiogenesis and vascular permeability.

http://www.wikidata.org/entity/Q34478521

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PI3K/AKT/mTOR Pathway in Angiogenesis Thrombospondin-1 inhibits endothelial cell responses to nitric oxide in a cGMP-dependent manner Heme oxygenase-1 modulates the expression of the anti-angiogenic chemokine CXCL-10 in renal tubular epithelial cells Low-dose irradiation promotes tissue revascularization through VEGF release from mast cells and MMP-9-mediated progenitor cell mobilization Aberrant Pulmonary Vascular Growth and Remodeling in Bronchopulmonary Dysplasia Mesenchymal Stem/Stromal Cells in Stromal Evolution and Cancer Progression Nitric oxide synthases, S-nitrosylation and cardiovascular health: from molecular mechanisms to therapeutic opportunities (review)Signaling regulation of fetoplacental angiogenesis Renal endothelial dysfunction in diabetic nephropathy Vitreous mediators in retinal hypoxic diseases The critical role of Akt in cardiovascular function Vascular permeability--the essentials CD47 is necessary for inhibition of nitric oxide-stimulated vascular cell responses by thrombospondin-1 CYP1B1 and endothelial nitric oxide synthase combine to sustain proangiogenic functions of endothelial cells under hyperoxic stress New Delivery Systems of Stem Cells for Vascular Regeneration in Ischemia VEGFR2 induces c-Src signaling and vascular permeability in vivo via the adaptor protein TSAd.Normalization of the vasculature for treatment of cancer and other diseases.The release of nitric oxide from S-nitrosothiols promotes angiogenesis Repetitive H-wave device stimulation and program induces significant increases in the range of motion of post operative rotator cuff reconstruction in a double-blinded randomized placebo controlled human study.Molecular regulation of tumor angiogenesis and perfusion via redox signaling.Molecular dialogs between the ischemic brain and the peripheral immune system: dualistic roles in injury and repair Recapitulating physiological and pathological shear stress and oxygen to model vasculature in health and disease.Circulating VEGF and eNOS variations as predictors of outcome in metastatic colorectal cancer patients receiving bevacizumab.Rapid development of hypertension and proteinuria with cediranib, an oral vascular endothelial growth factor receptor inhibitor.Measuring Vascular Permeability In Vivo Suppression of Transforming Growth Factor-β Signaling Delays Cellular Senescence and Preserves the Function of Endothelial Cells Derived from Human Pluripotent Stem Cells.eNOS transfection of adipose-derived stem cells yields bioactive nitric oxide production and improved results in vascular tissue engineering Endothelial nitric oxide synthase activation is critical for vascular leakage during acute inflammation in vivo.The role of tumour-derived iNOS in tumour progression and angiogenesis Automated computation of functional vascular density using laser speckle imaging in a rodent window chamber model.Syntenin promotes VEGF-induced VEGFR2 endocytosis and angiogenesis by increasing ephrin-B2 function in endothelial cells.Endothelial nitric oxide synthase is critical for ischemic remodeling, mural cell recruitment, and blood flow reserve.Molecular and cellular basis of viable dysfunctional myocardium.The NO cascade, eNOS location, and microvascular permeability.Deciphering mechanisms controlling placental artery endothelial cell migration stimulated by vascular endothelial growth factor.Effect of IBD sera on expression of inducible and endothelial nitric oxide synthase in human umbilical vein endothelial cells.Small molecule screening in zebrafish: an in vivo approach to identifying new chemical tools and drug leads.Targeting the mammalian target of Rapamycin to inhibit VEGF and cytokines for the treatment of primary effusion lymphoma.Hyperbaric oxygen, vasculogenic stem cells, and wound healing.Prenatal hypoxic-ischemic insult changes the distribution and number of NADPH-diaphorase cells in the cerebellum.

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Predominant role of endothelial nitric oxide synthase in vascular endothelial growth factor-induced angiogenesis and vascular permeability.

http://www.wikidata.org/entity/Q34478521

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2001 nî lūn-bûn

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Predominant role of endothelia ...... sis and vascular permeability.

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Predominant role of endothelia ...... sis and vascular permeability.

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Predominant role of endothelia ...... sis and vascular permeability.

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Predominant role of endothelia ...... sis and vascular permeability.

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Predominant role of endothelia ...... sis and vascular permeability.

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Predominant role of endothelia ...... sis and vascular permeability.

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Predominant role of endothelia ...... sis and vascular permeability.

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Predominant role of endothelia ...... sis and vascular permeability.

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Proceedings of the National Academy of Sciences of the United States of America

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Predominant role of endothelia ...... sis and vascular permeability.

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A Kadambi

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D Fukumura

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P2860

Vascular endothelial growth factor regulates endothelial cell survival through the phosphatidylinositol 3'-kinase/Akt signal transduction pathway. Requirement for Flk-1/KDR activation

VEGF-A induces expression of eNOS and iNOS in endothelial cells via VEGF receptor-2 (KDR)

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

Neuropilin-1 is expressed by endothelial and tumor cells as an isoform-specific receptor for vascular endothelial growth factor

Migration of human monocytes in response to vascular endothelial growth factor (VEGF) is mediated via the VEGF receptor flt-1

Angiogenesis in cancer and other diseases

Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

Nitric oxide synthase modulates angiogenesis in response to tissue ischemia

Cardiovascular failure in mouse embryos deficient in VEGF receptor-3

RAG-1-deficient mice have no mature B and T lymphocytes

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2604-2609

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10.1073/PNAS.041359198

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98

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11226286

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30185

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