Relationship between Fibroblast Growth Factor-23 and Mineral Metabolism in Chronic Kidney Disease - Wikidata - awgldk - TriplyDB

Relationship between Fibroblast Growth Factor-23 and Mineral Metabolism in Chronic Kidney Disease

Relationship between Fibroblast Growth Factor-23 and Mineral Metabolism in Chronic Kidney Disease

http://www.wikidata.org/entity/Q34479134

about

Magnesium and FGF-23 are independent predictors of pulse pressure in pre-dialysis diabetic chronic kidney disease patients.Low Magnesium Levels and FGF-23 Dysregulation Predict Mitral Valve Calcification as well as Intima Media Thickness in Predialysis Diabetic Patients Fibroblast growth factor-23 helps explain the biphasic cardiovascular effects of vitamin D in chronic kidney disease.Paricalcitol- or cinacalcet-centred therapy affects markers of bone mineral disease in patients with secondary hyperparathyroidism receiving haemodialysis: results of the IMPACT-SHPT study.

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Relationship between Fibroblast Growth Factor-23 and Mineral Metabolism in Chronic Kidney Disease

http://www.wikidata.org/entity/Q34479134

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Relationship between Fibroblas ...... lism in Chronic Kidney Disease

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Relationship between Fibroblas ...... lism in Chronic Kidney Disease

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Relationship between Fibroblas ...... lism in Chronic Kidney Disease

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Relationship between Fibroblas ...... lism in Chronic Kidney Disease

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Relationship between Fibroblas ...... lism in Chronic Kidney Disease

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International Journal of Nephrology

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Relationship between Fibroblas ...... lism in Chronic Kidney Disease

@en

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Kosaku Nitta

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P2860

FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis

Mutation of the mouse klotho gene leads to a syndrome resembling ageing

Phosphate regulation of vascular smooth muscle cell calcification

Identification of a novel fibroblast growth factor, FGF-23, preferentially expressed in the ventrolateral thalamic nucleus of the brain

The autosomal dominant hypophosphatemic rickets R176Q mutation in fibroblast growth factor 23 resists proteolytic cleavage and enhances in vivo biological potency

Klotho, a gene related to a syndrome resembling human premature aging, functions in a negative regulatory circuit of vitamin D endocrine system

Role of the sodium-dependent phosphate cotransporter, Pit-1, in vascular smooth muscle cell calcification

Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice

Effect of hydrolysis-resistant FGF23-R179Q on dietary phosphate regulation of the renal type-II Na/Pi transporter

Regulation of fibroblast growth factor-23 signaling by klotho

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167984

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scholarly article

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10.4061/2010/167984

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2010

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2010-12-30T00:00:00Z

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49754788

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21234310

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3018630

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