Gene expression changes associated with progression and response in chronic myeloid leukemia. - Wikidata - awgldk - TriplyDB

Gene expression changes associated with progression and response in chronic myeloid leukemia.

Gene expression changes associated with progression and response in chronic myeloid leukemia.

http://www.wikidata.org/entity/Q34479561

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Musashi-2 regulates normal hematopoiesis and promotes aggressive myeloid leukemia Regulation of myeloid leukaemia by the cell-fate determinant Musashi Biomarkers for determining the prognosis in chronic myelogenous leukemia Signal transduction in the chronic leukemias: implications for targeted therapies miRNAs as Biomarkers in Chronic Myelogenous Leukemia LSD1 inhibition: a therapeutic strategy in cancer?ATRA-induced cellular differentiation and CD38 expression inhibits acquisition of BCR-ABL mutations for CML acquired resistance Nanotechnology and cancer.A bead-based activity screen for small-molecule inhibitors of signal transduction in chronic myelogenous leukemia cells.DNA-encoded antibody libraries: a unified platform for multiplexed cell sorting and detection of genes and proteins.Dasatinib in the treatment of chronic myeloid leukemia in accelerated phase after imatinib failure: the START a trial.The derivation of diagnostic markers of chronic myeloid leukemia progression from microarray data.The use of knockout mice reveals a synergistic role of the Vav1 and Rasgrf2 gene deficiencies in lymphomagenesis and metastasis Human cancers express mutator phenotypes: origin, consequences and targeting.Immune Effector Recovery in Chronic Myeloid Leukemia and Treatment-Free Remission.A gene expression signature of CD34+ cells to predict major cytogenetic response in chronic-phase chronic myeloid leukemia patients treated with imatinib.Methylation pattern of preferentially expressed antigen of melanoma in acute myeloid leukemia and myelodysplastic syndromes.Biological and clinical significance of GSK-3beta in mantle cell lymphoma--an immunohistochemical study.High-avidity cytotoxic T lymphocytes specific for a new PRAME-derived peptide can target leukemic and leukemic-precursor cells The mutator phenotype in cancer: molecular mechanisms and targeting strategies Loss of the Alox5 gene impairs leukemia stem cells and prevents chronic myeloid leukemia.CBP/catenin antagonist safely eliminates drug-resistant leukemia-initiating cells.Chronic myeloid leukemia: mechanisms of blastic transformation Association between imatinib-resistant BCR-ABL mutation-negative leukemia and persistent activation of LYN kinase.Wnt/Ca2+/NFAT signaling maintains survival of Ph+ leukemia cells upon inhibition of Bcr-Abl.SL-401 and SL-501, targeted therapeutics directed at the interleukin-3 receptor, inhibit the growth of leukaemic cells and stem cells in advanced phase chronic myeloid leukaemia.Arachidonate 15-lipoxygenase is required for chronic myeloid leukemia stem cell survival.Predicting relapse prior to transplantation in chronic myeloid leukemia by integrating expert knowledge and expression data A therapeutically targetable mechanism of BCR-ABL-independent imatinib resistance in chronic myeloid leukemia.Wnt/β-catenin pathway regulates ABCB1 transcription in chronic myeloid leukemia.Two clinical phenotypes in polycythemia vera.Characterization of chronic myeloid leukemia stem cells.Combined Population Dynamics and Entropy Modelling Supports Patient Stratification in Chronic Myeloid Leukemia NCCN Task Force report: tyrosine kinase inhibitor therapy selection in the management of patients with chronic myelogenous leukemia Expression and activity of Fyn mediate proliferation and blastic features of chronic myelogenous leukemia.A novel Bcr-Abl-mTOR-eIF4A axis regulates IRES-mediated translation of LEF-1 Identifying and validating a combined mRNA and microRNA signature in response to imatinib treatment in a chronic myeloid leukemia cell line.Regulation of CDX4 gene transcription by HoxA9, HoxA10, the Mll-Ell oncogene and Shp2 during leukemogenesis Wnt/catenin signaling in adult stem cell physiology and disease.Functionally deregulated AML1/RUNX1 cooperates with BCR-ABL to induce a blastic phase-like phenotype of chronic myelogenous leukemia in mice.

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P2860

Gene expression changes associated with progression and response in chronic myeloid leukemia.

http://www.wikidata.org/entity/Q34479561

description

2006 nî lūn-bûn

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2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2006 թվականի փետրվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Proceedings of the National Academy of Sciences of the United States of America

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Gene expression changes associ ...... e in chronic myeloid leukemia.

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Charles Sawyers

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Cheryl L Willman

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Hongyue Dai

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Neil Shah

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Vivian Oehler

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Wendy Stock

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P2860

The complete coding sequence of arg defines the Abelson subfamily of cytoplasmic tyrosine kinases

I-mfa domain proteins interact with Axin and affect its regulation of the Wnt and c-Jun N-terminal kinase signaling pathways.

A retinoic acid-responsive human zinc finger gene, MZF-1, preferentially expressed in myeloid cells

Sp1/Sp3 and the myeloid zinc finger gene MZF1 regulate the human N-cadherin promoter in osteoblasts

I-mf, a novel myogenic repressor, interacts with members of the MyoD family

The human tumor antigen PRAME is a dominant repressor of retinoic acid receptor signaling

Snail and SIP1 increase cancer invasion by upregulating MMP family in hepatocellular carcinoma cells

Identification of a melanoma antigen, PRAME, as a BCR/ABL-inducible gene

Quantitative monitoring of the PRAME gene for the detection of minimal residual disease in leukaemia

Snail induction of epithelial to mesenchymal transition in tumor cells is accompanied by MUC1 repression and ZEB1 expression

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Brian J. Druker

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7300024

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chronic myeloid leukemia

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1413797

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