Cognitive decline is associated with reduced reelin expression in the entorhinal cortex of aged rats - Wikidata - awgldk - TriplyDB

Cognitive decline is associated with reduced reelin expression in the entorhinal cortex of aged rats

Cognitive decline is associated with reduced reelin expression in the entorhinal cortex of aged rats

http://www.wikidata.org/entity/Q34488109

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The involvement of Reelin in neurodevelopmental disorders.Reelin Proteolysis Affects Signaling Related to Normal Synapse Function and Neurodegeneration Aging in the olfactory system Molecular drivers and cortical spread of lateral entorhinal cortex dysfunction in preclinical Alzheimer's disease Cortical gene transcription response patterns to water maze training in aged mice Selective vulnerability of neurons in layer II of the entorhinal cortex during aging and Alzheimer's disease.Reelin Signaling, Hippocampal Neurogenesis, and Efficacy of Aspirin Intake & Stem Cell Transplantation in Aging and Alzheimer's disease.Epigenetic contributions to cognitive aging: disentangling mindspan and lifespan.Obesity elicits interleukin 1-mediated deficits in hippocampal synaptic plasticity Lowering corticosterone levels reinstates hippocampal brain-derived neurotropic factor and Trkb expression without influencing deficits in hypothalamic brain-derived neurotropic factor expression in leptin receptor-deficient mice.Age-related memory deficits linked to circuit-specific disruptions in the hippocampus Interference with reelin signaling in the lateral entorhinal cortex impairs spatial memory.Response of the medial temporal lobe network in amnestic mild cognitive impairment to therapeutic intervention assessed by fMRI and memory task performance Perinatal α-linolenic acid availability alters the expression of genes related to memory and to epigenetic machinery, and the Mecp2 DNA methylation in the whole brain of mouse offspring.Hippocampal expression of myelin-associated inhibitors is induced with age-related cognitive decline and correlates with deficits of spatial learning and memory.Visual steady state in relation to age and cognitive function.Senescent-induced dysregulation of cAMP/CREB signaling and correlations with cognitive decline.Reelin immunoreactivity in neuritic varicosities in the human hippocampal formation of non-demented subjects and Alzheimer's disease patients.Neurocognitive Aging and the Hippocampus across Species.Aging reduces total neuron number in the dorsal component of the rodent prefrontal cortex Spatial discrimination deficits as a function of mnemonic interference in aged adults with and without memory impairment.Reelin signaling in development, maintenance, and plasticity of neural networks Neurotoxic saboteurs: straws that break the hippo's (hippocampus) back drive cognitive impairment and Alzheimer's Disease.The entorhinal cortex and neurotrophin signaling in Alzheimer's disease and other disorders.The regulation of pituitary-thyroid abnormalities by Peripheral administration of Levothyroxine increased BDNF and Reelin proteins expression in an animal model of Alzheimer's disease.Selective vulnerabilities and biomarkers in neurocognitive aging.Targeting Neural Hyperactivity as a Treatment to Stem Progression of Late-Onset Alzheimer's Disease.Reelin in Alzheimer's Disease, Increased Levels but Impaired Signaling: When More is Less.Reelin Expression in Creutzfeldt-Jakob Disease and Experimental Models of Transmissible Spongiform Encephalopathies.Positivity effect specific to older adults with subclinical memory impairment Age-related individual variability in memory performance is associated with amygdala-hippocampal circuit function and emotional pattern separation.Greater loss of object than spatial mnemonic discrimination in aged adults.Ground zero in Alzheimer's disease.Integrating new findings and examining clinical applications of pattern separation.Passive Double-Sensory Evoked Coherence Correlates with Long-Term Memory Capacity.Selective plasticity of hippocampal GABAergic interneuron populations following kindling of different brain regions.Functional Imbalance of Anterolateral Entorhinal Cortex and Hippocampal Dentate/CA3 Underlies Age-Related Object Pattern Separation Deficits.Glucocorticoid-mediated activation of GSK3β promotes tau phosphorylation and impairs memory in type 2 diabetes.

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Cognitive decline is associated with reduced reelin expression in the entorhinal cortex of aged rats

http://www.wikidata.org/entity/Q34488109

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Cerebral Cortex

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Cognitive decline is associate ...... entorhinal cortex of aged rats

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Alexis M Stranahan

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Michela Gallagher

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Rebecca P Haberman

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P2860

Direct binding of Reelin to VLDL receptor and ApoE receptor 2 induces tyrosine phosphorylation of disabled-1 and modulates tau phosphorylation

Protein kinase MARK/PAR-1 is required for neurite outgrowth and establishment of neuronal polarity.

The Reelin signaling pathway promotes dendritic spine development in hippocampal neurons

Reelin signaling antagonizes beta-amyloid at the synapse

Interaction of reelin with amyloid precursor protein promotes neurite outgrowth

Extracellular matrix molecules and synaptic plasticity: immunomapping of intracellular and secreted Reelin in the adult rat brain

Reelin regulates postnatal neurogenesis and enhances spine hypertrophy and long-term potentiation

Hyperphosphorylated tau in parahippocampal cortex impairs place learning in aged mice expressing wild-type human tau

Rapid encoding of new information alters the profile of plasticity-related mRNA transcripts in the hippocampal CA3 region

Neurocognitive aging: prior memories hinder new hippocampal encoding.

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392-400

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10.1093/CERCOR/BHQ106

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2

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21

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2010-06-10T00:00:00Z

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44664221

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20538740

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3020582

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