Strong cross-bridges potentiate the Ca(2+) affinity changes produced by hypertrophic cardiomyopathy cardiac troponin C mutants in myofilaments: a fast kinetic approach.
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Cardiac Troponin and Tropomyosin: Structural and Cellular Perspectives to Unveil the Hypertrophic Cardiomyopathy PhenotypeA mutation in TNNC1-encoded cardiac troponin C, TNNC1-A31S, predisposes to hypertrophic cardiomyopathy and ventricular fibrillationStructure and function of cardiac troponin C (TNNC1): Implications for heart failure, cardiomyopathies, and troponin modulating drugsHypertrophic Cardiomyopathy Cardiac Troponin C Mutations Differentially Affect Slow Skeletal and Cardiac Muscle Regulation.Amide hydrogens reveal a temperature-dependent structural transition that enhances site-II Ca2+-binding affinity in a C-domain mutant of cardiac troponin CEffects of troponin T cardiomyopathy mutations on the calcium sensitivity of the regulated thin filament and the actomyosin cross-bridge kinetics of human β-cardiac myosin.In Vivo Analysis of Troponin C Knock-In (A8V) Mice: Evidence that TNNC1 Is a Hypertrophic Cardiomyopathy Susceptibility GeneEnhanced troponin I binding explains the functional changes produced by the hypertrophic cardiomyopathy mutation A8V of cardiac troponin CThe myosin-activated thin filament regulatory state, M⁻-open: a link to hypertrophic cardiomyopathy (HCM).Biochemical characterisation of Troponin C mutations causing hypertrophic and dilated cardiomyopathies.Cardiac troponin structure-function and the influence of hypertrophic cardiomyopathy associated mutations on modulation of contractility.Effect of hypertrophic cardiomyopathy-linked troponin C mutations on the response of reconstituted thin filaments to calcium upon troponin I phosphorylation.Troponin through the looking-glass: emerging roles beyond regulation of striated muscle contraction.Evidence for troponin C (TNNC1) as a gene for autosomal recessive restrictive cardiomyopathy with fatal outcome in infancy.
P2860
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P2860
Strong cross-bridges potentiate the Ca(2+) affinity changes produced by hypertrophic cardiomyopathy cardiac troponin C mutants in myofilaments: a fast kinetic approach.
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2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Strong cross-bridges potentiate the Ca
@nl
Strong cross-bridges potentiat ...... ents: a fast kinetic approach.
@ast
Strong cross-bridges potentiat ...... ents: a fast kinetic approach.
@en
type
label
Strong cross-bridges potentiate the Ca
@nl
Strong cross-bridges potentiat ...... ents: a fast kinetic approach.
@ast
Strong cross-bridges potentiat ...... ents: a fast kinetic approach.
@en
prefLabel
Strong cross-bridges potentiate the Ca
@nl
Strong cross-bridges potentiat ...... ents: a fast kinetic approach.
@ast
Strong cross-bridges potentiat ...... ents: a fast kinetic approach.
@en
P2093
P2860
P356
P1476
Strong cross-bridges potentiat ...... ments: a fast kinetic approach
@en
P2093
Daniel P Reynaldo
David Dweck
James D Potter
Jingsheng Liang
Martha M Sorenson
Michelle A Jones
Michelle S Parvatiyar
P2860
P304
P356
10.1074/JBC.M110.168583
P407
P577
2010-11-05T00:00:00Z