p38 MAPK activation is downstream of the loss of intercellular adhesion in pemphigus vulgaris.
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Desmosomes in acquired diseaseDesmosomes: regulators of cellular signaling and adhesion in epidermal health and disease.Signaling dependent and independent mechanisms in pemphigus vulgaris blister formationDesmoglein 2 is less important than desmoglein 3 for keratinocyte cohesion.Desmoglein as a target in skin disease and beyond.Structure, function, and regulation of desmosomes.Pathogenic relevance of IgG and IgM antibodies against desmoglein 3 in blister formation in pemphigus vulgarisNanorobotic investigation identifies novel visual, structural and functional correlates of autoimmune pathology in a blistering skin disease model.Interleukin-10 Enhances the Intestinal Epithelial Barrier in the Presence of Corticosteroids through p38 MAPK Activity in Caco-2 Monolayers: A Possible Mechanism for Steroid Responsiveness in Ulcerative ColitisSuper-Resolution Microscopy Reveals Altered Desmosomal Protein Organization in Tissue from Patients with Pemphigus Vulgaris.Setting the target for pemphigus vulgaris therapy.Pemphigus autoimmunity: hypotheses and realities.Desmoglein 3, its pathogenecity and a possibility for therapeutic target in pemphigus vulgaris.p38 MAPK Signaling in Pemphigus: Implications for Skin AutoimmunityDesmosomes and extradesmosomal adhesive signaling contacts in pemphigus.The dual nature of interleukin-10 in pemphigus vulgaris.Cell adhesion in epidermal development and barrier formation.Mechanisms of Disease: Pemphigus and Bullous Pemphigoid.Hepatocyte growth factor activator inhibitor type 1 maintains the assembly of keratin into desmosomes in keratinocytes by regulating protease-activated receptor 2-dependent p38 signaling.MAPKAP kinase 2 (MK2)-dependent and -independent models of blister formation in pemphigus vulgaris.EGFR inhibitors erlotinib and lapatinib ameliorate epidermal blistering in pemphigus vulgaris in a non-linear, V-shaped relationship.An adult passive transfer mouse model to study desmoglein 3 signaling in pemphigus vulgaris.Non-pathogenic pemphigus foliaceus (PF) IgG acts synergistically with a directly pathogenic PF IgG to increase blistering by p38MAPK-dependent desmoglein 1 clustering.Crosstalk between Signaling Pathways in Pemphigus: A Role for Endoplasmic Reticulum Stress in p38 Mitogen-Activated Protein Kinase Activation?Different signaling patterns contribute to loss of keratinocyte cohesion dependent on autoantibody profile in pemphigus.Pathogenic IgG4 autoantibodies from endemic pemphigus foliaceus recognize a desmoglein-1 conformational epitope.Signalling pathways in pemphigus vulgaris.Emerging treatment options for the management of pemphigus vulgaris.PemphigusCadherins: cellular adhesive molecules serving as signalling mediators
P2860
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P2860
p38 MAPK activation is downstream of the loss of intercellular adhesion in pemphigus vulgaris.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@ast
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@en
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@nl
type
label
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@ast
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@en
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@nl
prefLabel
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@ast
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@en
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@nl
P2093
P2860
P356
P1476
p38 MAPK activation is downstr ...... dhesion in pemphigus vulgaris.
@en
P2093
Aimee S Payne
Jin Mo Park
Xuming Mao
Yasuyo Sano
P2860
P304
P356
10.1074/JBC.M110.172874
P407
P577
2010-11-15T00:00:00Z