Antigen-responsive CD4+ T cells from C3H mice chronically infected with Leishmania amazonensis are impaired in the transition to an effector phenotype.
about
Chronic infection by Leishmania amazonensis mediated through MAPK ERK mechanismsA deficiency in the B cell response of C57BL/6 mice correlates with loss of macrophage-mediated killing of Leishmania amazonensisAn in vitro model of antibody-enhanced killing of the intracellular parasite Leishmania amazonensisIL-2 limits IL-12 enhanced lymphocyte proliferation during Leishmania amazonensis infection.Promotion of a functional B cell germinal center response after Leishmania species co-infection is associated with lesion resolutionRole of interleukin-1beta in activating the CD11c(high) CD45RB- dendritic cell subset and priming Leishmania amazonensis-specific CD4+ T cells in vitro and in vivo.Phosphatidylserine exposure on the surface of Leishmania amazonensis amastigotes modulates in vivo infection and dendritic cell function.Altered dendritic cell phenotype in response to Leishmania amazonensis amastigote infection is mediated by MAP kinase, ERKTargeted extracellular signal-regulated kinase activation mediated by Leishmania amazonensis requires MP1 scaffold.A systems approach to designing next generation vaccines: combining α-galactose modified antigens with nanoparticle platforms.A gut pathobiont synergizes with the microbiota to instigate inflammatory disease marked by immunoreactivity against other symbionts but not itself.
P2860
Q26825223-D0F30CD9-B86B-4931-B4F2-CF509A647FC0Q33623837-33247739-205F-49D8-A8D7-4988CA6A95FDQ34142338-3960EBFB-121C-459E-A8CE-8E6402DC617BQ35085800-5625C139-F34F-4705-A51C-1DA9DA7A164EQ35951825-F50982EA-8A2F-4B66-8B3F-DD040D6B8579Q36097200-2D232EFF-6162-4A6D-9A76-ABCB13F686E2Q36589026-2B157955-04BA-4F2A-9570-D36142B386B2Q37164233-E738CFBC-46DC-45AF-8496-580E667A4E26Q39030620-E62937C0-1C67-4732-AB17-C9D6ED48D633Q41879397-D7D955ED-8418-4C72-A09D-CE53B015D3AAQ46237071-2797E8A4-5AD5-4BF9-8345-68391E51DE21
P2860
Antigen-responsive CD4+ T cells from C3H mice chronically infected with Leishmania amazonensis are impaired in the transition to an effector phenotype.
description
2006 nî lūn-bûn
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2006 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մարտին հրատարակված գիտական հոդված
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2006年の論文
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2006年論文
@yue
2006年論文
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2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@ast
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@en
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@nl
type
label
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@ast
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@en
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@nl
prefLabel
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@ast
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@en
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@nl
P2093
P2860
P1476
Antigen-responsive CD4+ T cell ...... tion to an effector phenotype.
@en
P2093
Amanda E Ramer
Douglas E Jones
Yannick F Vanloubbeeck
P2860
P304
P356
10.1128/IAI.74.3.1547-1554.2006
P407
P577
2006-03-01T00:00:00Z