Oxidative stress damage as a detrimental factor in preterm birth pathology - Wikidata - awgldk - TriplyDB

Oxidative stress damage as a detrimental factor in preterm birth pathology

Oxidative stress damage as a detrimental factor in preterm birth pathology

http://www.wikidata.org/entity/Q34496420

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Novel concepts on pregnancy clocks and alarms: redundancy and synergy in human parturition Placental origins of adverse pregnancy outcomes: potential molecular targets: an Executive Workshop Summary of the Eunice Kennedy Shriver National Institute of Child Health and Human Development Maternal exposure to di-2-ethylhexylphthalate and adverse delivery outcomes: A systematic review.Relationships among Different Water-Soluble Choline Compounds Differ between Human Preterm and Donor Milk.Soluble receptors for advanced glycation end products and receptor activator of NF-κB ligand serum levels as markers of premature labor.Role of Oxidative RNA Damage in Chronic-Degenerative Diseases.Intraamniotic Inflammation in Women with Preterm Prelabor Rupture of Membranes Telomere Fragment Induced Amnion Cell Senescence: A Contributor to Parturition?Ancestry informative markers and selected single nucleotide polymorphisms in immunoregulatory genes on preterm labor and preterm premature rupture of membranes: a case control study Amnion-Epithelial-Cell-Derived Exosomes Demonstrate Physiologic State of Cell under Oxidative Stress Untargeted Metabolomic Analysis of Amniotic Fluid in the Prediction of Preterm Delivery and Bronchopulmonary Dysplasia.Umbilical cord gene expression reveals the molecular architecture of the fetal inflammatory response in extremely preterm newborns.Evaluation of proteomic biomarkers associated with circulating microparticles as an effective means to stratify the risk of spontaneous preterm birth.The influence of oxidative stress and autophagy cross regulation on pregnancy outcome.Organochlorine pesticides exposure & preterm birth.Mediation of the Relationship between Maternal Phthalate Exposure and Preterm Birth by Oxidative Stress with Repeated Measurements across Pregnancy.Human fetal membranes at term: Dead tissue or signalers of parturition?Mechanistic Differences Leading to Infectious and Sterile Inflammation.Oxidative stress, placental ageing-related pathologies and adverse pregnancy outcomes.p38 Mitogen activated protein kinase (MAPK): a new therapeutic target for reducing the risk of adverse pregnancy outcomes.Preclinical evaluation of drugs to block inflammation-driven preterm birth.The Burden of Genetic and Epigenetic Traits in Prematurity.The relationship between the latency period, infection markers, and oxidant and antioxidant states in women with preterm premature rupture of membranes.Combinations and loads of bacteria affect the cytokine production by fetal membranes: An in vitro study.Oxidative stress damage-associated molecular signaling pathways differentiate spontaneous preterm birth and preterm premature rupture of the membranes.Feto-Maternal Trafficking of Exosomes in Murine Pregnancy Models.The role of oxidative stress, inflammation and acetaminophen exposure from birth to early childhood in the induction of autism.Potential application of the oxidative nucleic acid damage biomarkers in detection of diseases.The Unfolded Protein Response Regulates Uterine Myocyte Antioxidant Responsiveness During Pregnancy.The Role of Epithelial to Mesenchymal Transition in Human Amniotic Membrane Rupture.Screening of lysyl oxidase (LOX) and lysyl oxidase like (LOXL) enzyme expression and activity in preterm prelabor rupture of fetal membranes.Placental exosomes: A proxy to understand pregnancy complications.A distinct mechanism of senescence activation in amnion epithelial cells by infection, inflammation, and oxidative stress.Histologic chorioamnionitis does not modulate the oxidative stress and antioxidant status in pregnancies complicated by spontaneous preterm delivery.Birth and death: Evidence for the same biologic clock.Association Between Placental Polycyclic Aromatic Hydrocarbons (PAHS), Oxidative Stress, and Preterm Delivery: A Case-Control Study.Damage-Associated molecular pattern markers HMGB1 and cell-Free fetal telomere fragments in oxidative-Stressed amnion epithelial cell-Derived exosomes.The maternal thiol/disulfide homeostasis does not change in pregnancies complicated by preterm prelabor rupture of membranes.Preterm prelabor rupture of the membranes: A disease of the fetal membranes.Oxidative stress induces senescence and sterile inflammation in murine amniotic cavity.

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P2860

Oxidative stress damage as a detrimental factor in preterm birth pathology

http://www.wikidata.org/entity/Q34496420

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2014 nî lūn-bûn

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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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Oxidative stress damage as a detrimental factor in preterm birth pathology

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Oxidative stress damage as a detrimental factor in preterm birth pathology

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Oxidative stress damage as a detrimental factor in preterm birth pathology

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Oxidative stress damage as a detrimental factor in preterm birth pathology

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Oxidative stress damage as a detrimental factor in preterm birth pathology

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Progesterone acts via the nuclear glucocorticoid receptor to suppress IL-1β-induced COX-2 expression in human term myometrial cells

A placental clock controlling the length of human pregnancy

Vaginal progesterone reduces the rate of preterm birth in women with a sonographic short cervix: a multicenter, randomized, double-blind, placebo-controlled trial

Normal oxidative damage to mitochondrial and nuclear DNA is extensive

The human OGG1 gene: structure, functions, and its implication in the process of carcinogenesis

Infection and prematurity and the role of preventive strategies

The effects of oxidative stress on female reproduction: a review

Stimulation of human 8-oxoguanine-DNA glycosylase by AP-endonuclease: potential coordination of the initial steps in base excision repair

Oxygen toxicity, oxygen radicals, transition metals and disease

Signal transduction by reactive oxygen species

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