HCMV-infected cells maintain efficient nucleotide excision repair of the viral genome while abrogating repair of the host genome.
about
NEDDylation is essential for Kaposi's sarcoma-associated herpesvirus latency and lytic reactivation and represents a novel anti-KSHV targetPUL21a-Cyclin A2 interaction is required to protect human cytomegalovirus-infected cells from the deleterious consequences of mitotic entry.Network representations of immune system complexity.DNA repair mechanisms and human cytomegalovirus (HCMV) infection.Synthetic lethal mutations in the cyclin A interface of human cytomegalovirusInfection of a Single Cell Line with Distinct Strains of Human Cytomegalovirus Can Result in Large Variations in Virion Production and Facilitate Efficient Screening of Virus Protein Function.Maintenance of large numbers of virus genomes in human cytomegalovirus-infected T98G glioblastoma cellsA dual color Southern blot to visualize two genomes or genic regions simultaneously.Modulation of homology-directed repair in T98G glioblastoma cells due to interactions between wildtype p53, Rad51 and HCMV IE1-72.Cytomegalovirus Late Protein pUL31 Alters Pre-rRNA Expression and Nuclear Organization during Infection.The absence of p53 during Human Cytomegalovirus infection leads to decreased UL53 expression, disrupting UL50 localization to the inner nuclear membrane, and thereby inhibiting capsid nuclear egress.A novel DDB2-ATM feedback loop regulates human cytomegalovirus replication.
P2860
Q28544735-2E2B118E-0657-4F5C-A4F3-04E0C315153FQ34506903-D53A0A6A-2636-4719-BCB4-7DB72768F74FQ35118812-D11F6392-435E-466F-8E38-EAC31BBA08E2Q35598400-1B78399B-9EEC-4520-8E41-A0C4B586DC11Q36262707-33E324E5-FD3E-45DC-9888-19EF77651E95Q36736886-DDEA1AFD-BE2A-4F2C-9A13-49017F8A2535Q37713554-BFF2BCC2-AE8F-4B8B-91BE-6EEF2A3B9752Q38175685-38AB06C8-312A-4342-8D88-2953E7930007Q38562250-B56798B5-D910-4EEE-9966-42721CE7578DQ38698697-D15FC5D0-25A6-43AE-9C60-56198013CC10Q38753361-C505B44B-6ACC-4C3A-910D-407751512937Q41908881-EEC87ECB-6C34-441B-A90A-83208770E83D
P2860
HCMV-infected cells maintain efficient nucleotide excision repair of the viral genome while abrogating repair of the host genome.
description
2012 nî lūn-bûn
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2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年論文
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2012年論文
@zh-hant
2012年論文
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2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
HCMV-infected cells maintain e ...... ing repair of the host genome.
@ast
HCMV-infected cells maintain e ...... ing repair of the host genome.
@en
HCMV-infected cells maintain e ...... ing repair of the host genome.
@nl
type
label
HCMV-infected cells maintain e ...... ing repair of the host genome.
@ast
HCMV-infected cells maintain e ...... ing repair of the host genome.
@en
HCMV-infected cells maintain e ...... ing repair of the host genome.
@nl
prefLabel
HCMV-infected cells maintain e ...... ing repair of the host genome.
@ast
HCMV-infected cells maintain e ...... ing repair of the host genome.
@en
HCMV-infected cells maintain e ...... ing repair of the host genome.
@nl
P2093
P2860
P1433
P1476
HCMV-infected cells maintain e ...... ing repair of the host genome.
@en
P2093
Anamaria G Zavala
Celeste J Brown
Elizabeth A Fortunato
John M O'Dowd
Toshio Mori
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003038
P577
2012-11-29T00:00:00Z