Free fatty acids in the presence of high glucose amplify monocyte inflammation via Toll-like receptors.
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Autonomic neuropathy in diabetes mellitusMacrophages and Their Role in Atherosclerosis: Pathophysiology and Transcriptome AnalysisObesity-Driven Gut Microbiota Inflammatory Pathways to Metabolic SyndromeThe Crosstalk between Hypoxia and Innate Immunity in the Development of Obesity-Related Nonalcoholic Fatty Liver DiseasePostulates for validating TLR4 agonistsMolecular Pathogenesis of NASHSaturated palmitic acid induces myocardial inflammatory injuries through direct binding to TLR4 accessory protein MD2Phenotypic modulation of macrophages in response to plaque lipids.Toll-like receptor 2 and type 2 diabetes.Over-expression of TLR4-CD14, pro-inflammatory cytokines, metabolic markers and NEFAs in obese non-diabetic MexicansIncreased systemic Th17 cytokines are associated with diastolic dysfunction in children and adolescents with diabetic ketoacidosisThe oxidative state of chylomicron remnants influences their modulation of human monocyte activation.TLR-3 is present in human adipocytes, but its signalling is not required for obesity-induced inflammation in adipose tissue in vivo.Toll-like receptor 4 promotes tubular inflammation in diabetic nephropathy.Heat shock protein 60 as a mediator of adipose tissue inflammation and insulin resistance.Increased toll-like receptor activity in patients with metabolic syndromePrognostic value of cerebrospinal fluid free fatty acid levels in patients with acute ischemic stroke.Proinsulin-producing, hyperglycemia-induced adipose tissue macrophages underlie insulin resistance in high fat-fed diabetic miceLong-lasting partnership between insulin resistance and endothelial dysfunction: role of metabolic memory.Skeletal Muscle Insulin Resistance and Absence of Inflammation Characterize Insulin-Resistant Grade I Obese Women.Paying the Toll for Glucose Regulation: A Central Role for TLR3.Exendin-4 protected against cognitive dysfunction in hyperglycemic mice receiving an intrahippocampal lipopolysaccharide injection.High glucose induces and activates Toll-like receptor 4 in endothelial cells of diabetic retinopathyThe inflammation highway: metabolism accelerates inflammatory traffic in obesity.Improved Metabolic Control in Diabetes, HSP60, and Proinflammatory Mediators.Metabolic diseases and pro- and prebiotics: Mechanistic insightsPodocyte apoptosis is prevented by blocking the Toll-like receptor pathway.Fenugreek lactone attenuates palmitate-induced apoptosis and dysfunction in pancreatic β-cellsIncreased toll-like receptor 2 expression in peptidoglycan-treated blood monocytes is associated with insulin resistance in patients with nondiabetic rheumatoid arthritis.Effects of Toll-like receptor antagonist 4,5-dihydro-3-phenyl-5-isoxasole acetic acid on the progression of kidney disease in mice on a high-fat diet.Heat shock proteins induction reduces stress kinases activation, potentially improving insulin signalling in monocytes from obese subjectsVisceral and subcutaneous adipose tissue from lean women respond differently to lipopolysaccharide-induced alteration of inflammation and glyceroneogenesisEndothelial acyl-CoA synthetase 1 is not required for inflammatory and apoptotic effects of a saturated fatty acid-rich environment.Hyporesponsiveness to the anti-inflammatory action of interleukin-10 in type 2 diabetesDiminished anabolic signaling response to insulin induced by intramuscular lipid accumulation is associated with inflammation in aging but not obesityPreliminary analysis of immune activation in early onset type 2 diabetes.Increased expression of triggering receptor expressed on myeloid cells-1 in the population with obesity and insulin resistance.Defects in TLR3 expression and RNase L activation lead to decreased MnSOD expression and insulin resistance in muscle cells of obese people.Pubertal high fat diet: effects on mammary cancer development.Evidences of +896 A/G TLR4 polymorphism as an indicative of prevalence of complications in T2DM patients.
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P2860
Free fatty acids in the presence of high glucose amplify monocyte inflammation via Toll-like receptors.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Free fatty acids in the presen ...... ation via Toll-like receptors.
@ast
Free fatty acids in the presen ...... ation via Toll-like receptors.
@en
Free fatty acids in the presen ...... ation via Toll-like receptors.
@nl
type
label
Free fatty acids in the presen ...... ation via Toll-like receptors.
@ast
Free fatty acids in the presen ...... ation via Toll-like receptors.
@en
Free fatty acids in the presen ...... ation via Toll-like receptors.
@nl
prefLabel
Free fatty acids in the presen ...... ation via Toll-like receptors.
@ast
Free fatty acids in the presen ...... ation via Toll-like receptors.
@en
Free fatty acids in the presen ...... ation via Toll-like receptors.
@nl
P2860
P921
P1476
Free fatty acids in the presen ...... ation via Toll-like receptors.
@en
P2093
Ishwarlal Jialal
Mohan R Dasu
P2860
P304
P356
10.1152/AJPENDO.00490.2010
P577
2010-10-19T00:00:00Z