Depletion of neutrophils in a protective model of pulmonary cryptococcosis results in increased IL-17A production by γδ T cells. - Wikidata - awgldk - TriplyDB

Depletion of neutrophils in a protective model of pulmonary cryptococcosis results in increased IL-17A production by γδ T cells.

Depletion of neutrophils in a protective model of pulmonary cryptococcosis results in increased IL-17A production by γδ T cells.

http://www.wikidata.org/entity/Q34502561

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Cryptococcus and Phagocytes: Complex Interactions that Influence Disease Outcome Sphingolipids as Regulators of the Phagocytic Response to Fungal Infections Dancing cheek to cheek: Cryptococcus neoformans and phagocytes Host immunity to Cryptococcus neoformans Innate receptors and cellular defense against pulmonary infections Real-Time Imaging of Interactions of Neutrophils with Cryptococcus neoformans Demonstrates a Crucial Role of Complement C5a-C5aR Signaling Nitrite therapy improves survival postexposure to chlorine gas γδ T cells are required for pulmonary IL-17A expression after ozone exposure in mice: role of TNFα A Zebrafish Model of Cryptococcal Infection Reveals Roles for Macrophages, Endothelial Cells, and Neutrophils in the Establishment and Control of Sustained Fungemia.Cytokine profiles at admission can be related to outcome in AIDS patients with cryptococcal meningitis Cerebrospinal fluid cytokine profiles predict risk of early mortality and immune reconstitution inflammatory syndrome in HIV-associated cryptococcal meningitis Cryptococcus neoformans Infection in Mice Lacking Type I Interferon Signaling Leads to Increased Fungal Clearance and IL-4-Dependent Mucin Production in the Lungs.Depletion of Neutrophils Promotes the Resolution of Pulmonary Inflammation and Fibrosis in Mice Infected with Paracoccidioides brasiliensis.STAT1 signaling within macrophages is required for antifungal activity against Cryptococcus neoformans The Cnes2 locus on mouse chromosome 17 regulates host defense against cryptococcal infection through pleiotropic effects on host immunity Intravascular clearance of disseminating Cryptococcus neoformans in the brain can be improved by enhancing neutrophil recruitment in mice.Role of neutrophils in IL-17-dependent immunity to mucosal candidiasis.Therapeutic potential of interleukin-17 in inflammation and autoimmune diseases.When neutrophils meet T cells: beginnings of a tumultuous relationship with underappreciated potential.γδ T Cell-Mediated Immune Responses in Disease and Therapy Immunity to Cryptococcus neoformans and C. gattii during cryptococcosis Cytokine crowdsourcing: multicellular production of TH17-associated cytokines.The Jekyll and Hyde story of IL17-Producing γδT Cells.Innate host defenses against Cryptococcus neoformans.IL-17 in the lung: The Good, the Bad, and the Ugly.Neutrophils regulate the lung inflammatory response via γδ T cell infiltration in an experimental mouse model of human metapneumovirus infection.Depletion of Neutrophils Exacerbates the Early Inflammatory Immune Response in Lungs of Mice Infected with Paracoccidioides brasiliensis.Identification of Immune Effectors Essential to the Control of Primary and Secondary Intranasal Infection with Brucella melitensis in Mice.Innate Immune Responses to Cryptococcus.Induction of Broad-Spectrum Protective Immunity against Disparate Cryptococcus Serotypes.Galectin-3 impacts Cryptococcus neoformans infection through direct antifungal effects.Adaptive Immunity to Cryptococcus neoformans Infections.Contribution of IL-1RI Signaling to Protection against Cryptococcus neoformans 52D in a Mouse Model of Infection.Host response to pulmonary fungal infections: A highlight on cell-driven immunity to Cryptococcus species and Aspergillus fumigatus.Tumor-associated neutrophils suppress pro-tumoral IL-17+ γδ T cells through induction of oxidative stress.Innate Immunity against Cryptococcus, from Recognition to Elimination.Involvement of the capsular GalXM-induced IL-17 cytokine in the control of Cryptococcus neoformans infection

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P2860

Depletion of neutrophils in a protective model of pulmonary cryptococcosis results in increased IL-17A production by γδ T cells.

http://www.wikidata.org/entity/Q34502561

description

2012 nî lūn-bûn

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2012 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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Depletion of neutrophils in a ...... -17A production by γδ T cells.

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Depletion of neutrophils in a ...... -17A production by γδ T cells.

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Depletion of neutrophils in a ...... -17A production by γδ T cells.

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Depletion of neutrophils in a ...... L-17A production by γδ T cells

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Floyd L Wormley

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Jay K Kolls

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P2860

γδ T Cells Link Innate and Adaptive Immune Responses

Lymphoid tissue inducer-like cells are an innate source of IL-17 and IL-22

IL-17 and Th17 Cells

IL-17A-producing neutrophil-regulatory Tn lymphocytes

The function of gammadelta T cells in innate immunity

Cryptococcosis in the era of AIDS--100 years after the discovery of Cryptococcus neoformans.

IL-17A inhibits the expansion of IL-17A-producing T cells in mice through "short-loop" inhibition via IL-17 receptor.

Pertussis toxin stimulates IL-17 production in response to Bordetella pertussis infection in mice.

Innate invariant NKT cells recognize Mycobacterium tuberculosis-infected macrophages, produce interferon-gamma, and kill intracellular bacteria.

Insights into the mechanisms of protective immunity against Cryptococcus neoformans infection using a mouse model of pulmonary cryptococcosis

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Karen L. Wozniak

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