Interleukin-10 reduces the pathology of mdx muscular dystrophy by deactivating M1 macrophages and modulating macrophage phenotype. - Wikidata - awgldk - TriplyDB

Interleukin-10 reduces the pathology of mdx muscular dystrophy by deactivating M1 macrophages and modulating macrophage phenotype.

Interleukin-10 reduces the pathology of mdx muscular dystrophy by deactivating M1 macrophages and modulating macrophage phenotype.

http://www.wikidata.org/entity/Q34505654

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Pre-clinical drug tests in the mdx mouse as a model of dystrophinopathies: an overview Inflammation induced loss of skeletal muscle Proinflammatory chemokines are major mediators of exuberant immune response associated with Influenza A (H1N1) pdm09 virus infection.Potent pro-inflammatory and pro-fibrotic molecules, osteopontin and galectin-3, are not major disease modulators of laminin α2 chain-deficient muscular dystrophy.Influence of immune responses in gene/stem cell therapies for muscular dystrophies.Changes in circulating biomarkers of muscle atrophy, inflammation, and cartilage turnover in patients undergoing anterior cruciate ligament reconstruction and rehabilitation.Neuronal nitric oxide synthase-rescue of dystrophin/utrophin double knockout mice does not require nNOS localization to the cell membrane Macrophage phenotype as a predictor of constructive remodeling following the implantation of biologically derived surgical mesh materials.IL-6 signaling blockade increases inflammation but does not affect muscle function in the mdx mouse.Matrix metalloproteinase-9 inhibition improves proliferation and engraftment of myogenic cells in dystrophic muscle of mdx mice Association of the four common polymorphisms in interleukin-10 (rs1800890, rs1800896, rs1800871, and rs1800872) with non-Hodgkin's lymphoma risk: a meta-analysis.Sarcolemma instability during mechanical activity in Largemyd cardiac myocytes with loss of dystroglycan extracellular matrix receptor function pSiM24 is a novel versatile gene expression vector for transient assays as well as stable expression of foreign genes in plants.SHP-1-dependent macrophage differentiation exacerbates virus-induced myositis Platelet-rich plasma and skeletal muscle healing: a molecular analysis of the early phases of the regeneration process in an experimental animal model.Toll-like receptor 4 ablation in mdx mice reveals innate immunity as a therapeutic target in Duchenne muscular dystrophy IFN-γ promotes muscle damage in the mdx mouse model of Duchenne muscular dystrophy by suppressing M2 macrophage activation and inhibiting muscle cell proliferation The anti-inflammatory cytokine, interleukin-10, inhibits inflammatory mediators in human epithelial cells and mouse macrophages exposed to live and UV-inactivated Chlamydia trachomatis.Meeting Report: New Directions in the Biology and Disease of Skeletal Muscle 2014 PEDF-derived peptide promotes skeletal muscle regeneration through its mitogenic effect on muscle progenitor cells Increases of M2a macrophages and fibrosis in aging muscle are influenced by bone marrow aging and negatively regulated by muscle-derived nitric oxide.The immune system in Duchenne muscular dystrophy: Friend or foe.IL-10 triggers changes in macrophage phenotype that promote muscle growth and regeneration.Rotator cuff tear reduces muscle fiber specific force production and induces macrophage accumulation and autophagy The PGC-1 coactivators promote an anti-inflammatory environment in skeletal muscle in vivo.Highly purified eicosapentaenoic acid increases interleukin-10 levels of peripheral blood monocytes in obese patients with dyslipidemia.Requirement of inducible nitric oxide synthase for skeletal muscle regeneration after acute damage.Monocyte/Macrophage-derived IGF-1 Orchestrates Murine Skeletal Muscle Regeneration and Modulates Autocrine Polarization.Regulatory T cells suppress muscle inflammation and injury in muscular dystrophy.Age-related loss of nitric oxide synthase in skeletal muscle causes reductions in calpain S-nitrosylation that increase myofibril degradation and sarcopenia.Tissue Tregs.Wasting mechanisms in muscular dystrophy.Osteopontin ablation ameliorates muscular dystrophy by shifting macrophages to a pro-regenerative phenotype Monocyte depletion increases local proliferation of macrophage subsets after skeletal muscle injury Klotho gene silencing promotes pathology in the mdx mouse model of Duchenne muscular dystrophy.Shared signaling systems in myeloid cell-mediated muscle regeneration.Longitudinal PET imaging of muscular inflammation using 18F-DPA-714 and 18F-Alfatide II and differentiation with tumors.Macrophage polarization: an opportunity for improved outcomes in biomaterials and regenerative medicine Macrophages in injured skeletal muscle: a perpetuum mobile causing and limiting fibrosis, prompting or restricting resolution and regeneration.Mechanisms of muscle injury, repair, and regeneration.

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Interleukin-10 reduces the pathology of mdx muscular dystrophy by deactivating M1 macrophages and modulating macrophage phenotype.

http://www.wikidata.org/entity/Q34505654

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Interleukin-10 reduces the pat ...... dulating macrophage phenotype.

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Interleukin-10 reduces the pat ...... dulating macrophage phenotype.

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Interleukin-10 reduces the pat ...... dulating macrophage phenotype.

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Interleukin-10 reduces the pat ...... dulating macrophage phenotype.

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Interleukin-10 reduces the pat ...... dulating macrophage phenotype.

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Interleukin-10 reduces the pat ...... dulating macrophage phenotype.

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Human Molecular Genetics

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Bo Deng

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Brian Fedor

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Chiara Rinaldi

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Grace Liu

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S Armando Villalta

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P2860

Glucocorticoid corticosteroids for Duchenne muscular dystrophy

Interleukin-10 and the interleukin-10 receptor

Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

Interleukin-10: new perspectives on an old cytokine

The MIQE guidelines: minimum information for publication of quantitative real-time PCR experiments

Pro-inflammatory cytokines and environmental stress cause p38 mitogen-activated protein kinase activation by dual phosphorylation on tyrosine and threonine

Quantification of mRNA using real-time RT-PCR

Prototypical anti-inflammatory cytokine IL-10 prevents loss of IGF-I-induced myogenin protein expression caused by IL-1beta

IL-4 acts as a myoblast recruitment factor during mammalian muscle growth

Molecular cloning and characterization of the mouse CD163 homologue, a highly glucocorticoid-inducible member of the scavenger receptor cysteine-rich family

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21118895

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