Cutoff in potency implicates alcohol inhibition of N-methyl-D-aspartate receptors in alcohol intoxication.
about
Isoform-specific enhancement of adenylyl cyclase activity by n-alkanolsGeneral anaesthetic actions on ligand-gated ion channelsMethionine 286 in transmembrane domain 3 of the GABAA receptor beta subunit controls a binding cavity for propofol and other alkylphenol general anestheticsStraight-chain alcohols exhibit a cutoff in potency for the inhibition of recombinant glutamate receptor subunitsEthanol opens G-protein-activated inwardly rectifying K+ channelsAlcohol inhibits cell-cell adhesion mediated by human L1Acute effects of ethanol on glutamate receptors.Signaling cascades regulating NMDA receptor sensitivity to ethanol.Hydrocarbon molar water solubility predicts NMDA vs. GABAA receptor modulation.Ethanol is a fast channel inhibitor of P2X4 receptorsInhibition of glycine receptor function of native neurons by aliphatic n-alcohols.Alcohol's effects on lipid bilayer properties.Role of altered structure and function of NMDA receptors in development of alcohol dependence.Ethanol inhibition of m-current and ethanol-induced direct excitation of ventral tegmental area dopamine neurons.n-Alcohols inhibit voltage-gated Na+ channels expressed in Xenopus oocytes.Alcohol related changes in regulation of NMDA receptor functionsNeuroscience of alcoholism: molecular and cellular mechanismsAcute ethanol has biphasic effects on short- and long-term memory in both foreground and background contextual fear conditioning in C57BL/6 mice.What makes good antioxidants in lipid-based systems? The next theories beyond the polar paradox.A site of alcohol action in the fourth membrane-associated domain of the N-methyl-D-aspartate receptor.Differential effects of ethanol on glycine uptake mediated by the recombinant GLYT1 and GLYT2 glycine transporters.Actions of long chain alcohols on GABAA and glutamate receptors: relation to in vivo effects.Ethanol reduces excitability in a subgroup of primary sensory neurons by activation of BK(Ca) channels.
P2860
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P2860
Cutoff in potency implicates alcohol inhibition of N-methyl-D-aspartate receptors in alcohol intoxication.
description
1995 nî lūn-bûn
@nan
1995 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մարտին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@ast
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@en
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@nl
type
label
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@ast
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@en
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@nl
prefLabel
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@ast
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@en
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@nl
P2860
P356
P1476
Cutoff in potency implicates a ...... ptors in alcohol intoxication.
@en
P2093
P2860
P304
P356
10.1073/PNAS.92.7.2825
P407
P577
1995-03-01T00:00:00Z