Th17 responses in chronic allergic airway inflammation abrogate regulatory T-cell-mediated tolerance and contribute to airway remodeling - Wikidata - awgldk - TriplyDB

Th17 responses in chronic allergic airway inflammation abrogate regulatory T-cell-mediated tolerance and contribute to airway remodeling

Th17 responses in chronic allergic airway inflammation abrogate regulatory T-cell-mediated tolerance and contribute to airway remodeling

http://www.wikidata.org/entity/Q34519468

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Th17/Treg ratio derived using DNA methylation analysis is associated with the late phase asthmatic response Recent advances in understanding the molecular mechanisms of the development and function of Th17 cells Elucidating novel disease mechanisms in severe asthma Leukocyte homing, fate, and function are controlled by retinoic acid Microbial Ligand Costimulation Drives Neutrophilic Steroid-Refractory Asthma Neutralization of interleukin-17 suppresses allergic rhinitis symptoms by downregulating Th2 and Th17 responses and upregulating the Treg response.Intraepithelial neutrophils in pediatric severe asthma are associated with better lung function.Airway Inflammation after Bronchial Thermoplasty for Severe Asthma.Dendritic Cell-Airway Epithelial Cell Cross-Talk Changes with Age and Contributes to Chronic Lung Inflammatory Diseases in the Elderly Aligning mouse models of asthma to human endotypes of disease.Design of a ProDer f 1 vaccine delivered by the MHC class II pathway of antigen presentation and analysis of the effectiveness for specific immunotherapy.IL-22 contributes to TGF-β1-mediated epithelial-mesenchymal transition in asthmatic bronchial epithelial cells.IL-17 in severe asthma. Where do we stand?All-trans retinoic acid attenuates airway inflammation by inhibiting Th2 and Th17 response in experimental allergic asthma The endogenous Th17 response in NO2-promoted allergic airway disease is dispensable for airway hyperresponsiveness and distinct from Th17 adoptive transfer.Exogenous interleukin-10 alleviates allergic inflammation but inhibits local interleukin-10 expression in a mouse allergic rhinitis model.Severe acute respiratory syndrome-associated coronavirus vaccines formulated with delta inulin adjuvants provide enhanced protection while ameliorating lung eosinophilic immunopathology.Immune modulatory effects of IL-22 on allergen-induced pulmonary inflammation.IL-17 Enhances Chemotaxis of Primary Human B Cells during Asthma.FOXP3(+)Treg/Th17 cell imbalance in lung tissues of mice with asthma.Differential induction of total IgE by two Salmonella enterica serotypes.γ-Secretase Inhibitor Alleviates Acute Airway Inflammation of Allergic Asthma in Mice by Downregulating Th17 Cell Differentiation.The relationship of serum vitamins A, D, E and LL-37 levels with allergic status, tonsillar virus detection and immune response.Acute exposure to methamphetamine alters TLR9-mediated cytokine expression in human macrophage.Th17 Cytokines Disrupt the Airway Mucosal Barrier in Chronic Rhinosinusitis.Modified Foxp3 mRNA protects against asthma through an IL-10-dependent mechanism.Blockade of STAT3 in T Cells Inhibits Germinal Center Reactions against Intranasal Allergens.High Fat Diet Inhibits Dendritic Cell and T Cell Response to Allergens but Does Not Impair Inhalational Respiratory Tolerance.IL-12 and IL-4 activate a CD39-dependent intrinsic peripheral tolerance mechanism in CD8(+) T cells.Inflammatory cytokines in pediatric obstructive sleep apnea.Blockade of IL-23 ameliorates allergic lung inflammation via decreasing the infiltration of Tc17 cells Airway remodeling in asthma: what really matters.Neutralization of interleukin-9 ameliorates symptoms of allergic rhinitis by reducing Th2, Th9, and Th17 responses and increasing the Treg response in a murine model.Asthma: the importance of dysregulated barrier immunity.Immunoregulatory mechanisms of vitamin D relevant to respiratory health and asthma.Vitamin D influences asthmatic pathology through its action on diverse immunological pathways.The impact of diet on asthma and allergic diseases.Immunopathology alters Th17 cell glucocorticoid sensitivity.Mouse models of severe asthma: Understanding the mechanisms of steroid resistance, tissue remodelling and disease exacerbation.Pre-existing tolerance shapes the outcome of mucosal allergen sensitization in a murine model of asthma.

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Th17 responses in chronic allergic airway inflammation abrogate regulatory T-cell-mediated tolerance and contribute to airway remodeling

http://www.wikidata.org/entity/Q34519468

description

2012 nî lūn-bûn

@nan

2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի օգոստոսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Th17 responses in chronic alle ...... ontribute to airway remodeling

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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type

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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Mucosal Immunology

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Th17 responses in chronic alle ...... ontribute to airway remodeling

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A Noble

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C M Lloyd

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J Zhao

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P2860

Antigen-specific regulatory T cells develop via the ICOS-ICOS-ligand pathway and inhibit allergen-induced airway hyperreactivity

Role of IL-17A, IL-17F, and the IL-17 receptor in regulating growth-related oncogene-alpha and granulocyte colony-stimulating factor in bronchial epithelium: implications for airway inflammation in cystic fibrosis

T cell interleukin-17 induces stromal cells to produce proinflammatory and hematopoietic cytokines

IL-17 and Th17 Cells

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

Reciprocal TH17 and regulatory T cell differentiation mediated by retinoic acid

MyD88-dependent induction of allergic Th2 responses to intranasal antigen.

Signal peptide cleavage is essential for surface expression of a regulatory T cell surface protein, leucine rich repeat containing 32 (LRRC32).

IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines.

Resolution of allergic airway inflammation and airway hyperreactivity is mediated by IL-17-producing {gamma}{delta}T cells

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10.1038/MI.2012.76

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