Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis? - Wikidata - awgldk - TriplyDB

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

http://www.wikidata.org/entity/Q34522154

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Identification of novel in vivo phosphorylation sites of the human proapoptotic protein BAD: pore-forming activity of BAD is regulated by phosphorylation Mechanisms of acetaminophen-induced liver necrosis Ceramide-induced apoptosis in renal tubular cells: a role of mitochondria and sphingosine-1-phoshate Mitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategies Mechanistic insights into the oncolytic activity of vesicular stomatitis virus in cancer immunotherapy Plants vs. cancer: a review on natural phytochemicals in preventing and treating cancers and their druggability Reactive oxygen species, cellular redox systems, and apoptosis Upregulation of Bcl2 inhibits apoptosis-driven BAX insertion but favors BAX relocalization in mitochondria Mitochondrial apoptosis is amplified through gap junctions.Mitochondrial ion channels as therapeutic targets.Liquiritin modulates ERK‑ and AKT/GSK‑3β‑dependent pathways to protect against glutamate‑induced cell damage in differentiated PC12 cells.Chemical glycosylation of cytochrome c improves physical and chemical protein stability.Is mPTP the gatekeeper for necrosis, apoptosis, or both?Mitochondrial apoptosis-induced channel (MAC) function triggers a Bax/Bak-dependent bystander effect.MAC inhibitors suppress mitochondrial apoptosis.Glutathione and apoptosis.African flora has the potential to fight multidrug resistance of cancer Mitochondrial Ion Channels in Cancer Transformation Cinnamomum cassia Suppresses Caspase-9 through Stimulation of AKT1 in MCF-7 Cells but Not in MDA-MB-231 Cells.The novel protective role of P27 in MLN4924-treated gastric cancer cells.Chamaejasmin B exerts anti-MDR effect in vitro and in vivo via initiating mitochondria-dependant intrinsic apoptosis pathway.The therapeutic potential of mitochondrial channels in cancer, ischemia-reperfusion injury, and neurodegeneration.Paroxetine ameliorates changes in hippocampal energy metabolism in chronic mild stress-exposed rats Ethanol influences on Bax associations with mitochondrial membrane proteins in neonatal rat cerebellum.Bax and Bak expression in cervical smears of women with low-and high-risk HPV types: A study of 120 cases VDAC1: from structure to cancer therapy.Mitochondrial ion channels.A mechanistic view of mitochondrial death decision pores.Ethanol exposure decreases mitochondrial outer membrane permeability in cultured rat hepatocytes.Assembly of the mitochondrial apoptosis-induced channel, MAC.Overproduction of reactive oxygen species - obligatory or not for induction of apoptosis by anticancer drugs Anti-apoptotic Bcl-2 Family Proteins Disassemble Ceramide Channels N-acetyl-serotonin offers neuroprotection through inhibiting mitochondrial death pathways and autophagic activation in experimental models of ischemic injury.MAC and Bcl-2 family proteins conspire in a deadly plot.Siamese crocodile bile induces apoptosis in NCI-H1299 human non-small cell lung cancer cells via a mitochondria-mediated intrinsic pathway and inhibits tumorigenesis.Protective effect of DHEA on hydrogen peroxide-induced oxidative damage and apoptosis in primary rat Leydig cells.Mitochondrial channels: ion fluxes and more.Induction of Cell Death Mechanisms and Apoptosis by Nanosecond Pulsed Electric Fields (nsPEFs).Central (ICV) leptin injection increases bone formation, bone mineral density, muscle mass, serum IGF-1, and the expression of osteogenic genes in leptin-deficient ob/ob mice.Cross-talk between apoptosis and autophagy in lung epithelial cell death.

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P2860

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

http://www.wikidata.org/entity/Q34522154

description

2006 nî lūn-bûn

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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2006 թվականի մայիսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

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Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

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Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

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type

label

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

@ast

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

@en

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

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prefLabel

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

@ast

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

@en

Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

@nl

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Cell Death & Differentiation

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Is MAC the knife that cuts cytochrome c from mitochondria during apoptosis?

@en

P2093

Dejean LM

http://www.w3.org/2001/XMLSchema#string

Kinnally KW

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Martinez-Caballero S

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P2860

Regulation of the mitochondrial apoptosis-induced channel, MAC, by BCL-2 family proteins

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

Inhibition of Bax channel-forming activity by Bcl-2

Oligomeric Bax is a component of the putative cytochrome c release channel MAC, mitochondrial apoptosis-induced channel

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

Conformation of the Bax C-terminus regulates subcellular location and cell death

Microsecond time-scale discrimination among polycytidylic acid, polyadenylic acid, and polyuridylic acid as homopolymers or as segments within single RNA molecules.

A novel, high conductance channel of mitochondria linked to apoptosis in mammalian cells and Bax expression in yeast

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

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1387-1395

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10.1038/SJ.CDD.4401949

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16676005

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