T-cell-independent resistance to infection and generation of immunity to Francisella tularensis.
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TularemiaThe involvement of IL-17A in the murine response to sub-lethal inhalational infection with Francisella tularensisTLR-dependent control of Francisella tularensis infection and host inflammatory responsesMacrophage pro-inflammatory response to Francisella novicida infection is regulated by SHIP.Mucosal immunotherapy for protection from pneumonic infection with Francisella tularensisImportance of B cells, but not specific antibodies, in primary and secondary protective immunity to the intracellular bacterium Francisella tularensis live vaccine strainPurified lipopolysaccharide from Francisella tularensis live vaccine strain (LVS) induces protective immunity against LVS infection that requires B cells and gamma interferon.Host-pathogen interactions and immune evasion strategies in Francisella tularensis pathogenicityCellular and humoral immunity are synergistic in protection against types A and B Francisella tularensis.Intranasal vaccination with a defined attenuated Francisella novicida strain induces gamma interferon-dependent antibody-mediated protection against tularemia.Transfer of immunity against lethal murine Francisella infection by specific antibody depends on host gamma interferon and T cells.CD4+ and CD8+ T-cell-dependent and -independent host defense mechanisms can operate to control and resolve primary and secondary Francisella tularensis LVS infection in miceIKKβ in myeloid cells controls the host response to lethal and sublethal Francisella tularensis LVS infection.Non-FcεR bearing mast cells secrete sufficient interleukin-4 to control Francisella tularensis replication within macrophagesCytokine expression in the liver during the early phase of murine tularemia.Minimal requirements for murine resistance to infection with Francisella tularensis LVS.Coactivating signals for the hepatic lymphocyte gamma interferon response to Francisella tularensisA defined O-antigen polysaccharide mutant of Francisella tularensis live vaccine strain has attenuated virulence while retaining its protective capacity.Protective immunity against tularemia provided by an adenovirus-vectored vaccine expressing Tul4 of Francisella tularensis.Phenotypic, morphological, and functional heterogeneity of splenic immature myeloid cells in the host response to tularemia.CD4-CD8- T cells control intracellular bacterial infections both in vitro and in vivo.Mast cells inhibit intramacrophage Francisella tularensis replication via contact and secreted products including IL-4.MAIT cells are critical for optimal mucosal immune responses during in vivo pulmonary bacterial infection.Oral live vaccine strain-induced protective immunity against pulmonary Francisella tularensis challenge is mediated by CD4+ T cells and antibodies, including immunoglobulin A.An approach to the identification of T cell epitopes in the genomic era: application to Francisella tularensisIdentification of a dominant CD4 T cell epitope in the membrane lipoprotein Tul4 from Francisella tularensis LVS.Humoral and cell-mediated immunity to the intracellular pathogen Francisella tularensis.Global transcriptional response to spermine, a component of the intramacrophage environment, reveals regulation of Francisella gene expression through insertion sequence elements.Working toward the future: insights into Francisella tularensis pathogenesis and vaccine development.IgM in microbial infections: taken for granted?Distinct roles of reactive nitrogen and oxygen species to control infection with the facultative intracellular bacterium Francisella tularensis.Susceptibility to secondary Francisella tularensis live vaccine strain infection in B-cell-deficient mice is associated with neutrophilia but not with defects in specific T-cell-mediated immunity.In vivo clearance of an intracellular bacterium, Francisella tularensis LVS, is dependent on the p40 subunit of interleukin-12 (IL-12) but not on IL-12 p70.Interleukin-12 in infectious diseases.Multiple T cell subsets control Francisella tularensis LVS intracellular growth without stimulation through macrophage interferon gamma receptors.NK cells and gamma interferon coordinate the formation and function of hepatic granulomas in mice infected with the Francisella tularensis live vaccine strain.CD11c expression identifies a population of extrafollicular antigen-specific splenic plasmablasts responsible for CD4 T-independent antibody responses during intracellular bacterial infection.Adaptive Immunity to Francisella tularensis and Considerations for Vaccine Development.
P2860
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P2860
T-cell-independent resistance to infection and generation of immunity to Francisella tularensis.
description
1993 nî lūn-bûn
@nan
1993 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի մարտին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
T-cell-independent resistance ...... ity to Francisella tularensis.
@ast
T-cell-independent resistance ...... ity to Francisella tularensis.
@en
T-cell-independent resistance ...... ity to Francisella tularensis.
@nl
type
label
T-cell-independent resistance ...... ity to Francisella tularensis.
@ast
T-cell-independent resistance ...... ity to Francisella tularensis.
@en
T-cell-independent resistance ...... ity to Francisella tularensis.
@nl
prefLabel
T-cell-independent resistance ...... ity to Francisella tularensis.
@ast
T-cell-independent resistance ...... ity to Francisella tularensis.
@en
T-cell-independent resistance ...... ity to Francisella tularensis.
@nl
P2093
P2860
P1476
T-cell-independent resistance ...... nity to Francisella tularensis
@en
P2093
Fortier AH
Rhinehart-Jones T
Winegar RK
P2860
P304
P407
P577
1993-03-01T00:00:00Z