Proinflammatory cytokine gene expression in the stomach correlates with vaccine-induced protection against Helicobacter pylori infection in mice: an important role for interleukin-17 during the effector phase.
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Vaccine-induced immunity against Helicobacter pylori in the absence of IL-17A.Immune evasion strategies used by Helicobacter pylori.Polymorphisms of the DNA methyltransferase 1 associated with reduced risks of Helicobacter pylori infection and increased risks of gastric atrophyTh1-mediated immunity against Helicobacter pylori can compensate for lack of Th17 cells and can protect mice in the absence of immunization.Subcomponent vaccine based on CTA1-DD adjuvant with incorporated UreB class II peptides stimulates protective Helicobacter pylori immunity.Th1 and Th17 responses to Helicobacter pylori in Bangladeshi infants, children and adults.Defining the Roles of IFN-γ and IL-17A in Inflammation and Protection against Helicobacter pylori Infection.Immunodominant epitope-specific Th1 but not Th17 responses mediate protection against Helicobacter pylori infection following UreB vaccination of BALB/c miceImmunization with Staphylococcus aureus iron regulated surface determinant B (IsdB) confers protection via Th17/IL17 pathway in a murine sepsis modelLeptin, CD4(+) T(reg) and the prospects for vaccination against H. pylori infection.A double mutant heat-labile toxin from Escherichia coli, LT(R192G/L211A), is an effective mucosal adjuvant for vaccination against Helicobacter pylori infection.Vaccinating against Helicobacter pylori in the developing world.Inflammation, immunity, and vaccine development for Helicobacter pylori.Targeting STAT3 in gastric cancer.STAT3: a critical component in the response to Helicobacter pylori infection.The study of H. pylori putative candidate factors for single- and multi-component vaccine development.Induction of mucosal immune responses against Helicobacter pylori infection after sublingual and intragastric route of immunization.IL-4 Protects the Mitochondria Against TNFα and IFNγ Induced Insult During Clearance of Infection with Citrobacter rodentium and Escherichia coli.Host Nonresponsiveness Does not Interfere With Vaccine-Mediated Protection Against Gastric Helicobacter Infection.Production and delivery of Helicobacter pylori NapA in Lactococcus lactis and its protective efficacy and immune modulatory activity.
P2860
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P2860
Proinflammatory cytokine gene expression in the stomach correlates with vaccine-induced protection against Helicobacter pylori infection in mice: an important role for interleukin-17 during the effector phase.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Proinflammatory cytokine gene ...... -17 during the effector phase.
@ast
Proinflammatory cytokine gene ...... -17 during the effector phase.
@en
Proinflammatory cytokine gene ...... -17 during the effector phase.
@nl
type
label
Proinflammatory cytokine gene ...... -17 during the effector phase.
@ast
Proinflammatory cytokine gene ...... -17 during the effector phase.
@en
Proinflammatory cytokine gene ...... -17 during the effector phase.
@nl
prefLabel
Proinflammatory cytokine gene ...... -17 during the effector phase.
@ast
Proinflammatory cytokine gene ...... -17 during the effector phase.
@en
Proinflammatory cytokine gene ...... -17 during the effector phase.
@nl
P2093
P2860
P356
P1476
Proinflammatory cytokine gene ...... -17 during the effector phase.
@en
P2093
Anna Karin Östberg
Anne-Therese Nilsson
Carl-Fredrik Flach
Rene De Waal Malefyt
Sukanya Raghavan
P2860
P304
P356
10.1128/IAI.00756-10
P407
P577
2010-11-15T00:00:00Z