Antibody to receptor activator of NF-κB ligand ameliorates T cell-mediated periodontal bone resorption.
about
Immune cells link obesity-associated type 2 diabetes and periodontitisPorphyromonas gingivalis suppresses adaptive immunity in periodontitis, atherosclerosis, and Alzheimer's diseaseGenetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis.Defective neutrophil recruitment in leukocyte adhesion deficiency type I disease causes local IL-17-driven inflammatory bone loss.Porphyromonas gingivalis exacerbates ligature-induced, RANKL-dependent alveolar bone resorption via differential regulation of Toll-like receptor 2 (TLR2) and TLR4.RNAi-mediated silencing of Atp6i and Atp6i haploinsufficiency prevents both bone loss and inflammation in a mouse model of periodontal diseaseTLR2 signaling and Th2 responses drive Tannerella forsythia-induced periodontal bone loss.Diabetes as a risk factor for medication-related osteonecrosis of the jaw.The Effects of Cigarette Smoke Condensate and Nicotine on Periodontal Tissue in a Periodontitis Model MouseLocal complement-targeted intervention in periodontitis: proof-of-concept using a C5a receptor (CD88) antagonist.Inhibiting periapical lesions through AAV-RNAi silencing of cathepsin K.RNA interference-mediated silencing of Atp6i prevents both periapical bone erosion and inflammation in the mouse model of endodontic diseasePorphyromonas gingivalis infection-associated periodontal bone resorption is dependent on receptor activator of NF-κB ligandThe RANKL-OPG system in clinical periodontology.Recent advances in host defense mechanisms/therapies against oral infectious diseases and consequences for systemic disease.Novel techniques in the development of osteoporosis drug therapy: the osteoclast ruffled-border vacuolar H(+)-ATPase as an emerging target.Aggregatibacter actinomycetemcomitans, a potent immunoregulator of the periodontal host defense system and alveolar bone homeostasis.DC-STAMP Is an Osteoclast Fusogen Engaged in Periodontal Bone Resorption.Soluble RANKL Cleaved from Activated Lymphocytes by TNF-α-Converting Enzyme Contributes to Osteoclastogenesis in Periodontitis.IL-33 Exacerbates Periodontal Disease through Induction of RANKL.T-lymphocyte phenotype and function triggered by Aggregatibacter actinomycetemcomitans is serotype-dependent.Oral administration of all-trans retinoic acid suppresses experimental periodontitis by modulating the Th17/Treg imbalance.Revisiting the Page & Schroeder model: the good, the bad and the unknowns in the periodontal host response 40 years later.Serotype b of Aggregatibacter actinomycetemcomitans increases osteoclast and memory T-lymphocyte activation.
P2860
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P2860
Antibody to receptor activator of NF-κB ligand ameliorates T cell-mediated periodontal bone resorption.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
name
Antibody to receptor activator ...... d periodontal bone resorption.
@ast
Antibody to receptor activator ...... d periodontal bone resorption.
@en
Antibody to receptor activator ...... d periodontal bone resorption.
@nl
type
label
Antibody to receptor activator ...... d periodontal bone resorption.
@ast
Antibody to receptor activator ...... d periodontal bone resorption.
@en
Antibody to receptor activator ...... d periodontal bone resorption.
@nl
prefLabel
Antibody to receptor activator ...... d periodontal bone resorption.
@ast
Antibody to receptor activator ...... d periodontal bone resorption.
@en
Antibody to receptor activator ...... d periodontal bone resorption.
@nl
P2093
P2860
P356
P1476
Antibody to receptor activator ...... d periodontal bone resorption.
@en
P2093
Martin A Taubman
Toshihisa Kawai
Xiaoping Lin
Xiaozhe Han
P2860
P304
P356
10.1128/IAI.00944-10
P407
P577
2010-11-15T00:00:00Z