Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
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Regulation of arsenic trioxide-induced cellular responses by Mnk1 and Mnk2Distinct gene expression profiles in immortalized human urothelial cells exposed to inorganic arsenite and its methylated trivalent metabolitesThe mechanistic basis of arsenicosis: pathogenesis of skin cancerDivergent Effects of Arsenic on NF-κB Signaling in Different Cells or Tissues: A Systematic Review and Meta-AnalysisThe Link between Autoimmunity and Lymphoma: Does NOTCH Signaling Play a Contributing Role?NF-kappaB and IKK as therapeutic targets in cancerColorectal cancer and NF-κB signaling pathwayThe NRF2-mediated oxidative stress response pathway is associated with tumor cell resistance to arsenic trioxide across the NCI-60 panel.Arsenic trioxide-dependent activation of thousand-and-one amino acid kinase 2 and transforming growth factor-beta-activated kinase 1.Phase II study of arsenic trioxide and ascorbic acid for relapsed or refractory lymphoid malignancies: a Wisconsin Oncology Network studyMolecular biology of Hodgkin's and Reed/Sternberg cells in Hodgkin's lymphoma.GLUT1 expression patterns in different Hodgkin lymphoma subtypes and progressively transformed germinal centers.Downregulation of hTERT: an important As2O3 induced mechanism of apoptosis in myelodysplastic syndromeThe role of NF-{kappa}B-1 and NF-{kappa}B-2-mediated resistance to apoptosis in lymphomasSuppression of the proinflammatory response of metastatic melanoma cells increases TRAIL-induced apoptosisArsenite sensitizes human melanomas to apoptosis via tumor necrosis factor alpha-mediated pathway.Arsenic trioxide, a potent inhibitor of NF-kappaB, abrogates allergen-induced airway hyperresponsiveness and inflammation.Tetraarsenictetrasulfide and Arsenic Trioxide Exert Synergistic Effects on Induction of Apoptosis and Differentiation in Acute Promyelocytic Leukemia Cells.Arsenic trioxide for the treatment of myelodysplastic syndromes.Arsenic trioxide and ascorbic acid interfere with the BCL2 family genes in patients with myelodysplastic syndromes: an ex-vivo studyDose-dependent effects of selenite (Se(4+)) on arsenite (As(3+))-induced apoptosis and differentiation in acute promyelocytic leukemia cells.Arsenic induced complete remission in a refractory T-ALL patient with a distinct T-cell clonal evolution without molecular complete remission: A case report.Cadmium-induced apoptosis in rat kidney epithelial cells involves decrease in nuclear factor-kappa B activity.Inhibition of nicotinamide phosphoribosyltransferase and depletion of nicotinamide adenine dinucleotide contribute to arsenic trioxide suppression of oral squamous cell carcinoma.Morphine Attenuated the Cytotoxicity Induced by Arsenic Trioxide in H9c2 Cardiomyocytes.para-Phenylenediamine induces apoptosis through activation of reactive oxygen species-mediated mitochondrial pathway, and inhibition of the NF-κB, mTOR, and Wnt pathways in human urothelial cells.Nuclear translocation of nuclear factor kappa B is regulated by G protein signaling pathway in arsenite-induced apoptosis in HBE cell line.Notch is an essential upstream regulator of NF-κB and is relevant for survival of Hodgkin and Reed-Sternberg cells.Proteasome inhibition in myelodysplastic syndromes and acute myelogenous leukemia cell lines.Bfl-1 is a crucial pro-survival nuclear factor-κB target gene in Hodgkin/Reed-Sternberg cells.Combined treatment with EGFR inhibitors and arsenite upregulated apoptosis in human EGFR-positive melanomas: a role of suppression of the PI3K-AKT pathway.A modified version of galectin-9 induces cell cycle arrest and apoptosis of Burkitt and Hodgkin lymphoma cells.Trolox enhances the anti-lymphoma effects of arsenic trioxide, while protecting against liver toxicity.IkappaBalpha independent induction of NF-kappaB and its inhibition by DHMEQ in Hodgkin/Reed-Sternberg cells.Activation of the mitogen- and stress-activated kinase 1 by arsenic trioxide.Dual treatment with COX-2 inhibitor and sodium arsenite leads to induction of surface Fas Ligand expression and Fas-Ligand-mediated apoptosis in human melanoma cells.Hodgkin's lymphoma cells are efficiently engrafted and tumor marker CD30 is expressed with constitutive nuclear factor-kappaB activity in unconditioned NOD/SCID/gammac(null) mice.Signaling via the anti-CD30 mAb SGN-30 sensitizes Hodgkin's disease cells to conventional chemotherapeutics.Suppression of NF-kappaB and NF-kappaB-regulated gene expression by sulforaphane and PEITC through IkappaBalpha, IKK pathway in human prostate cancer PC-3 cells.Phosphoinositide 3-kinase/Akt involvement in arsenic trioxide resistance of human leukemia cells.
P2860
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P2860
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@ast
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@en
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@nl
type
label
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@ast
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@en
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@nl
prefLabel
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@ast
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@en
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.
@nl
P2093
P1433
P1476
Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis
@en
P2093
Andreas Lietz
Bernd Dorken
Claus Scheidereit
Kurt Bommert
Martin Janz
Stephan Mathas
P304
P356
10.1182/BLOOD-2002-04-1154
P407
P577
2003-04-03T00:00:00Z