The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming - Wikidata - awgldk - TriplyDB

The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

http://www.wikidata.org/entity/Q34533755

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Multifunctional DDX3: dual roles in various cancer development and its related signaling pathways Emerging Transcriptional Mechanisms in the Regulation of Epithelial to Mesenchymal Transition and Cellular Plasticity in the Kidney Distinctive properties of metastasis-initiating cells Choices for Induction of Pluripotency: Recent Developments in Human Induced Pluripotent Stem Cell Reprogramming Strategies Promyelocytic Leukemia Protein Is an Essential Regulator of Stem Cell Pluripotency and Somatic Cell Reprogramming.Sequential EMT-MET induces neuronal conversion through Sox2.Oncostatin M promotes cancer cell plasticity through cooperative STAT3-SMAD3 signaling.Coronary Artery Development: Progenitor Cells and Differentiation Pathways.Origin of cells and network information Canonical MicroRNA Activity Facilitates but May Be Dispensable for Transcription Factor-Mediated Reprogramming.Mechanisms underlying the formation of induced pluripotent stem cells.Genome-wide gene expression analyses reveal unique cellular characteristics related to the amenability of HPC/HSCs into high-quality induced pluripotent stem cells Regulatory Roles of Dclk1 in Epithelial Mesenchymal Transition and Cancer Stem Cells The mesenchymal transcription factor SNAI-1 instructs human liver specification Clinical impact of studying epithelial-mesenchymal plasticity in pluripotent stem cells.Reprogramming barriers and enhancers: strategies to enhance the efficiency and kinetics of induced pluripotency MicroRNA-Mediated Reprogramming of Somatic Cells into Neural Stem Cells or Neurons.Naturally occurring compounds acting as potent anti-metastatic agents and their suppressing effects on Hedgehog and WNT/β-catenin signalling pathways.TGF-β Family Signaling in Embryonic and Somatic Stem-Cell Renewal and Differentiation.Brief Report: Inhibition of miR-145 Enhances Reprogramming of Human Dermal Fibroblasts to Induced Pluripotent Stem Cells.Rad51 Regulates Reprogramming Efficiency through DNA Repair Pathway.Direct comparison of distinct naive pluripotent states in human embryonic stem cells.miR302 regulates SNAI1 expression to control mesangial cell plasticity.ZSCAN10 expression corrects the genomic instability of iPSCs from aged donors.Reprogramming to pluripotency does not require transition through a primitive streak-like state.Silencing the Snail-dependent RNA splice regulator ESRP1 drives malignant transformation of human pulmonary epithelial cells.Suppression of the ERK-SRF axis facilitates somatic cell reprogramming.Snail knockdown reverses stemness and inhibits tumour growth in ovarian cancer.Fine-Tuning Mybl2 Is Required for Proper Mesenchymal-to-Epithelial Transition during Somatic Reprogramming

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The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

http://www.wikidata.org/entity/Q34533755

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2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

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The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

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The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

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type

label

The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

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The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

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The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

@nl

prefLabel

The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

@ast

The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

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The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

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J.STEMCR.2014.09.008

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Stem Cell Reports

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The epithelial-mesenchymal transition factor SNAIL paradoxically enhances reprogramming

@en

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John T Powers

http://www.w3.org/2001/XMLSchema#string

Juli J Unternaehrer

http://www.w3.org/2001/XMLSchema#string

Kitai Kim

http://www.w3.org/2001/XMLSchema#string

Marcella Cesana

http://www.w3.org/2001/XMLSchema#string

Rui Zhao

http://www.w3.org/2001/XMLSchema#string

Sutheera Ratanasirintrawoot

http://www.w3.org/2001/XMLSchema#string

Tamer Onder

http://www.w3.org/2001/XMLSchema#string

Tsukasa Shibue

http://www.w3.org/2001/XMLSchema#string

P2860

A feedback loop comprising lin-28 and let-7 controls pre-let-7 maturation during neural stem-cell commitment

Selective blockade of microRNA processing by Lin28

The Lin28/let-7 axis regulates glucose metabolism

A long noncoding RNA controls muscle differentiation by functioning as a competing endogenous RNA

The epithelial-mesenchymal transition generates cells with properties of stem cells

Up-regulation of miR-200 and let-7 by natural agents leads to the reversal of epithelial-to-mesenchymal transition in gemcitabine-resistant pancreatic cancer cells

Induced pluripotent stem cell lines derived from human somatic cells

Induction of pluripotent stem cells from adult human fibroblasts by defined factors

Reprogramming of human somatic cells to pluripotency with defined factors

Snail blocks the cell cycle and confers resistance to cell death

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691-698

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scholarly article

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10.1016/J.STEMCR.2014.09.008

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5

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3

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George Q. Daley

Robert Weinberg

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2014-10-11T00:00:00Z

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266945607

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25316190

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4235745

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