Opposite role of tumor necrosis factor receptors in dextran sulfate sodium-induced colitis in mice.

Opposite role of tumor necrosis factor receptors in dextran sulfate sodium-induced colitis in mice.

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http://www.wikidata.org/entity/Q34534877
Redeeming an old foe: protective as well as pathophysiological roles for tumor necrosis factor in inflammatory bowel diseaseA gut microbial metabolite of linoleic acid, 10-hydroxy-cis-12-octadecenoic acid, ameliorates intestinal epithelial barrier impairment partially via GPR40-MEK-ERK pathway.The Tumor Suppressor Hace1 Is a Critical Regulator of TNFR1-Mediated Cell Fate.TNFR2 activates MLCK-dependent tight junction dysregulation to cause apoptosis-mediated barrier loss and experimental colitis.TNFR2 expression by CD4 effector T cells is required to induce full-fledged experimental colitisNLRP6 function in inflammatory monocytes reduces susceptibility to chemically induced intestinal injury.Dextran sulfate sodium (DSS)-induced colitis in mice.TNF blocking therapies and immunomonitoring in patients with inflammatory bowel diseaseCell death at the intestinal epithelial front line.G2A Signaling Dampens Colitic Inflammation via Production of IFN-γ.Protective role of tumor necrosis factor (TNF) receptors in chronic intestinal inflammation: TNFR1 ablation boosts systemic inflammatory response.Dextran sulphate sodium (DSS) causes intestinal histopathology and inflammatory changes consistent with increased gut leakiness in chickens.Targeting tumour necrosis factor receptor 1 assembly reverses Th17-mediated colitis through boosting a Th2 response.Effect of genetic deletion or pharmacological antagonism of tumor necrosis factor alpha on colitis-associated carcinogenesis in mice.Neutrophil infiltration favors colitis-associated tumorigenesis by activating the interleukin-1 (IL-1)/IL-6 axis.MDSCs are involved in the protumorigenic potentials of GM-CSF in colitis-associated cancer.Critical Role of Tumor Necrosis Factor Signaling in Mesenchymal Stem Cell-Based Therapy for Autoimmune and Inflammatory Diseases
P2860

Opposite role of tumor necrosis factor receptors in dextran sulfate sodium-induced colitis in mice.

Item
http://www.wikidata.org/entity/Q34534877
description
2012 nî lūn-bûn
@nan
2012 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@ast
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@en
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@nl
type
label
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@ast
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@en
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@nl
prefLabel
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@ast
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@en
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P698
P932
P356
P1433
P1476
Opposite role of tumor necrosi ...... odium-induced colitis in mice.
@en
P2093
P2860
P304
P31
P356
10.1371/JOURNAL.PONE.0052924
P407
P433
P478
P50
P577
P5875
P698
P932