Exercise training restores cardiac protein quality control in heart failure. - Wikidata - awgldk - TriplyDB

Exercise training restores cardiac protein quality control in heart failure.

Exercise training restores cardiac protein quality control in heart failure.

http://www.wikidata.org/entity/Q34541132

about

Personalized preventive medicine: genetics and the response to regular exercise in preventive interventions Mitochondrial Quality Control in Cardiac Diseases What can we learn about treating heart failure from the heart's response to acute exercise? Focus on the coronary microcirculation New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction Proteasomal and lysosomal protein degradation and heart disease.Aerobic interval training attenuates mitochondrial dysfunction in rats post-myocardial infarction: roles of mitochondrial network dynamics Aldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling.Targeting aldehyde dehydrogenase 2: new therapeutic opportunities.Exercise training reduces cardiac dysfunction and remodeling in ovariectomized rats submitted to myocardial infarction.Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of Alda-1 Impact of leucine supplementation on exercise training induced anti-cardiac remodeling effect in heart failure mice.Long-term low-level laser therapy promotes an increase in maximal oxygen uptake and exercise performance in a dose-dependent manner in Wistar rats.Light-emitting diode therapy (LEDT) improves functional capacity in rats with heart failure.Benefits of cardiac rehabilitation in heart failure patients according to etiology: INCARD French study.Maximal oxygen uptake and exercise tolerance are improved in rats with heart failure subjected to low-level laser therapy associated with resistance training.Regulation and therapeutic strategies of 4-hydroxy-2-nonenal metabolism in heart disease.Increased 4-hydroxy-2-nonenal-induced proteasome dysfunction is correlated with cardiac damage in streptozotocin-injected rats with isoproterenol infusion.Oxidative stress and lipotoxicity Cardiac rehabilitation improves the blood plasma properties of cardiac patients.Beneficial and harmful effects of exercise in hypertensive patients: the role of oxidative stress.Disruption of mitochondrial quality control in peripheral artery disease: New therapeutic opportunities.Aerobic exercise training rescues cardiac protein quality control and blunts endoplasmic reticulum stress in heart failure rats.The protective role of small heat shock proteins in cardiac diseases: key role in atrial fibrillation.Increased clearance of reactive aldehydes and damaged proteins in hypertension-induced compensated cardiac hypertrophy: impact of exercise training.Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure.Experimental Evidences Supporting the Benefits of Exercise Training in Heart Failure.Aerobic exercise training improves oxidative stress and ubiquitin proteasome system activity in heart of spontaneously hypertensive rats.Influence of training in the dark or light phase on physiologic and metabolic parameters ofWistarrats submitted to aerobic exercise Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy

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Exercise training restores cardiac protein quality control in heart failure.

http://www.wikidata.org/entity/Q34541132

description

2012 nî lūn-bûn

@nan

2012 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Exercise training restores cardiac protein quality control in heart failure.

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Exercise training restores cardiac protein quality control in heart failure.

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Exercise training restores cardiac protein quality control in heart failure.

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type

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Exercise training restores cardiac protein quality control in heart failure.

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Exercise training restores cardiac protein quality control in heart failure.

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Exercise training restores cardiac protein quality control in heart failure.

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prefLabel

Exercise training restores cardiac protein quality control in heart failure.

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Exercise training restores cardiac protein quality control in heart failure.

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Exercise training restores cardiac protein quality control in heart failure.

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JOURNAL.PONE.0052764

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Exercise training restores cardiac protein quality control in heart failure.

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Alicia J Kowaltowski

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Julio C B Ferreira

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Luiz R G Bechara

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Patricia C Brum

http://www.w3.org/2001/XMLSchema#string

Paulo M M Dourado

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Telma F Cunha

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Vanessa O Zambelli

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P2860

Activation of aldehyde dehydrogenase-2 reduces ischemic damage to the heart

2009 focused update incorporated into the ACC/AHA 2005 Guidelines for the Diagnosis and Management of Heart Failure in Adults: a report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice Guidelines: de

Mitochondria in heart failure

Protein quality control disruption by PKCβII in heart failure; rescue by the selective PKCβII inhibitor, βIIV5-3

Catalytic site-specific inhibition of the 20S proteasome by 4-hydroxynonenal.

Modification of Cytochrome c by 4-hydroxy- 2-nonenal: evidence for histidine, lysine, and arginine-aldehyde adducts.

Ischaemic preconditioning improves proteasomal activity and increases the degradation of deltaPKC during reperfusion.

Destruction of Myc by ubiquitin-mediated proteolysis: cancer-associated and transforming mutations stabilize Myc.

Physical exercise regulates p53 activity targeting SCO2 and increases mitochondrial COX biogenesis in cardiac muscle with age.

Mitochondrial aldehyde dehydrogenase and cardiac diseases.

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12

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Bruno Barros Queliconi

Juliane C Campos

Daria Mochly-Rosen

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2012-12-27T00:00:00Z

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quality control

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