Acrolein inhalation suppresses lipopolysaccharide-induced inflammatory cytokine production but does not affect acute airways neutrophilia - Wikidata - awgldk - TriplyDB

Acrolein inhalation suppresses lipopolysaccharide-induced inflammatory cytokine production but does not affect acute airways neutrophilia

Acrolein inhalation suppresses lipopolysaccharide-induced inflammatory cytokine production but does not affect acute airways neutrophilia

http://www.wikidata.org/entity/Q34543680

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Protective effects of aerobic exercise on acute lung injury induced by LPS in mice Acute Acrolein Exposure Induces Impairment of Vocal Fold Epithelial Barrier Function Exposure to acrolein by inhalation causes platelet activation Oral exposure to acrolein exacerbates atherosclerosis in apoE-null mice.Molecular mechanisms of acrolein toxicity: relevance to human disease Acrolein exposure is associated with increased cardiovascular disease risk.Modulation of NF-κB and hypoxia-inducible factor--1 by S-nitrosoglutathione does not alter allergic airway inflammation in mice.The tobacco smoke component, acrolein, suppresses innate macrophage responses by direct alkylation of c-Jun N-terminal kinase.Inhalation of the reactive aldehyde acrolein promotes antigen sensitization to ovalbumin and enhances neutrophilic inflammation.Acrolein exposure suppresses antigen-induced pulmonary inflammation.Janus-faced Acrolein prevents allergy but accelerates tumor growth by promoting immunoregulatory Foxp3+ cells: Mouse model for passive respiratory exposure Acrolein - a pulmonary hazard.Acrolein effects in pulmonary cells: relevance to chronic obstructive pulmonary disease.Factors affecting susceptibility to Mycobacterium tuberculosis: a close view of immunological defence mechanism.The tobacco smoke component acrolein induces glucocorticoid resistant gene expression via inhibition of histone deacetylase.Matrix metalloproteinase-14 mediates a phenotypic shift in the airways to increase mucin production.Acrolein and thiol-reactive electrophiles suppress allergen-induced innate airway epithelial responses by inhibition of DUOX1 and EGFR Bacteria challenge in smoke-exposed mice exacerbates inflammation and skews the inflammatory profile.Alda-1 Protects Against Acrolein-Induced Acute Lung Injury and Endothelial Barrier Dysfunction.Proposed Mode of Action for Acrolein Respiratory Toxicity Associated with Inhaled Tobacco Smoke.

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P2860

Acrolein inhalation suppresses lipopolysaccharide-induced inflammatory cytokine production but does not affect acute airways neutrophilia

http://www.wikidata.org/entity/Q34543680

description

2008 nî lūn-bûn

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2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հուլիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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type

label

Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

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JIMMUNOL.181.1.736

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Journal of Immunology

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Acrolein inhalation suppresses ...... ect acute airways neutrophilia

@en

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Albert van der Vliet

http://www.w3.org/2001/XMLSchema#string

David Itiro Kasahara

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Ziryan Othman

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P2860

Acrolein is a major cigarette-related lung cancer agent: Preferential binding at p53 mutational hotspots and inhibition of DNA repair

Chemistry and biochemistry of 4-hydroxynonenal, malonaldehyde and related aldehydes

The locus of tumor necrosis factor-alpha action in lung inflammation

Acrolein activates mitogen-activated protein kinase signal transduction pathways in rat vascular smooth muscle cells

Interleukin-12 and the regulation of innate resistance and adaptive immunity

Sequential recruitment of neutrophils into lung and bronchoalveolar lavage fluid in LPS-induced acute lung injury.

ICAM-1 and LFA-1 play critical roles in LPS-induced neutrophil recruitment into the alveolar space.

Acrolein inhibits cytokine gene expression by alkylating cysteine and arginine residues in the NF-kappaB1 DNA binding domain.

Lung glutathione and oxidative stress: implications in cigarette smoke-induced airway disease.

Current status of acrolein as a lipid peroxidation product.

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736-745

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10.4049/JIMMUNOL.181.1.736

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Albert van der Vliet

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5289029

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18566440

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lipopolysaccharide

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3031871

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