Modulation of cardiac macrophages by phosphatidylserine-presenting liposomes improves infarct repair. - Wikidata - awgldk - TriplyDB

Modulation of cardiac macrophages by phosphatidylserine-presenting liposomes improves infarct repair.

Modulation of cardiac macrophages by phosphatidylserine-presenting liposomes improves infarct repair.

http://www.wikidata.org/entity/Q34549960

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How Biomaterials Can Influence Various Cell Types in the Repair and Regeneration of the Heart after Myocardial Infarction Modulators of Macrophage Polarization Influence Healing of the Infarcted Myocardium Nanoparticles for Cardiovascular Imaging and Therapeutic Delivery, Part 1: Compositions and Features Apoptotic cell clearance: basic biology and therapeutic potential.Resolution of inflammation: an integrated view The physiology of cardiovascular disease and innovative liposomal platforms for therapy "Eat me" imaging and therapy Monocyte and/or macrophage infiltration of heart after myocardial infarction: MR imaging by using T1-shortening liposomes.Towards programming immune tolerance through geometric manipulation of phosphatidylserine.Fragile TIM-4-expressing tissue resident macrophages are migratory and immunoregulatory.Macrophages in Tissue Repair, Regeneration, and Fibrosis.Passive targeting of lipid-based nanoparticles to mouse cardiac ischemia-reperfusion injury.Macrophage targeted theranostics as personalized nanomedicine strategies for inflammatory diseases.Modulation of mononuclear phagocyte inflammatory response by liposome-encapsulated voltage gated sodium channel inhibitor ameliorates myocardial ischemia/reperfusion injury in rats.Silencing collapsin response mediator protein-2 reprograms macrophage phenotype and improves infarct healing in experimental myocardial infarction model Nanoparticles targeting the infarcted heart Use of autoantigen-loaded phosphatidylserine-liposomes to arrest autoimmunity in type 1 diabetes Effect of pegylated phosphatidylserine-containing liposomes in experimental chronic arthritis Micro- and Nanoparticles for Treating Cardiovascular Disease.Nanoparticles containing a liver X receptor agonist inhibit inflammation and atherosclerosis Modulation of macrophage efferocytosis in inflammation.In vivo targeting of inflammation-associated myeloid-related protein 8/14 via gadolinium immunonanoparticles.Theranostic nanoemulsions for macrophage COX-2 inhibition in a murine inflammation model.Enhancement of Anti-Inflammatory Activity of Curcumin Using Phosphatidylserine-Containing Nanoparticles in Cultured Macrophages Improving selective targeting to macrophage subpopulations through modifying liposomes with arginine based materials.Differential Ly-6C expression identifies the recruited macrophage phenotype, which orchestrates the regression of murine liver fibrosis Endometriosis, a disease of the macrophage Recent Advances in Targeted, Self-Assembling Nanoparticles to Address Vascular Damage Due to Atherosclerosis.Myeloid-Epithelial-Reproductive Receptor Tyrosine Kinase and Milk Fat Globule Epidermal Growth Factor 8 Coordinately Improve Remodeling After Myocardial Infarction via Local Delivery of Vascular Endothelial Growth Factor Fibrocytes: emerging effector cells in chronic inflammation Multimodality imaging demonstrates trafficking of liposomes preferentially to ischemic myocardium.Interleukin-2/Anti-Interleukin-2 Immune Complex Attenuates Cardiac Remodeling after Myocardial Infarction through Expansion of Regulatory T Cells.Monocyte and macrophage contributions to cardiac remodeling.Nanocomposite hydrogels: an emerging biomimetic platform for myocardial therapy and tissue engineering.Increase in cholinergic modulation with pyridostigmine induces anti-inflammatory cell recruitment soon after acute myocardial infarction in rats Alternatively activated macrophages determine repair of the infarcted adult murine heart.Cross-talk between macrophages and atrial myocytes in atrial fibrillation Enhanced efferocytosis of apoptotic cardiomyocytes through myeloid-epithelial-reproductive tyrosine kinase links acute inflammation resolution to cardiac repair after infarction.Myelin alters the inflammatory phenotype of macrophages by activating PPARs.HCMV reprogramming of infected monocyte survival and differentiation: a Goldilocks phenomenon.

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Modulation of cardiac macrophages by phosphatidylserine-presenting liposomes improves infarct repair.

http://www.wikidata.org/entity/Q34549960

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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Modulation of cardiac macropha ...... somes improves infarct repair.

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PNAS.1015623108

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Proceedings of the National Academy of Sciences of the United States of America

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Modulation of cardiac macropha ...... somes improves infarct repair.

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P2093

Jonathan Leor

http://www.w3.org/2001/XMLSchema#string

Micha S Feinberg

http://www.w3.org/2001/XMLSchema#string

Smadar Cohen

http://www.w3.org/2001/XMLSchema#string

Tamar Ben Mordechai

http://www.w3.org/2001/XMLSchema#string

Tamar Harel-Adar

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Yoram Amsalem

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P2860

The phosphatidylserine receptor has essential functions during embryogenesis but not in apoptotic cell removal

Alternative activation of macrophages

Immunogenicity of apoptotic cells in vivo: role of antigen load, antigen-presenting cells, and cytokines

Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Exposure of phosphatidylserine on the surface of apoptotic lymphocytes triggers specific recognition and removal by macrophages

Delay of neutrophil apoptosis in acute coronary syndromes.

Macrophage roles following myocardial infarction

The inflammatory response in myocardial infarction.

Phagocytosis of apoptotic cells and the resolution of inflammation.

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21245355

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3033268

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