Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis. - Wikidata - awgldk - TriplyDB

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

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Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

http://www.wikidata.org/entity/Q34550706

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2017 nî lūn-bûn

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2017 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2017 թվականի փետրվարին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

name

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@ast

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@en

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@nl

type

label

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@ast

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@en

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@nl

prefLabel

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@ast

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@en

Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@nl

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J.NEO.2017.01.002

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Loss of Pin1 Suppresses Hedgehog-Driven Medulloblastoma Tumorigenesis.

@en

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Andrzej A Dlugosz

http://www.w3.org/2001/XMLSchema#string

Ashok Srinivasan

http://www.w3.org/2001/XMLSchema#string

Chiyoko Uchida

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Honglai Zhang

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James M Olson

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Kimberly Ha

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Lowell Evan Michael

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Mariarita Santi

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Rork Kuick

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Sandra Camelo-Piragua

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P2860

Function of WW domains as phosphoserine- or phosphothreonine-binding modules

Pin1 regulates turnover and subcellular localization of beta-catenin by inhibiting its interaction with APC

The prolyl-isomerase Pin1 is a Notch1 target that enhances Notch1 activation in cancer

The essential mitotic peptidyl-prolyl isomerase Pin1 binds and regulates mitosis-specific phosphoproteins

Molecular subgroups of medulloblastoma: the current consensus

The magnitude of hedgehog signaling activity defines skin tumor phenotype

Prolyl isomerase Pin1 as a molecular switch to determine the fate of phosphoproteins

Structural and functional analysis of the mitotic rotamase Pin1 suggests substrate recognition is phosphorylation dependent

Genome sequencing of SHH medulloblastoma predicts genotype-related response to smoothened inhibition

Vismodegib Exerts Targeted Efficacy Against Recurrent Sonic Hedgehog-Subgroup Medulloblastoma: Results From Phase II Pediatric Brain Tumor Consortium Studies PBTC-025B and PBTC-032.

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216-225

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scholarly article

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10.1016/J.NEO.2017.01.002

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3

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19

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Jean-François Rual

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2017-02-03T00:00:00Z

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28167297

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medulloblastoma

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5293723

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