Expression of ARC (apoptosis repressor with caspase recruitment domain), an antiapoptotic protein, is strongly prognostic in AML
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A critical role for the protein apoptosis repressor with caspase recruitment domain in hypoxia-induced pulmonary hypertensionAutophagic program is regulated by miR-325Matrine induces Akt/mTOR signalling inhibition-mediated autophagy and apoptosis in acute myeloid leukaemia cells.Apoptosis repressor with caspase recruitment domain modulates second mitochondrial-derived activator of caspases mimetic-induced cell death through BIRC2/MAP3K14 signalling in acute myeloid leukaemiaApoptosis inhibitor ARC promotes breast tumorigenesis, metastasis, and chemoresistanceThe Cell Death Inhibitor ARC Is Induced in a Tissue-Specific Manner by Deletion of the Tumor Suppressor Gene Men1, but Not Required for Tumor Development and Growth.Survivin is highly expressed in CD34(+)38(-) leukemic stem/progenitor cells and predicts poor clinical outcomes in AMLThe apoptosis inhibitor ARC alleviates the ER stress response to promote β-cell survival.Bone marrow niche-mediated survival of leukemia stem cells in acute myeloid leukemia: Yin and Yang.Anti-apoptotic ARC protein confers chemoresistance by controlling leukemia-microenvironment interactions through a NFκB/IL1β signaling network.Interplay of Phosphorylated Apoptosis Repressor with CARD, Casein Kinase-2 and Reactive Oxygen Species in Regulating Endothelin-1-Induced Cardiomyocyte HypertrophyOuabain-induced apoptosis in cochlear hair cells and spiral ganglion neurons in vitro.Mechanisms of cell death in heart disease.Cathepsin G is broadly expressed in acute myeloid leukemia and is an effective immunotherapeutic target.Apoptosis repressor with caspase recruitment domain is regulated by MAPK/PI3K and confers drug resistance and survival advantage to AML.Synergistic targeting of AML stem/progenitor cells with IAP antagonist birinapant and demethylating agents.MicroRNA-185 regulates chemotherapeutic sensitivity in gastric cancer by targeting apoptosis repressor with caspase recruitment domain.Affinity proteomics: the role of specific binding reagents in human proteome analysis.A myeloid tumor suppressor role for NOL3Robust microarray production of freshly expressed proteins in a human milieuThe apoptosis repressor with a CARD domain (ARC) gene is a direct hypoxia-inducible factor 1 target gene and promotes survival and proliferation of VHL-deficient renal cancer cells.Focal Adhesion Kinase as a Potential Target in AML and MDS.Cathepsin G Is Expressed by Acute Lymphoblastic Leukemia and Is a Potential Immunotherapeutic Target.Disruption of Wnt/β-catenin exerts anti-leukemia activity and synergizes with FLT3 inhibition in FLT3-mutant acute myeloid leukemia.
P2860
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P2860
Expression of ARC (apoptosis repressor with caspase recruitment domain), an antiapoptotic protein, is strongly prognostic in AML
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2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@ast
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@en
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@nl
type
label
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@ast
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@en
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@nl
prefLabel
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@ast
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@en
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@nl
P2093
P2860
P50
P1433
P1476
Expression of ARC (apoptosis r ...... is strongly prognostic in AML
@en
P2093
Bing Z Carter
Deborah A Thomas
Duncan H Mak
Erich Koller
Nianxiang Zhang
Steven M Kornblau
Yi Hua Qiu
P2860
P304
P356
10.1182/BLOOD-2010-04-280503
P407
P577
2010-11-01T00:00:00Z