A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance

A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance

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http://www.wikidata.org/entity/Q34560402

Q34560402

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Thrombopoietin as biomarker and mediator of cardiovascular damage in critical diseases Low systemic vascular resistance: differential diagnosis and outcome Clinical review: Myocardial depression in sepsis and septic shock Sepsis-induced myocardial dysfunction: pathophysiology and management A review of sepsis-induced cardiomyopathy Novel regulatory mechanism of cardiomyocyte contractility involving ICAM-1 and the cytoskeleton Effects of burn injury on myocardial signaling and cytokine secretion: Possible role of PKC Elevated cardiac troponin I in sepsis and septic shock: no evidence for thrombus associated myocardial necrosis [Septic shock and septic cardiomyopathy].Characterization of human septic sera induced gene expression modulation in human myocytes.Caspase inhibition prevents cardiac dysfunction and heart apoptosis in a rat model of sepsis.Myocardial oxidative stress correlates with left ventricular dysfunction on strain echocardiography in a rodent model of sepsis.Cardiomyopathy associated with high-dose interleukin-2 therapy.Indications, technical considerations, and strategies for renal replacement therapy in the intensive care unit.Cardiac Troponin Release is Associated with Biomarkers of Inflammation and Ventricular Dilatation During Critical Illness.Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes Gender dimorphism of the cardiac dysfunction in murine sepsis: signalling mechanisms and age-dependency.Sepsis: clinical dilemmas.Tumor necrosis factor-alpha gene and protein expression in adult feline myocardium after endotoxin administration.The lethal effects of cytokine-induced nitric oxide on cardiac myocytes are blocked by nitric oxide synthase antagonism or transforming growth factor beta Severe postpartum sepsis with prolonged myocardial dysfunction: a case report.Role of endotoxemia in cardiovascular dysfunction and mortality. Escherichia coli and Staphylococcus aureus challenges in a canine model of human septic shock Multiple organ dysfunction syndrome in humans and animals.Do cardiomyocytes mount an immune response to Group A Streptococcus?Complement dependency of cardiomyocyte release of mediators during sepsis Mesenteric lymph from rats with trauma-hemorrhagic shock causes abnormal cardiac myocyte function and induces myocardial contractile dysfunction.Emergency management of meningococcal disease.The role of nitric oxide in experimental murine sepsis due to pyrogenic exotoxin A-producing Streptococcus pyogenes.Tumour necrosis factor alpha gene polymorphism: a predisposing factor to non-ischaemic myocardial dysfunction?Sepsis-induced cardiomyopathy.Mitogen-activated protein kinase phosphatase-1 inhibits myocardial TNF-α expression and improves cardiac function during endotoxemia.Gram-negative bacteremia produces both severe systolic and diastolic cardiac dysfunction in a canine model that simulates human septic shock The tissue distribution of tumor necrosis factor biosynthesis during endotoxemia Quercetin reduces inflammatory responses in LPS-stimulated cardiomyoblasts.Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction Levosimendan for Septic Myocardial Dysfunction: Not Yet Ready for Prime Time An essential role for complement C5a in the pathogenesis of septic cardiac dysfunction.Early and innovative interventions for severe sepsis and septic shock: taking advantage of a window of opportunity.Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock.Cardiorespiratory control and cytokine profile in response to heat stress, hypoxia, and lipopolysaccharide (LPS) exposure during early neonatal period.

P2860

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P2860

A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance

Item

http://www.wikidata.org/entity/Q34560402

description

1985 nî lūn-bûn

@nan

1985 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1985 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

1985年の論文

@ja

1985年論文

@yue

1985年論文

@zh-hant

1985年論文

@zh-hk

1985年論文

@zh-mo

1985年論文

@zh-tw

1985年论文

@wuu