The MAGE proteins: emerging roles in cell cycle progression, apoptosis, and neurogenetic disease.
about
Interactions between the Nse3 and Nse4 components of the SMC5-6 complex identify evolutionarily conserved interactions between MAGE and EID FamiliesNecdin-related MAGE proteins differentially interact with the E2F1 transcription factor and the p75 neurotrophin receptorDAMAGE, a novel alpha-dystrobrevin-associated MAGE protein in dystrophin complexesMAGE-RING protein complexes comprise a family of E3 ubiquitin ligasesIdentification of the proteins, including MAGEG1, that make up the human SMC5-6 protein complexBRCA2 suppresses cell proliferation via stabilizing MAGE-D1A cleaved form of MAGE-A4 binds to Miz-1 and induces apoptosis in human cellsComposition and architecture of the Schizosaccharomyces pombe Rad18 (Smc5-6) complexMAGE-A4 interacts with the liver oncoprotein gankyrin and suppresses its tumorigenic activityAndrogen receptor molecular biology and potential targets in prostate cancerSeparate necdin domains bind ARNT2 and HIF1alpha and repress transcriptionEstablishment of six new human biliary tract carcinoma cell lines and identification of MAGEH1 as a candidate biomarker for predicting the efficacy of gemcitabine treatment.Qri2/Nse4, a component of the essential Smc5/6 DNA repair complex.UNC5H1 induces apoptosis via its juxtamembrane region through an interaction with NRAGEThe p75 neurotrophin receptor interacts with multiple MAGE proteinshNRAGE, a human neurotrophin receptor interacting MAGE homologue, regulates p53 transcriptional activity and inhibits cell proliferationNecdin interacts with the Msx2 homeodomain protein via MAGE-D1 to promote myogenic differentiation of C2C12 cellsMagel2, a Prader-Willi syndrome candidate gene, modulates the activities of circadian rhythm proteins in cultured cellsThe mouse Mageb18 gene encodes a ubiquitously expressed type I MAGE protein and regulates cell proliferation and apoptosis in melanoma B16-F0 cellsNSCL-1 and NSCL-2 synergistically determine the fate of GnRH-1 neurons and control necdin gene expressionDestabilized SMC5/6 complex leads to chromosome breakage syndrome with severe lung diseaseSensory defects in Necdin deficient mice result from a loss of sensory neurons correlated within an increase of developmental programmed cell death.Screening a genome-wide S. pombe deletion library identifies novel genes and pathways involved in genome stability maintenanceMaged1, a new regulator of skeletal myogenic differentiation and muscle regeneration.Necdin promotes ubiquitin-dependent degradation of PIAS1 SUMO E3 ligaseThe p75 neurotrophin receptor localization in blood-CSF barrier: expression in choroid plexus epitheliumhMAGEA2 promotes progression of breast cancer by regulating Akt and Erk1/2 pathways.NRAGE, a p75 neurotrophin receptor-interacting protein, induces caspase activation and cell death through a JNK-dependent mitochondrial pathway.Necdin enhances myoblasts survival by facilitating the degradation of the mediator of apoptosis CCAR1/CARP1Functional modulation of the metastatic suppressor Nm23-H1 by oncogenic virusesThe neurotrophin receptor p75(NTR): novel functions and implications for diseases of the nervous system.Centrosomal localisation of the cancer/testis (CT) antigens NY-ESO-1 and MAGE-C1 is regulated by proteasome activity in tumour cellsMapping of NRAGE domains reveals clues to cell viability in BMP signaling.High expression of MAGE-A9 in tumor and stromal cells of non-small cell lung cancer was correlated with patient poor survivalImmunity to melanoma: unraveling the relation of tumor immunity and autoimmunity.Gene systems network inferred from expression profiles in hepatocellular carcinogenesis by graphical Gaussian model.Insulin/IGF-I regulation of necdin and brown adipocyte differentiation via CREB- and FoxO1-associated pathways.Dependence receptors: what is the mechanism?A pathway for the control of anoikis sensitivity by E-cadherin and epithelial-to-mesenchymal transition.Impaired hypothalamic regulation of endocrine function and delayed counterregulatory response to hypoglycemia in Magel2-null mice.
P2860
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P2860
The MAGE proteins: emerging roles in cell cycle progression, apoptosis, and neurogenetic disease.
description
2002 nî lūn-bûn
@nan
2002 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի մարտին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@ast
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@en
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@nl
type
label
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@ast
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@en
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@nl
prefLabel
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@ast
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@en
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@nl
P2860
P356
P1476
The MAGE proteins: emerging ro ...... sis, and neurogenetic disease.
@en
P2093
Amir Salehi
Philip A Barker
P2860
P304
P356
10.1002/JNR.10160
P577
2002-03-01T00:00:00Z