Genomic imprinting is a barrier to parthenogenesis in mammals. - Wikidata - awgldk - TriplyDB

Genomic imprinting is a barrier to parthenogenesis in mammals.

Genomic imprinting is a barrier to parthenogenesis in mammals.

http://www.wikidata.org/entity/Q34566100

about

Virgin birth in a hammerhead shark Successive virgin births of viable male progeny in the checkered gartersnake,Thamnophis marcianus Parthenogenetic activation of bovine oocytes using bovine and murine phospholipase C zeta.Long noncoding RNAs in imprinting and X chromosome inactivation.The effects of melatonin on bovine uniparental embryos development in vitro and the hormone secretion of COCs Altered cell cycle gene expression and apoptosis in post-implantation dog parthenotes.Inducing human parthenogenetic embryonic stem cells into islet‑like clusters Lineage Selection and the Maintenance of Sex.First record of second-generation facultative parthenogenesis in a vertebrate species, the whitespotted bambooshark Chiloscyllium plagiosum.Downregulation of H19 improves the differentiation potential of mouse parthenogenetic embryonic stem cells.Defining contributions of paternally methylated imprinted genes at the Igf2-H19 and Dlk1-Gtl2 domains to mouse placentation by transcriptomic analysis.Concise review: parthenote stem cells for regenerative medicine: genetic, epigenetic, and developmental features Genetic causes of transitions from sexual reproduction to asexuality in plants and animals.The safety of human pluripotent stem cells in clinical treatment.Androgenesis: where males hijack eggs to clone themselves.Non-coding RNAs: emerging regulatory factors in the derivation and differentiation of mammalian parthenogenetic embryonic stem cells.Parentally imprinted genes regulate hematopoiesis-new evidence from the Dlk1-Gtl2 locus.Can modern biology interpret the mystery of the birth of Christ?Aggregation of pre-implantation embryos improves establishment of parthenogenetic stem cells and expression of imprinted genes.Nuclear reprogramming: the strategy used in normal development is also used in somatic cell nuclear transfer and parthenogenesis.

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P2860

Genomic imprinting is a barrier to parthenogenesis in mammals.

http://www.wikidata.org/entity/Q34566100

description

2006 nî lūn-bûn

@nan

2006 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2006 թվականի հունվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Genomic imprinting is a barrier to parthenogenesis in mammals.

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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type

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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prefLabel

Genomic imprinting is a barrier to parthenogenesis in mammals.

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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Cytogenetics and Genome Research

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Genomic imprinting is a barrier to parthenogenesis in mammals.

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Kono T

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P2860

The product of the H19 gene may function as an RNA

CTCF mediates methylation-sensitive enhancer-blocking activity at the H19/Igf2 locus

Completion of mouse embryogenesis requires both the maternal and paternal genomes.

Mice lacking paternally expressed Pref-1/Dlk1 display growth retardation and accelerated adiposity

Maternal primary imprinting is established at a specific time for each gene throughout oocyte growth.

The ubiquitin ligase Hyperplastic discs negatively regulates hedgehog and decapentaplegic expression by independent mechanisms.

Gene-specific timing and epigenetic memory in oocyte imprinting.

Epigenetic modifications during oocyte growth correlates with extended parthenogenetic development in the mouse.

In vitro and early in vivo development of sheep gynogenones and putative androgenones.

Gtl2lacZ, an insertional mutation on mouse chromosome 12 with parental origin-dependent phenotype.

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31-35

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scholarly article

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10.1159/000090812

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1-4

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113

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