The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration. - Wikidata - awgldk - TriplyDB

The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

http://www.wikidata.org/entity/Q34569329

about

Antagonism of LIN-17/Frizzled and LIN-18/Ryk in nematode vulva induction reveals evolutionary alterations in core developmental pathways Wnt modulators in the biotech pipeline ROR1, an embryonic protein with an emerging role in cancer biology The role of Ryk and Ror receptor tyrosine kinases in Wnt signal transduction Neurons refine the Caenorhabditis elegans body plan by directing axial patterning by Wnts Neuronal target identification requires AHA-1-mediated fine-tuning of Wnt signaling in C. elegans Ror2 receptor requires tyrosine kinase activity to mediate Wnt5A signaling Purified Wnt5a protein activates or inhibits beta-catenin-TCF signaling depending on receptor context Neuronal migration is regulated by endogenous RNAi and chromatin-binding factor ZFP-1/AF10 in Caenorhabditis elegans Wnt5a induces ROR1 to complex with HS1 to enhance migration of chronic lymphocytic leukemia cells.A Wnt-Frz/Ror-Dsh pathway regulates neurite outgrowth in Caenorhabditis elegans.Bioinformatic analysis of nematode migration-associated genes identifies novel vertebrate neural crest markers.Inverted selective plane illumination microscopy (iSPIM) enables coupled cell identity lineaging and neurodevelopmental imaging in Caenorhabditis elegans.Concentration-dependent effects of WNTLESS on WNT1/3A signaling.The role of the cysteine-rich domain of Frizzled in Wingless-Armadillo signaling.Dissecting the PCP pathway: one or more pathways?: Does a separate Wnt-Fz-Rho pathway drive morphogenesis?A systematic survey of expression and function of zebrafish frizzled genes Ror receptor tyrosine kinases: orphans no more.Autonomous and nonautonomous regulation of Wnt-mediated neuronal polarity by the C. elegans Ror kinase CAM-1 β-catenin-dependent Wnt signaling in C. elegans: teaching an old dog a new trick Complex network of Wnt signaling regulates neuronal migrations during Caenorhabditis elegans development.Wnt signaling in Pristionchus pacificus gonadal arm extension and the evolution of organ shape.The MIG-15 NIK kinase acts cell-autonomously in neuroblast polarization and migration in C. elegans The long and the short of Wnt signaling in C. elegans.Emerging mechanisms in morphogen-mediated axon guidance.Wnt signaling through the Ror receptor in the nervous system.Wnt-signaling and planar cell polarity genes regulate axon guidance along the anteroposterior axis in C. elegans.The Caenorhabditis elegans Q neuroblasts: A powerful system to study cell migration at single-cell resolution in vivo.ROR1 contributes to melanoma cell growth and migration by regulating N-cadherin expression via the PI3K/Akt pathway.Diverse roles for the ror-family receptor tyrosine kinases in neurons and glial cells during development and repair of the nervous system.Ror1-Ror2 complexes modulate synapse formation in hippocampal neurons.Wnt-Ror signaling to SIA and SIB neurons directs anterior axon guidance and nerve ring placement in C. elegans.Regulation of WNT Signaling at the Neuromuscular Junction by the Immunoglobulin Superfamily Protein RIG-3 in Caenorhabditis elegans Regulation of Axon Guidance by the Wnt Receptor Ror/CAM-1 in the PVT Guidepost Cell in Caenorhabditis elegans.Coordination of Heparan Sulfate Proteoglycans with Wnt Signaling To Control Cellular Migrations and Positioning in Caenorhabditis elegans.A pilgrim's progress: Seeking meaning in primordial germ cell migration.

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The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

http://www.wikidata.org/entity/Q34569329

description

2004 nî lūn-bûn

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2004 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2004 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

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The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

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The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

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type

label

The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

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The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

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The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

@nl

prefLabel

The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

@ast

The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

@en

The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

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GENETICS.104.031781

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The Caenorhabditis elegans Ror RTK CAM-1 inhibits EGL-20/Wnt signaling in cell migration.

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Changsung Kim

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Gian Garriga

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Wayne C Forrester

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P2860

Secreted frizzled-related protein-1 binds directly to Wingless and is a biphasic modulator of Wnt signaling

A novel family of cell surface receptors with tyrosine kinase-like domain

The genetics of Caenorhabditis elegans

Isolation and characterization of high-temperature-induced Dauer formation mutants in Caenorhabditis elegans

Loss of mRor1 enhances the heart and skeletal abnormalities in mRor2-deficient mice: redundant and pleiotropic functions of mRor1 and mRor2 receptor tyrosine kinases

Identification of a Frizzled-like cysteine rich domain in the extracellular region of developmental receptor tyrosine kinases

Insights into Wnt binding and signalling from the structures of two Frizzled cysteine-rich domains

Mutation of the gene encoding the ROR2 tyrosine kinase causes autosomal recessive Robinow syndrome

Ror2, encoding a receptor-like tyrosine kinase, is required for cartilage and growth plate development

Dominant mutations in ROR2, encoding an orphan receptor tyrosine kinase, cause brachydactyly type B

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