Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
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PET Study of Sphingosine-1-Phosphate Receptor 1 Expression in Response to Vascular Inflammation in a Rat Model of Carotid Injury.An update on the biology of sphingosine 1-phosphate receptors.The development and maintenance of paclitaxel-induced neuropathic pain require activation of the sphingosine 1-phosphate receptor subtype 1.Targeting the sphingosine-1-phosphate axis in cancer, inflammation and beyond.Activated platelets release sphingosine 1-phosphate and induce hypersensitivity to noxious heat stimuli in vivo.Molecular and cellular mechanisms that initiate pain and itch.Sphingosine lysolipids in the CNS: endogenous cannabinoid antagonists or a parallel pain modulatory system?Are GPCRs still a source of new targets?Sphingolipids in neutrophil function and inflammatory responses: Mechanisms and implications for intestinal immunity and inflammation in ulcerative colitis.Curcumin inhibits superoxide anion-induced pain-like behavior and leukocyte recruitment by increasing Nrf2 expression and reducing NF-κB activation.Sphingosine kinase 2-deficiency mediated changes in spinal pain processing.Micronized/ultramicronized palmitoylethanolamide displays superior oral efficacy compared to nonmicronized palmitoylethanolamide in a rat model of inflammatory pain.Targeting the S1P/S1PR1 axis mitigates cancer-induced bone pain and neuroinflammation.The TrkA receptor mediates experimental thermal hyperalgesia produced by nerve growth factor: Modulation by the p75 neurotrophin receptor.Anti-inflammatory effects of propranolol in the temporomandibular joint of female rats and its contribution to antinociceptive action.Repeated bouts of fast eccentric contraction produce sciatic nerve damage in rats.The signaling lipid sphingosine 1-phosphate regulates mechanical pain.Sphingosine-1-phosphate receptors and innate immunity.New extracellular factors in glioblastoma multiforme development: neurotensin, growth differentiation factor-15, sphingosine-1-phosphate and cytomegalovirus infection.The ruthenium nitric oxide donor, [Ru(HEDTA)NO], inhibits acute nociception in mice by modulating oxidative stress, cytokine production and activating the cGMP/PKG/ATP-sensitive potassium channel signaling pathway.
P2860
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P2860
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
description
2013 nî lūn-bûn
@nan
2013 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@ast
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@en
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@nl
type
label
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@ast
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@en
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@nl
prefLabel
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@ast
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@en
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@nl
P2093
P2860
P1433
P1476
Sphingosine 1-phosphate mediates hyperalgesia via a neutrophil-dependent mechanism.
@en
P2093
Amanda Finley
Daniela Salvemini
Emanuela Esposito
Roger Sabbadini
Zhoumou Chen
P2860
P304
P356
10.1371/JOURNAL.PONE.0055255
P407
P577
2013-01-25T00:00:00Z