Infusion of tumor necrosis factor/cachectin promotes muscle catabolism in the rat. A synergistic effect with interleukin 1 - Wikidata - awgldk - TriplyDB

Infusion of tumor necrosis factor/cachectin promotes muscle catabolism in the rat. A synergistic effect with interleukin 1

Infusion of tumor necrosis factor/cachectin promotes muscle catabolism in the rat. A synergistic effect with interleukin 1

http://www.wikidata.org/entity/Q34572671

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Muscle dysfunction in chronic obstructive pulmonary disease: update on causes and biological findings The mechanisms of cachexia underlying muscle dysfunction in COPD Understanding cachexia as a cancer metabolism syndrome Inflammatory Mechanisms Associated with Skeletal Muscle Sequelae after Stroke: Role of Physical Exercise Nuclear export of phosphorylated C/EBPbeta mediates the inhibition of albumin expression by TNF-alpha TNF-Alpha in the Locomotor System beyond Joints: High Degree of Involvement in Myositis in a Rabbit Model Recombinant human interleukin-1 induces meningitis and blood-brain barrier injury in the rat. Characterization and comparison with tumor necrosis factor Tumor necrosis factor-alpha mediates changes in tissue protein turnover in a rat cancer cachexia model.Mechanisms accelerating muscle atrophy in catabolic diseases Role of IGF-I and the TNFα/NF-κB pathway in the induction of muscle atrogenes by acute inflammation.Heart failure as a metabolic problem.Induction of muscle protein degradation by a tumour factor Cell walls of Saccharomyces cerevisiae differentially modulated innate immunity and glucose metabolism during late systemic inflammation.The muscle fiber type-fiber size paradox: hypertrophy or oxidative metabolism?Administration of endotoxin, tumor necrosis factor, or interleukin 1 to rats activates skeletal muscle branched-chain alpha-keto acid dehydrogenase.Nutrition and tumor promotion: in vivo methods for measurement of cellular proliferation and protein metabolism.Cytokine-cytokine interactions in the context of cytokine networking.Body Mass Index, High-Sensitivity C-Reactive Protein and Mortality in Chinese with Coronary Artery Disease Increased protein degradation and decreased protein synthesis in skeletal muscle during cancer cachexia Mechanism of muscle protein degradation in cancer cachexia Rhabdomyolysis associated with antimicrobial drug-resistant Mycoplasma pneumoniae.Bioactivity of skeletal muscle proteolysis-inducing factors in the plasma proteins from cancer patients with weight loss.Cytokines in idiopathic inflammatory myopathies.Emerging Roles of Branched-Chain Amino Acid Supplementation in Human Diseases.The role of albumin in human physiology and pathophysiology, Part III: Albumin and disease states.Muscle wasting in a rat model of long-lasting sepsis results from the activation of lysosomal, Ca2+ -activated, and ubiquitin-proteasome proteolytic pathways.Increased mRNA levels for components of the lysosomal, Ca2+-activated, and ATP-ubiquitin-dependent proteolytic pathways in skeletal muscle from head trauma patients.Tumor necrosis factor-alpha as a potential therapeutic target in idiopathic inflammatory myopathies.Metabolic responses to cachectin/TNF. A brief review.Respiratory diseases and muscle dysfunction.Hormonal regulation of protein metabolism.Targeted immunotherapy trials for idiopathic inflammatory myopathies.Characterization and regulation of mechanical loading-induced compensatory muscle hypertrophy.Muscle wasting and dedifferentiation induced by oxidative stress in a murine model of cachexia is prevented by inhibitors of nitric oxide synthesis and antioxidants.Acute rhabdomyolysis and inflammation.Sesamol down-regulates the lipopolysaccharide-induced inflammatory response by inhibiting nuclear factor-kappa B activation.The omega-3 fatty acid, eicosapentaenoic acid (EPA), prevents the damaging effects of tumour necrosis factor (TNF)-alpha during murine skeletal muscle cell differentiation.Stimulation of monokine production by lipoteichoic acids.Effects of Escherichia coli hemolysin on human monocytes. Cytocidal action and stimulation of interleukin 1 release.Metabolic effects of cachectin/tumor necrosis factor are modified by site of production. Cachectin/tumor necrosis factor-secreting tumor in skeletal muscle induces chronic cachexia, while implantation in brain induces predominantly acute anorexia

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P2860

Infusion of tumor necrosis factor/cachectin promotes muscle catabolism in the rat. A synergistic effect with interleukin 1

http://www.wikidata.org/entity/Q34572671

description

1989 nî lūn-bûn

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1989 թուականի Մայիսին հրատարակուած գիտական յօդուած

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1989 թվականի մայիսին հրատարակված գիտական հոդված

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1989年の論文

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1989年論文

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1989年論文

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1989年論文

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1989年論文

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1989年論文

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1989年论文

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name

Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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type

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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prefLabel

Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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Journal of Clinical Investigation

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Infusion of tumor necrosis fac ...... stic effect with interleukin 1

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P2093

B R Bistrian

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C A Dinarello

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E A Flores

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G L Blackburn

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J J Pomposelli

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P2860

Increased proteolysis. An effect of increases in plasma cortisol within the physiologic range

Leucine degradation in cell-free extracts of skeletal muscle

Interleukin 1 induces a shock-like state in rabbits. Synergism with tumor necrosis factor and the effect of cyclooxygenase inhibition

Shock and tissue injury induced by recombinant human cachectin.

Activation of protein breakdown and prostaglandin E2 production in rat skeletal muscle in fever is signaled by a macrophage product distinct from interleukin 1 or other known monokines.

Tumor necrosis factor can induce fever in rats without activating protein breakdown in muscle or lipolysis in adipose tissue

Tumor necrosis factor (cachectin) is an endogenous pyrogen and induces production of interleukin 1

Cachectin/tumor necrosis factor induces cachexia, anemia, and inflammation.

Multiple biological activities of human recombinant interleukin 1.

Effect of ibuprofen on fever and metabolic changes induced by continuous infusion of leukocytic pyrogen (interleukin 1) or endotoxin

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1614-1622

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10.1172/JCI114059

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5

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83

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2785120

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303868

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