Endothelial cells promote the colorectal cancer stem cell phenotype through a soluble form of Jagged-1. - Wikidata - awgldk - TriplyDB

Endothelial cells promote the colorectal cancer stem cell phenotype through a soluble form of Jagged-1.

Endothelial cells promote the colorectal cancer stem cell phenotype through a soluble form of Jagged-1.

http://www.wikidata.org/entity/Q34572680

about

The notch ligand JAGGED1 as a target for anti-tumor therapy Cancer Stem Cells in Human Gastrointestinal Cancer Cytokine-Induced Modulation of Colorectal Cancer An Overview of Alternating Electric Fields Therapy (NovoTTF Therapy) for the Treatment of Malignant Glioma Epithelial-mesenchymal transition in gastric cancer Cancer stem cells and the tumor microenvironment: interplay in tumor heterogeneity Implications of the Hybrid Epithelial/Mesenchymal Phenotype in Metastasis Genetic instability in the tumor microenvironment: a new look at an old neighbor Cancer heterogeneity--a multifaceted view Microenvironmental control of stem cell fate in intestinal homeostasis and disease The function of miRNA in hepatic cancer stem cell From fly wings to targeted cancer therapies: a centennial for notch signaling Molecular pathways: not a simple tube--the many functions of blood vessels A Disintegrin and Metalloproteinase Domain 17 Regulates Colorectal Cancer Stem Cells and Chemosensitivity Via Notch1 Signaling.Crosstalk between cancer cells and blood endothelial and lymphatic endothelial cells in tumour and organ microenvironment VEGFR-1 Pseudogene Expression and Regulatory Function in Human Colorectal Cancer Cells Angiocrine factors deployed by tumor vascular niche induce B cell lymphoma invasiveness and chemoresistance Silencing of Jagged1 inhibits cell growth and invasion in colorectal cancer.Profilin-1 phosphorylation directs angiocrine expression and glioblastoma progression through HIF-1α accumulation.CoCl2, a mimic of hypoxia, induces formation of polyploid giant cells with stem characteristics in colon cancer.Anti-angiogenic therapy for cancer: current progress, unresolved questions and future directions.MicroRNA-205 signaling regulates mammary stem cell fate and tumorigenesis.Molecular Checkpoint Decisions Made by Subverted Vascular Niche Transform Indolent Tumor Cells into Chemoresistant Cancer Stem Cells Microparticles mediated cross-talk between tumoral and endothelial cells promote the constitution of a pro-metastatic vascular niche through Arf6 up regulation.Hypoxia as a biomarker for radioresistant cancer stem cells.Concomitant Notch activation and p53 deletion trigger epithelial-to-mesenchymal transition and metastasis in mouse gut.TP53 loss creates therapeutic vulnerability in colorectal cancer.Endothelial cells provide a notch-dependent pro-tumoral niche for enhancing breast cancer survival, stemness and pro-metastatic properties.Endothelial cells promote stem-like phenotype of glioma cells through activating the Hedgehog pathway.Cancer stem cells maintain a hierarchy of differentiation by creating their niche T cells bearing a chimeric antigen receptor against prostate-specific membrane antigen mediate vascular disruption and result in tumor regression.Dynamics of Notch pathway expression during mouse testis post-natal development and along the spermatogenic cycle Oncogenic Ras stimulates Eiger/TNF exocytosis to promote growth.Notch3/Jagged1 circuitry reinforces notch signaling and sustains T-ALL Jagged-Delta asymmetry in Notch signaling can give rise to a Sender/Receiver hybrid phenotype.Maintenance of stemness in oxaliplatin-resistant hepatocellular carcinoma is associated with increased autocrine of IGF1.Cleaved NOTCH1 Expression Pattern in Head and Neck Squamous Cell Carcinoma Is Associated with NOTCH1 Mutation, HPV Status, and High-Risk Features.Therapeutic silencing of KRAS using systemically delivered siRNAs.Optimum 3D Matrix Stiffness for Maintenance of Cancer Stem Cells Is Dependent on Tissue Origin of Cancer Cells.CCN1 induces hepatic ductular reaction through integrin αvβ₅-mediated activation of NF-κB.

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P2860

Endothelial cells promote the colorectal cancer stem cell phenotype through a soluble form of Jagged-1.

http://www.wikidata.org/entity/Q34572680

description

2013 nî lūn-bûn

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2013 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2013 թվականի հունվարին հրատարակված գիտական հոդված

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2013年の論文

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J.CCR.2012.12.021

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Endothelial cells promote the ...... ugh a soluble form of Jagged-1

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Dipen M Maru

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Eric Sceusi

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Fan Fan

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Federico Tozzi

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Isamu Tachibana

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Janusz Rak

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Jia Lu

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Lee M Ellis

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Ling Xia

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Seth Bellister

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P2860

Soluble Jagged 1 represses the function of its transmembrane form to induce the formation of the Src-dependent chord-like phenotype

Jagged-1 mediated activation of notch signaling induces complete maturation of human keratinocytes through NF-kappaB and PPARgamma

Aldehyde dehydrogenase 1 is a marker for normal and malignant human colonic stem cells (SC) and tracks SC overpopulation during colon tumorigenesis

Delta-like ligand 4 (Dll4) is induced by VEGF as a negative regulator of angiogenic sprouting

Tumor angiogenesis: therapeutic implications

Identification and expansion of human colon-cancer-initiating cells

A human colon cancer cell capable of initiating tumour growth in immunodeficient mice

The human homolog of rat Jagged1 expressed by marrow stroma inhibits differentiation of 32D cells through interaction with Notch1

Cancer stem cells--perspectives on current status and future directions: AACR Workshop on cancer stem cells

Signalling downstream of activated mammalian Notch

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2

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Patrick A Zweidler-McKay

Sendurai A. Mani

Rajat Bhattacharya

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2013-01-31T00:00:00Z

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235395895

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23375636

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colorectal cancer

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3574187

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