Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy - Wikidata - awgldk - TriplyDB

Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

http://www.wikidata.org/entity/Q34575119

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Inhibiting Immune Checkpoints for the Treatment of Bladder Cancer The programmed death-1 immune-suppressive pathway: barrier to antitumor immunity New antibody approaches to lymphoma therapy High-Resolution PET Imaging with Therapeutic Antibody-based PD-1/PD-L1 Checkpoint Tracers B7-H1 blockade increases survival of dysfunctional CD8(+) T cells and confers protection against Leishmania donovani infections Specific CD8(+) T cell response immunotherapy for hepatocellular carcinoma and viral hepatitis PD-1 and PD-L1 antibodies for melanoma Blockade of programmed death-1 in young (New Zealand Black x New Zealand White)F1 mice promotes the suppressive capacity of CD4+ regulatory T cells protecting from lupus-like disease Phase I study of single-agent anti-programmed death-1 (MDX-1106) in refractory solid tumors: safety, clinical activity, pharmacodynamics, and immunologic correlates Regulation of T-cell Tolerance by Lymphatic Endothelial Cells.Low-Intensity Focused Ultrasound Induces Reversal of Tumor-Induced T Cell Tolerance and Prevents Immune Escape.Lymphatic endothelial cells induce tolerance via PD-L1 and lack of costimulation leading to high-level PD-1 expression on CD8 T cells.Lymphatic endothelial cells - key players in regulation of tolerance and immunity PD-1 deficiency reveals various tissue-specific autoimmunity by H-2b and dose-dependent requirement of H-2g7 for diabetes in NOD mice.PD-1/PD-L blockade prevents anergy induction and enhances the anti-tumor activities of glycolipid-activated invariant NKT cells CpG-enhanced CD8+ T-cell responses to peptide immunization are severely inhibited by B cells New Cancer Immunotherapy Agents in Development: a report from an associated program of the 31stAnnual Meeting of the Society for Immunotherapy of Cancer, 2016 PD-L1 has distinct functions in hematopoietic and nonhematopoietic cells in regulating T cell responses during chronic infection in mice.B7-H1/CD80 interaction is required for the induction and maintenance of peripheral T-cell tolerance PD-1, PD-L1 (B7-H1) and Tumor-Site Immune Modulation Therapy: The Historical Perspective.Interferon γ limits the effectiveness of melanoma peptide vaccines.Soluble PD-1 rescues the proliferative response of simian immunodeficiency virus-specific CD4 and CD8 T cells during chronic infection.PD-1 on dendritic cells impedes innate immunity against bacterial infection.Differences in allergen-induced T cell activation between allergic asthma and rhinitis: Role of CD28, ICOS and CTLA-4 LAG-3 regulates CD8+ T cell accumulation and effector function in murine self- and tumor-tolerance systems.Interaction of human PD-L1 and B7-1.B7H1/CD80 interaction augments PD-1-dependent T cell apoptosis and ameliorates graft-versus-host disease Blockade of PD-1/PD-L1 promotes adoptive T-cell immunotherapy in a tolerogenic environment.B7-H1 enhances proliferation ability of gastric cancer stem-like cells as a receptor.Cell surface signaling molecules in the control of immune responses: a tide model.PD-L1 over-expression and survival in patients with non-small cell lung cancer: a meta-analysis.Clinicopathological and prognostic significance of programmed cell death ligand1 (PD-L1) expression in patients with non-small cell lung cancer: a meta-analysis PD-1 as an immune modulatory receptor.Program death-1 regulates peripheral T cell tolerance via an anergy-independent mechanism.Elevated tumor-associated antigen expression suppresses variant peptide vaccine responses.Oral Squamous Carcinoma Cells Express B7-H1 and B7-DC Receptors in Vivo.Anti-PD-1/PD-L1 therapy of human cancer: past, present, and future.Monoclonal antibodies specific to human Δ42PD1: A novel immunoregulator potentially involved in HIV-1 and tumor pathogenesis.Mannose-Capped Lipoarabinomannan from Mycobacterium tuberculosis Induces CD4+ T Cell Anergy via GRAIL PD-1 Blunts the Function of Ovarian Tumor-Infiltrating Dendritic Cells by Inactivating NF-κB

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

http://www.wikidata.org/entity/Q34575119

description

2007 nî lūn-bûn

@nan

2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի փետրվարին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Interaction between B7-H1 and PD-1 determines initiation and reversal of T-cell anergy

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Andrew Flies

http://www.w3.org/2001/XMLSchema#string

Drew M Pardoll

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Fumihiko Tsushima

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Haiying Xu

http://www.w3.org/2001/XMLSchema#string

Koji Tamada

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Lieping Chen

http://www.w3.org/2001/XMLSchema#string

Sarah Flies

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Sheng Yao

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Tahiro Shin

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B7-H1, a third member of the B7 family, co-stimulates T-cell proliferation and interleukin-10 secretion

B7-DC, a new dendritic cell molecule with potent costimulatory properties for T cells

Involvement of PD-L1 on tumor cells in the escape from host immune system and tumor immunotherapy by PD-L1 blockade.

Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

Tumor-associated B7-H1 promotes T-cell apoptosis: a potential mechanism of immune evasion

Development of lupus-like autoimmune diseases by disruption of the PD-1 gene encoding an ITIM motif-carrying immunoreceptor

Autoimmune dilated cardiomyopathy in PD-1 receptor-deficient mice

PD-L2 is a second ligand for PD-1 and inhibits T cell activation

Expression of programmed death 1 ligands by murine T cells and APC

The B7 family revisited

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180-185

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10.1182/BLOOD-2006-11-060087

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