Asymmetric N-cadherin expression results in synapse dysfunction, synapse elimination, and axon retraction in cultured mouse neurons.

Asymmetric N-cadherin expression results in synapse dysfunction, synapse elimination, and axon retraction in cultured mouse neurons.

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http://www.wikidata.org/entity/Q34575340
δ-Catenin Regulates Spine Architecture via Cadherin and PDZ-dependent Interactions.Cadherin-based transsynaptic networks in establishing and modifying neural connectivity.Cadherins and catenins in dendrite and synapse morphogenesisLearning From Animal Models of Obsessive-Compulsive DisorderN-cadherin regulates molecular organization of excitatory and inhibitory synaptic circuits in adult hippocampus in vivoThe Caenorhabditis elegans microtubule minus-end binding homolog PTRN-1 stabilizes synapses and neurites.N-cadherin participated in invasion and metastasis of human esophageal squamous cell carcinoma via taking part in the formation of vasculogenic mimicry.Candidate genes and functional noncoding variants identified in a canine model of obsessive-compulsive disorder.Long-term depression-associated signaling is required for an in vitro model of NMDA receptor-dependent synapse pruning.Properties of Retinal Precursor Cells Grown on Vertically Aligned Multiwalled Carbon Nanotubes Generated for the Modification of Retinal Implant-Embedded Microelectrode Arrays.The gene encoding the mouse contactin-1 axonal glycoprotein is regulated by the collier/Olf1/EBF family early B-Cell factor 2 transcription factor.Alzheimer's disease-related amyloid-β induces synaptotoxicity in human iPS cell-derived neurons.Release activity-dependent control of vesicle endocytosis by the synaptic adhesion molecule N-cadherin.Expression profile of N-cadherin and protocadherin-19 in postnatal mouse limbic structures.Postsynaptic Y654 dephosphorylation of β-catenin modulates presynaptic vesicle turnover through increased n-cadherin-mediated transsynaptic signaling.
P2860

Asymmetric N-cadherin expression results in synapse dysfunction, synapse elimination, and axon retraction in cultured mouse neurons.

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http://www.wikidata.org/entity/Q34575340
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2013 nî lūn-bûn
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2013 թուականի Յունուարին հրատարակուած գիտական յօդուած
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2013 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@ast
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@en
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@nl
type
label
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@ast
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@en
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@nl
prefLabel
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@ast
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@en
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P932
P356
P1433
P1476
Asymmetric N-cadherin expressi ...... ion in cultured mouse neurons.
@en
P2093
P2860
P304
P31
P356
10.1371/JOURNAL.PONE.0054105
P407
P433
P478
P577
P5875
P698
P932