Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection.
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Paracoccidioides-host Interaction: An Overview on Recent Advances in the ParacoccidioidomycosisOptimal structural design of mannosylated nanocarriers for macrophage targeting.TNF-α and CD8+ T cells mediate the beneficial effects of nitric oxide synthase-2 deficiency in pulmonary paracoccidioidomycosis.In pulmonary paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology.Indoleamine 2,3-dioxygenase controls fungal loads and immunity in Paracoccidioidomicosis but is more important to susceptible than resistant hosts.Innate Defense against Fungal PathogensLoss- and Gain-of-Function Approaches Indicate a Dual Role Exerted by Regulatory T Cells in Pulmonary Paracoccidioidomycosis.Deletion of the α-(1,3)-glucan synthase genes induces a restructuring of the conidial cell wall responsible for the avirulence of Aspergillus fumigatus.New approaches to transcutaneous immunotherapy: targeting dendritic cells with novel allergen conjugates.NOD-Like Receptor P3 Inflammasome Controls Protective Th1/Th17 Immunity against Pulmonary Paracoccidioidomycosis.The Effects of Paracoccidioides brasiliensis Infection on GM-CSF- and M-CSF-Induced Mouse Bone Marrow-Derived Macrophage from Resistant and Susceptible Mice Strains.Extracellular vesicles from Paracoccidioides brasiliensis induced M1 polarization in vitro.Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosisParacoccin Induces M1 Polarization of Macrophages via Interaction with TLR4.Lipoxin Inhibits Fungal Uptake by Macrophages and Reduces the Severity of Acute Pulmonary Infection Caused by Paracoccidioides brasiliensis.Promising landscape for regulating macrophage polarization: epigenetic viewpointDisease Tolerance Mediated by Phosphorylated Indoleamine-2,3 Dioxygenase Confers Resistance to a Primary Fungal Pathogen.Paracoccidioidomycosis: Current Perspectives from Brazil.CD40 ligand deficiency causes functional defects of peripheral neutrophils that are improved by exogenous IFN-γ.The Interaction of Human Pathogenic Fungi With C-Type Lectin Receptors.Phospholipase Cγ2 is critical for Ca2+ flux and cytokine production in anti-fungal innate immunity of human corneal epithelial cells.
P2860
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P2860
Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection.
description
2013 nî lūn-bûn
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2013 թուականի Յունուարին հրատարակուած գիտական յօդուած
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2013 թվականի հունվարին հրատարակված գիտական հոդված
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@ast
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@en
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@nl
type
label
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@ast
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@en
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@nl
prefLabel
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@ast
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@en
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@nl
P2093
P2860
P1433
P1476
Mannosyl-recognizing receptors ...... sistant to a fungal infection.
@en
P2093
Eliseu Frank de Araújo
Tania Alves da Costa
Vera L G Calich
P2860
P304
P356
10.1371/JOURNAL.PONE.0054845
P407
P577
2013-01-30T00:00:00Z