Mutations in FOXC2 are strongly associated with primary valve failure in veins of the lower limb. - Wikidata - awgldk - TriplyDB

Mutations in FOXC2 are strongly associated with primary valve failure in veins of the lower limb.

Mutations in FOXC2 are strongly associated with primary valve failure in veins of the lower limb.

http://www.wikidata.org/entity/Q34577939

about

FOXC2 controls formation and maturation of lymphatic collecting vessels through cooperation with NFATc1 Foxc2 transcription factor: a newly described regulator of angiogenesis Development of the mammalian lymphatic vasculature EPHB4 kinase-inactivating mutations cause autosomal dominant lymphatic-related hydrops fetalis.A case of lymphedema-distichiasis syndrome carrying a new de novo frameshift FOXC2 mutation The Foxc2 transcription factor regulates angiogenesis via induction of integrin beta3 expression New horizons for imaging lymphatic function.Extracorporeal shock waves as curative therapy for varicose veins?Contractile physiology of lymphatics Method for the quantitative measurement of collecting lymphatic vessel contraction in mice Determining the combined effect of the lymphatic valve leaflets and sinus on resistance to forward flow Lymphatic transport in patients with chronic venous insufficiency and venous leg ulcers following sequential pneumatic compression.Phosphorylation regulates FOXC2-mediated transcription in lymphatic endothelial cells Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives Primary and secondary lymphatic valve development: molecular, functional and mechanical insights.Lymphatic and angiogenic candidate genes predict the development of secondary lymphedema following breast cancer surgery.Transcutaneous laser treatment of leg veins.Venous endothelial injury in central nervous system diseases Forkhead box C2 promoter variant c.-512C>T is associated with increased susceptibility to chronic venous diseases Lymphatic vascular morphogenesis in development, physiology, and disease Absence of venous valves in mice lacking Connexin37 Segregated Foxc2, NFATc1 and Connexin expression at normal developing venous valves, and Connexin-specific differences in the valve phenotypes of Cx37, Cx43, and Cx47 knockout mice.Pathogenesis of varicose veins and implications for clinical management.Foxc1 and Foxc2 deletion causes abnormal lymphangiogenesis and correlates with ERK hyperactivation Forkhead transcription factors and cardiovascular biology Novel FOXC2 Mutation in Hereditary Distichiasis Impairs DNA-Binding Activity and Transcriptional Activation.Diagnosis and therapy in children with lymphoedema.New developments in clinical aspects of lymphatic disease.Lymphangiogenesis, myeloid cells and inflammation.Perspectives on lymphangiogenesis and angiogenesis in cancer.Massively parallel sequencing and identification of genes for primary lymphoedema: a perfect fit.The transcriptional control of lymphatic vascular development.The lymphatic vasculature in disease.Evidence for a genetic role in varicose veins and chronic venous insufficiency.Flow control in our vessels: vascular valves make sure there is no way back.The classification and diagnostic algorithm for primary lymphatic dysplasia: an update from 2010 to include molecular findings.Cellular and molecular basis of Venous insufficiency.Lymphatic pumping: mechanics, mechanisms and malfunction.Building the drains: the lymphatic vasculature in health and disease.New diagnostic modalities in the evaluation of lymphedema.

P2860

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P2860

Mutations in FOXC2 are strongly associated with primary valve failure in veins of the lower limb.

http://www.wikidata.org/entity/Q34577939

description

2007 nî lūn-bûn

@nan

2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

@zh-hk

2007年論文

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2007年論文

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2007年论文

@wuu

name

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@ast

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@en

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@nl

type

label

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@ast

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@en

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@nl

prefLabel

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@ast

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@en

Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@nl

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CIRCULATIONAHA.106.675348

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Mutations in FOXC2 are strongl ...... re in veins of the lower limb.

@en

P2093

Alberto Smith

http://www.w3.org/2001/XMLSchema#string

Anthony W B Stanton

http://www.w3.org/2001/XMLSchema#string

Glen Brice

http://www.w3.org/2001/XMLSchema#string

J Rodney Levick

http://www.w3.org/2001/XMLSchema#string

Jane French

http://www.w3.org/2001/XMLSchema#string

Kevin G Burnand

http://www.w3.org/2001/XMLSchema#string

Lymphoedema Research Consortium

http://www.w3.org/2001/XMLSchema#string

Peter S Mortimer

http://www.w3.org/2001/XMLSchema#string

Russell H Mellor

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Steve Jeffery

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1912-1920

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scholarly article

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10.1161/CIRCULATIONAHA.106.675348

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14

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115

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2007-03-19T00:00:00Z

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17372167

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