Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells - Wikidata - awgldk - TriplyDB

Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

http://www.wikidata.org/entity/Q34578170

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Clinical utility of nivolumab in the treatment of advanced melanoma Exploiting the Immunomodulatory Properties of Chemotherapeutic Drugs to Improve the Success of Cancer Immunotherapy Impaired T-Cell Function in B-Cell Lymphoma: A Direct Consequence of Events at the Immunological Synapse?B7-H1 blockade increases survival of dysfunctional CD8(+) T cells and confers protection against Leishmania donovani infections Antigen-specific immunotherapy of cervical and ovarian cancer High-programmed death-1 levels on hepatitis C virus-specific T cells during acute infection are associated with viral persistence and require preservation of cognate antigen during chronic infection Combination Approaches with Immune-Checkpoint Blockade in Cancer Therapy Specific CD8(+) T cell response immunotherapy for hepatocellular carcinoma and viral hepatitis Immune checkpoint blockade: a common denominator approach to cancer therapy High avidity cytotoxic T lymphocytes can be selected into the memory pool but they are exquisitely sensitive to functional impairment Phase I study of single-agent anti-programmed death-1 (MDX-1106) in refractory solid tumors: safety, clinical activity, pharmacodynamics, and immunologic correlates Regulation of T-cell Tolerance by Lymphatic Endothelial Cells.Control of pathogenic effector T-cell activities in situ by PD-L1 expression on respiratory inflammatory dendritic cells during respiratory syncytial virus infection.Lymphatic endothelial cells induce tolerance via PD-L1 and lack of costimulation leading to high-level PD-1 expression on CD8 T cells.Lymphatic endothelial cells - key players in regulation of tolerance and immunity Blockade of PD-1/B7-H1 interaction restores effector CD8+ T cell responses in a hepatitis C virus core murine model A2A receptor signaling promotes peripheral tolerance by inducing T-cell anergy and the generation of adaptive regulatory T cells.DNA vaccines for cervical cancer Metastatic melanoma: New paradigms of treatment and new toxicities Self-antigen prevents CD8 T cell effector differentiation by CD134 and CD137 dual costimulation.Breathing new life into immunotherapy: review of melanoma, lung and kidney cancer Perspectives for preventive and therapeutic HPV vaccines.Immunotherapy for cervical cancer: Research status and clinical potential B7-H1/CD80 interaction is required for the induction and maintenance of peripheral T-cell tolerance LAG-3, TGF-β, and cell-intrinsic PD-1 inhibitory pathways contribute to CD8 but not CD4 T-cell tolerance induced by allogeneic BMT with anti-CD40L.Perspective for prophylaxis and treatment of cervical cancer: an immunological approach.Immune inhibitory molecules LAG-3 and PD-1 synergistically regulate T-cell function to promote tumoral immune escape PD-1, PD-L1 (B7-H1) and Tumor-Site Immune Modulation Therapy: The Historical Perspective.PD-1 on dendritic cells impedes innate immunity against bacterial infection.LAG-3 regulates CD8+ T cell accumulation and effector function in murine self- and tumor-tolerance systems.Tc17 CD8 T cells: functional plasticity and subset diversity.Functionally distinct LAG-3 and PD-1 subsets on activated and chronically stimulated CD8 T cells Cyclophosphamide augments antitumor immunity: studies in an autochthonous prostate cancer model.Blockade of PD-1/PD-L1 promotes adoptive T-cell immunotherapy in a tolerogenic environment.DNA vaccines for cervical cancer: from bench to bedside.B7-h1 expressed by activated CD8 T cells is essential for their survival.Anti-tumor effects of endogenous prostate cancer-specific CD8 T cells in a murine TCR transgenic model Elevated tumor-associated antigen expression suppresses variant peptide vaccine responses.Anti-PD-1/PD-L1 therapy of human cancer: past, present, and future.Subset- and Antigen-Specific Effects of Treg on CD8+ T Cell Responses in Chronic HIV Infection

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

http://www.wikidata.org/entity/Q34578170

description

2007 nî lūn-bûn

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2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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BLOOD-2006-12-062422

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Role of PD-1 and its ligand, B7-H1, in early fate decisions of CD8 T cells

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Charles G Drake

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Charles H Maris

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Derese Getnet

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Dirk G Brockstedt

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Drew M Pardoll

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Edward L Hipkiss

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Joseph F Grosso

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Katharine A Whartenby

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Lijie Zhen

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Monica V Goldberg

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P2860

B7-DC, a new dendritic cell molecule with potent costimulatory properties for T cells

Blockade of programmed death-1 ligands on dendritic cells enhances T cell activation and cytokine production

Class I-restricted cross-presentation of exogenous self-antigens leads to deletion of autoreactive CD8(+) T cells

Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

Development of lupus-like autoimmune diseases by disruption of the PD-1 gene encoding an ITIM motif-carrying immunoreceptor

Autoimmune dilated cardiomyopathy in PD-1 receptor-deficient mice

The B7 family revisited

Lymphoproliferative disorders with early lethality in mice deficient in Ctla-4

Restoring function in exhausted CD8 T cells during chronic viral infection

Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4

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186-192

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scholarly article

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10.1182/BLOOD-2006-12-062422

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1

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Andrew S. Flies

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6416719

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1896112

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