MyoD expression restores defective myogenic differentiation of human mesoangioblasts from inclusion-body myositis muscle. - Wikidata - awgldk - TriplyDB

MyoD expression restores defective myogenic differentiation of human mesoangioblasts from inclusion-body myositis muscle.

MyoD expression restores defective myogenic differentiation of human mesoangioblasts from inclusion-body myositis muscle.

http://www.wikidata.org/entity/Q34578181

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How citation distortions create unfounded authority: analysis of a citation network Emerging therapeutic options for sporadic inclusion body myositis SHARP1/DEC2 inhibits adipogenic differentiation by regulating the activity of C/EBP Endothelial and myogenic differentiation of hematopoietic progenitor cells in inflammatory myopathies.Biocompatible tissue scaffold compliance promotes salivary gland morphogenesis and differentiation Enhancement of myogenic and muscle repair capacities of human adipose-derived stem cells with forced expression of MyoD.MYOD mediates skeletal myogenic differentiation of human amniotic fluid stem cells and regeneration of muscle injury.Immune and myodegenerative pathomechanisms in inclusion body myositis Satellite cells and the muscle stem cell niche Contribution of human muscle-derived cells to skeletal muscle regeneration in dystrophic host mice.Sphingosine 1-phosphate induces differentiation of mesoangioblasts towards smooth muscle. A role for GATA6 Vessel-associated stem cells from skeletal muscle: From biology to future uses in cell therapy.Transcription factor rational design improves directed differentiation of human mesenchymal stem cells into skeletal myocytes.Systemic delivery of allogenic muscle stem cells induces long-term muscle repair and clinical efficacy in duchenne muscular dystrophy dogs A Genome-Wide mRNA Screen and Functional Analysis Reveal FOXO3 as a Candidate Gene for Chicken Growth G9a mediates Sharp-1-dependent inhibition of skeletal muscle differentiation.bHLH-Orange Transcription Factors in Development and Cancer.Muscle Tissue Engineering Using Gingival Mesenchymal Stem Cells Encapsulated in Alginate Hydrogels Containing Multiple Growth Factors.Inclusion-body myositis: muscle-fiber molecular pathology and possible pathogenic significance of its similarity to Alzheimer's and Parkinson's disease brains.The origin, molecular regulation and therapeutic potential of myogenic stem cell populations.Mesoangioblasts of inclusion-body myositis: a twofold tool to study pathogenic mechanisms and enhance defective muscle regeneration Inclusion body myositis: from immunopathology and degenerative mechanisms to treatment perspectives.The role of satellite cells in muscle hypertrophy.Emerging gene editing strategies for Duchenne muscular dystrophy targeting stem cells.Endothelial sphingosine kinase/SPNS2 axis is critical for vessel-like formation by human mesoangioblasts.Codependent activators direct myoblast-specific MyoD transcription.Sources for skeletal muscle repair: from satellite cells to reprogramming Invasive fibroblasts: fundamental difference between sporadic inclusion body myositis and polymyositis.Reversible immortalisation enables genetic correction of human muscle progenitors and engineering of next-generation human artificial chromosomes for Duchenne muscular dystrophy.Labeling protocols for in vivo tracking of human skeletal muscle cells (HSkMCs) by magnetic resonance and bioluminescence imaging.Ecto-mesenchymal stem cells from facial process: potential for muscle regeneration.Localization and Expression of Nuclear Factor of Activated T-Cells 5 in Myoblasts Exposed to Pro-inflammatory Cytokines or Hyperosmolar Stress and in Biopsies from Myositis Patients.

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MyoD expression restores defective myogenic differentiation of human mesoangioblasts from inclusion-body myositis muscle.

http://www.wikidata.org/entity/Q34578181

description

2006 nî lūn-bûn

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2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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MyoD expression restores defec ...... nclusion-body myositis muscle.

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MyoD expression restores defec ...... nclusion-body myositis muscle.

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MyoD expression restores defec ...... nclusion-body myositis muscle.

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MyoD expression restores defec ...... nclusion-body myositis muscle.

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MyoD expression restores defec ...... nclusion-body myositis muscle.

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MyoD expression restores defec ...... nclusion-body myositis muscle.

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PNAS.0603386103

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Proceedings of the National Academy of Sciences of the United States of America

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MyoD expression restores defec ...... inclusion-body myositis muscle

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Aldobrando Broccolini

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Carla Gliubizzi

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Enrica Roncaglia

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Giulio Cossu

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Luciana De Angelis

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Manuela Papacci

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Pietro Attilio Tonali

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Stefano Ferrari

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Teresa Gidaro

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P2860

DEC1 negatively regulates the expression of DEC2 through binding to the E-box in the proximal promoter

Exploration, normalization, and summaries of high density oligonucleotide array probe level data

Mesoangioblasts--vascular progenitors for extravascular mesodermal tissues

Cell Therapy of -Sarcoglycan Null Dystrophic Mice Through Intra-Arterial Delivery of Mesoangioblasts

ID helix-loop-helix proteins in cell growth, differentiation and tumorigenesis.

Inflammatory myopathies: clinical, diagnostic and therapeutic aspects.

Polymyositis and dermatomyositis.

Muscle differentiation during repair of myocardial necrosis in rats via gene transfer with MyoD.

Type beta transforming growth factor is an inhibitor of myogenic differentiation.

Msx2 and necdin combined activities are required for smooth muscle differentiation in mesoangioblast stem cells.

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45

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103

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Maurilio Sampaolesi

Enrico Tagliafico

Massimiliano Mirabella

Roberta Morosetti

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2006-10-31T00:00:00Z

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6719708

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17077152

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1636567

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