C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells - Wikidata - awgldk - TriplyDB

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

http://www.wikidata.org/entity/Q34579613

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TLR activation enhances C5a-induced pro-inflammatory responses by negatively modulating the second C5a receptor, C5L2 The dual role of complement in cancer and its implication in anti-tumor therapy Immunological cells and functions in Gaucher disease Complement in monoclonal antibody therapy of cancer On the Functional Overlap between Complement and Anti-Microbial Peptides Complement: a key system for immune surveillance and homeostasis Complement regulation in renal disease models Septicaemia models using Streptococcus pneumoniae and Listeria monocytogenes: understanding the role of complement properdin.Role and mechanism of action of complement in regulating T cell immunity.Absence of signaling into CD4⁺ cells via C3aR and C5aR enables autoinductive TGF-β1 signaling and induction of Foxp3⁺ regulatory T cells.Opposing roles for complement component C5a in tumor progression and the tumor microenvironment.Potential immunological consequences of pharmacological suppression of gastric acid production in patients with multiple sclerosis.Immunogenicity to biologics: mechanisms, prediction and reduction.Complement anaphylatoxins as immune regulators in cancer.A new role of the complement system: C3 provides protection in a mouse model of lung infection with intracellular Chlamydia psittaci.Negative regulation of pulmonary Th17 responses by C3a anaphylatoxin during allergic inflammation in mice.Complement in organ transplantation.MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway.Evidence for anti-inflammatory effects of C5a on the innate IL-17A/IL-23 axis C5aR expression in a novel GFP reporter gene knockin mouse: implications for the mechanism of action of C5aR signaling in T cell immunity.C5a receptor-dependent cell activation by physiological concentrations of desarginated C5a: insights from a novel label-free cellular assay.Regulatory effects of C5a on IL-17A, IL-17F, and IL-23 Complement regulates conventional DC-mediated NK-cell activation by inducing TGF-β1 in Gr-1+ myeloid cells Targeting regulatory T cells in the treatment of type 1 diabetes mellitus.Immunoglobulin G1 and immunoglobulin G4 antibodies in multiple sclerosis patients treated with IFNβ interact with the endogenous cytokine and activate complement.Targeting the complement system for the management of retinal inflammatory and degenerative diseases.Abrogation of immune complex glomerulonephritis by native carboxypeptidase and pharmacological antagonism of the C5a receptor.Complement and periodontitis.The role of complement in the diagnosis and management of allergic rhinitis and allergic asthma.Regulation of humoral immunity by complement.Targeted complement inhibition and microvasculature in transplants: a therapeutic perspective.Let's Tie the Knot: Marriage of Complement and Adaptive Immunity in Pathogen Evasion, for Better or Worse.More than complementing Tolls: complement-Toll-like receptor synergy and crosstalk in innate immunity and inflammation.Old dogs-new tricks: immunoregulatory properties of C3 and C5 cleavage fragments.The "ins and outs" of complement-driven immune responses.Differential capacity for complement receptor-mediated immune evasion by Porphyromonas gingivalis depending on the type of innate leukocyte.Complement C5a induces PD-L1 expression and acts in synergy with LPS through Erk1/2 and JNK signaling pathways.Complement drives Th17 cell differentiation and triggers autoimmune arthritis.Complement anaphylatoxin C4a inhibits C5a-induced neointima formation following arterial injury.Immediate T-Helper 17 Polarization Upon Triggering CD11b/c on HIV-Exposed Dendritic Cells.

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P2860

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

http://www.wikidata.org/entity/Q34579613

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@ast

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@en

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@nl

type

label

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@ast

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@en

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@nl

prefLabel

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@ast

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@en

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@nl

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P1433

European Journal of Immunology

P1476

C5a receptor-deficient dendritic cells promote induction of Treg and Th17 cells

@en

P2093

Craig Gerard

http://www.w3.org/2001/XMLSchema#string

Donald J Weaver

http://www.w3.org/2001/XMLSchema#string

Edimara S Reis

http://www.w3.org/2001/XMLSchema#string

Gabriele Köhl

http://www.w3.org/2001/XMLSchema#string

Jörg Köhl

http://www.w3.org/2001/XMLSchema#string

Manoj K Pandey

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Nathaniel Harris

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P2860

Activation of human CD4+ cells with CD3 and CD46 induces a T-regulatory cell 1 phenotype

An anti-inflammatory function for the complement anaphylatoxin C5a-binding protein, C5L2

Mechanism of action of the group A streptococcal C5a inactivator

The biological functions of T helper 17 cell effector cytokines in inflammation

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Host recognition and target differentiation by factor H, a regulator of the alternative pathway of complement

C5a mutants are potent antagonists of the C5a receptor (CD88) and of C5L2: position 69 is the locus that determines agonism or antagonism

The orphan receptor C5L2 has high affinity binding sites for complement fragments C5a and C5a des-Arg(74)

C5A anaphylatoxin and its seven transmembrane-segment receptor

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

P304

710-721

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scholarly article

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10.1002/EJI.200939333

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3

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40

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2010-03-01T00:00:00Z

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40694568

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P698

20017191

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3040298

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