Aberrant glycogen synthase kinase 3β is involved in pancreatic cancer cell invasion and resistance to therapy.
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Glycogen synthase kinase-3β is a pivotal mediator of cancer invasion and resistance to therapyGSK-3 as potential target for therapeutic intervention in cancerRoles of GSK-3 and microRNAs on epithelial mesenchymal transition and cancer stem cellsMolecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward.GSK3β mediates the carcinogenic effect of HPV16 in cervical cancerChemopreventive effect of resveratrol and apocynin on pancreatic carcinogenesis via modulation of nuclear phosphorylated GSK3β and ERK1/2Novel MicroRNA Reporter Uncovers Repression of Let-7 by GSK-3βGlycogen synthase kinase 3β inhibition as a therapeutic approach in the treatment of endometrial cancer.ST6Gal-I overexpression facilitates prostate cancer progression via the PI3K/Akt/GSK-3β/β-catenin signaling pathwayModulation of glycogen synthase kinase-3β following TRAIL combinatorial treatment in cancer cells.Efficacy of glycogen synthase kinase-3β targeting against osteosarcoma via activation of β-cateninGlycogen Synthase Kinase 3β in Pancreatic Cancer and its Implications in Chemotherapy and Radiation Therapy.Inhibition of glycogen synthase kinase 3 beta (GSK3β) suppresses the progression of esophageal squamous cell carcinoma by modifying STAT3 activity.GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression.Glycogen synthase kinase-3 (GSK3) inhibition induces prosurvival autophagic signals in human pancreatic cancer cells.CXCR4 promotes GSK3β expression in pancreatic cancer cells via the Akt pathway.Differential activity of GSK-3 isoforms regulates NF-κB and TRAIL- or TNFα induced apoptosis in pancreatic cancer cellsFlavonoid apigenin modified gene expression associated with inflammation and cancer and induced apoptosis in human pancreatic cancer cells through inhibition of GSK-3β/NF-κB signaling cascade.AR-A 014418 Used against GSK3beta Downregulates Expression of hnRNPA1 and SF2/ASF Splicing Factors.GSK3β mediates pancreatic cancer cell invasion in vitro via the CXCR4/MMP-2 Pathway.Colorectal cancer cells require glycogen synthase kinase-3β for sustaining mitosis via translocated promoter region (TPR)-dynein interaction.GSK-3β inhibits autophagy and enhances radiosensitivity in non-small cell lung cancer.ISO, via Upregulating MiR-137 Transcription, Inhibits GSK3β-HSP70-MMP-2 Axis, Resulting in Attenuating Urothelial Cancer Invasion.
P2860
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P2860
Aberrant glycogen synthase kinase 3β is involved in pancreatic cancer cell invasion and resistance to therapy.
description
2013 nî lūn-bûn
@nan
2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@ast
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@en
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@nl
type
label
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@ast
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@en
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@nl
prefLabel
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@ast
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@en
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@nl
P2093
P2860
P1433
P1476
Aberrant glycogen synthase kin ...... ion and resistance to therapy.
@en
P2093
Ayako Kitano
Hiroshi Sato
Kazuyuki Kawakami
Ken-Ichi Miyamoto
Mayumi Hirose
Mitsutoshi Nakada
Takahisa Takino
Takeo Shimasaki
Tomomi Higashi
Toshinari Minamoto
P2860
P304
P356
10.1371/JOURNAL.PONE.0055289
P407
P577
2013-02-08T00:00:00Z