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Amyotrophic lateral sclerosis: human challenge for neuroscience

Amyotrophic lateral sclerosis: human challenge for neuroscience

http://www.wikidata.org/entity/Q34598050

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Cellular distribution of superoxide dismutases in the rat CNS.Modeling Lou Gehrig's disease in the fruit fly.Familial amyotrophic lateral sclerosis.Pathological characterization of astrocytic hyaline inclusions in familial amyotrophic lateral sclerosis.Reexamination of the mechanism of hydroxyl radical adducts formed from the reaction between familial amyotrophic lateral sclerosis-associated Cu,Zn superoxide dismutase mutants and H2O2.Base excision repair in physiology and pathology of the central nervous system Therapeutic developments in amyotrophic lateral sclerosis.Effects of environmental enrichment on the amyotrophic lateral sclerosis mouse model.Transgenic mouse models of Alzheimer's disease and amyotrophic lateral sclerosis.Regional and ultrastructural immunolocalization of copper-zinc superoxide dismutase in rat central nervous system.

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Amyotrophic lateral sclerosis: human challenge for neuroscience

http://www.wikidata.org/entity/Q34598050

description

1995 nî lūn-bûn

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1995 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

1995 թվականի փետրվարին հրատարակված գիտական հոդված

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年论文

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name

Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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Proceedings of the National Academy of Sciences of the United States of America

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Amyotrophic lateral sclerosis: human challenge for neuroscience

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P2093

Rowland LP

http://www.w3.org/2001/XMLSchema#string

P2860

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

A frequent ala 4 to val superoxide dismutase-1 mutation is associated with a rapidly progressive familial amyotrophic lateral sclerosis

A mutant neurofilament subunit causes massive, selective motor neuron death: implications for the pathogenesis of human motor neuron disease

Neurofilament-deficient axons and perikaryal aggregates in viable transgenic mice expressing a neurofilament-beta-galactosidase fusion protein

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

Superoxide dismutase is an abundant component in cell bodies, dendrites, and axons of motor neurons and in a subset of other neurons.

Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis

Chronic inhibition of superoxide dismutase produces apoptotic death of spinal neurons.

Down-regulation of copper/zinc superoxide dismutase causes apoptotic death in PC12 neuronal cells.

Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity.

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1251-1253

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scholarly article

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10.1073/PNAS.92.5.1251

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1995-02-01T00:00:00Z

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15321191

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7877963

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amyotrophic lateral sclerosis

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42496

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